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IGF‐1 deficiency impairs neurovascular coupling in mice: implications for cerebromicrovascular aging

Aging cell, 2015-12, Vol.14 (6), p.1034-1044

2015

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Sleep deprivation alters task‐related changes in functional connectivity of the frontal cortex: A near‐infrared spectroscopy study

Brain and behavior, 2021-08, Vol.11 (8), p.e02135-n/a

2021

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IGF‐1 Regulates Vertebral Bone Aging Through Sex‐Specific and Time‐Dependent Mechanisms

Journal of bone and mineral research, 2016-02, Vol.31 (2), p.443-454

2016

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Aging‐induced proinflammatory shift in cytokine expression profile in rat coronary arteries

The FASEB journal, 2003-06, Vol.17 (9), p.1183-1185

2003

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Reduced adenosine diphosphate sensitivity in skeletal muscle mitochondria increases reactive oxygen species production in mouse models of aging and oxidative stress but not denervation

JCSM rapid communications, 2021-01, Vol.4 (1), p.75-89

2021

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You Are as Old as Your Arteries ‐ And Microvessels

The FASEB journal, 2015-04, Vol.29 (S1)

Ungvari, Zoltan

2015

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Obesity in aging exacerbates blood brain barrier disruption, neuroinflammation and oxidative stress in the mouse hippocampus: effects on expression of genes involved in beta‐amyloid generation and Alzheimer’s disease (665.1)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a

2014

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Midlife Obesity Impairs Neurovascular Coupling Responses

Obesity (Silver Spring, Md.), 2021-01, Vol.29 (1), p.17-17

2021

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Overexpression of catalase targeted to mitochondria improves neurovascular coupling responses in aged mice

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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Cellular Senescence in the Rostral Ventrolateral Medulla (RVLM) – Novel Implications for Obesity‐Induced Sympathoexcitation

The FASEB journal, 2019-04, Vol.33 (S1), p.563.3-563.3

2019

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Treatment with the mitochondrial‐targeted antioxidant peptide SS‐31 rescues neurovascular coupling responses and cerebrovascular endothelial function and improves cognition in aged mice

Aging cell, 2018-04, Vol.17 (2), p.n/a

2018

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Chemotherapy‐Induced Vascular Cognitive Impairment: Role of Endothelial Senescence

The FASEB journal, 2021-05, Vol.35 (S1), p.n/a, Article fasebj.2021.35.S1.02839

2021

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Aging exacerbates hypertension‐induced cerebral microhemorrhages in mice: role of resveratrol treatment in vasoprotection

Aging cell, 2015-06, Vol.14 (3), p.400-408

2015

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Accelerated cerebromicrovascular senescence contributes to cognitive decline in a mouse model of paclitaxel (Taxol)‐induced chemobrain

Aging cell, 2023-07, Vol.22 (7), p.e13832-n/a

2023

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Insulin‐like growth factor 1 deficiency exacerbates hypertension‐induced cerebral microhemorrhages in mice, mimicking the aging phenotype

Aging cell, 2017-06, Vol.16 (3), p.469-479

2017

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Prostaglandin E 2 at low concentrations dilates whereas at higher concentrations constricts human cerebral parenchymal arterioles: implications for neurovascular coupling responses

The FASEB journal, 2019-04, Vol.33 (S1)

2019

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Prostaglandin E2 at low concentrations dilates whereas at higher concentrations constricts human cerebral parenchymal arterioles: implications for neurovascular coupling responses

The FASEB journal, 2019-04, Vol.33 (S1), p.528.7-528.7

2019

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Endothelial deficiency of insulin‐like growth factor‐1 receptor leads to blood–brain barrier disruption and accelerated endothelial senescence in mice, mimicking aspects of the brain aging phenotype

Microcirculation (New York, N.Y. 1994), 2024-02, Vol.31 (2), p.e12840-n/a

2024

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Insulin‐like Growth Factor I Regulates Astrocyte Function

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Selective disruption of IGF‐1 signaling in astrocytes impairs neurovascular coupling in mice: implications for cerebromicrovascular aging

The FASEB journal, 2018-04, Vol.32 (S1), p.711.10-711.10

2018

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Obesity in aging exacerbates neuroinflammation and alters eicosanoid profiles in the mouse hippocampus: potential role in impaired synaptic plasticity and cognitive decline

The FASEB journal, 2018-04, Vol.32 (S1), p.711.7-711.7

2018

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Cerebral microhemorrhages impair gait coordination in mice

The FASEB journal, 2018-04, Vol.32 (S1), p.578.9-578.9

2018

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Interaction of obesity and Nrf2 deficiency exacerbates vascular aging: potential role of endothelial senescence

The FASEB journal, 2019-04, Vol.33 (S1), p.518.9-518.9

2019

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IGF‐1 deficiency impairs neurovascular coupling in mice: implications for cerebromicrovascular aging

Sleep deprivation alters task‐related changes in functional connectivity of the frontal cortex: A near‐infrared spectroscopy study

IGF‐1 Regulates Vertebral Bone Aging Through Sex‐Specific and Time‐Dependent Mechanisms

Aging‐induced proinflammatory shift in cytokine expression profile in rat coronary arteries

Reduced adenosine diphosphate sensitivity in skeletal muscle mitochondria increases reactive oxygen species production in mouse models of aging and oxidative stress but not denervation

Impaired Neurovascular Coupling and Increased Functional Connectivity in the Frontal Cortex Predict Age‐Related Cognitive Dysfunction

You Are as Old as Your Arteries ‐ And Microvessels

Obesity in aging exacerbates blood brain barrier disruption, neuroinflammation and oxidative stress in the mouse hippocampus: effects on expression of genes involved in beta‐amyloid generation and Alzheimer’s disease (665.1)

Midlife Obesity Impairs Neurovascular Coupling Responses

Overexpression of catalase targeted to mitochondria improves neurovascular coupling responses in aged mice

Cellular Senescence in the Rostral Ventrolateral Medulla (RVLM) – Novel Implications for Obesity‐Induced Sympathoexcitation

Treatment with the mitochondrial‐targeted antioxidant peptide SS‐31 rescues neurovascular coupling responses and cerebrovascular endothelial function and improves cognition in aged mice

Chemotherapy‐Induced Vascular Cognitive Impairment: Role of Endothelial Senescence

Aging exacerbates hypertension‐induced cerebral microhemorrhages in mice: role of resveratrol treatment in vasoprotection

Accelerated cerebromicrovascular senescence contributes to cognitive decline in a mouse model of paclitaxel (Taxol)‐induced chemobrain

Insulin‐like growth factor 1 deficiency exacerbates hypertension‐induced cerebral microhemorrhages in mice, mimicking the aging phenotype

Prostaglandin E 2 at low concentrations dilates whereas at higher concentrations constricts human cerebral parenchymal arterioles: implications for neurovascular coupling responses

Prostaglandin E2 at low concentrations dilates whereas at higher concentrations constricts human cerebral parenchymal arterioles: implications for neurovascular coupling responses

Treatment with the poly(ADP‐ribose) polymerase inhibitor PJ‐34 improves cerebromicrovascular endothelial function, neurovascular coupling responses and cognitive performance in aged mice, supporting the NAD + depletion hypothesis of neurovascular aging

Endothelial deficiency of insulin‐like growth factor‐1 receptor leads to blood–brain barrier disruption and accelerated endothelial senescence in mice, mimicking aspects of the brain aging phenotype

Insulin‐like Growth Factor I Regulates Astrocyte Function

Selective disruption of IGF‐1 signaling in astrocytes impairs neurovascular coupling in mice: implications for cerebromicrovascular aging

Obesity in aging exacerbates neuroinflammation and alters eicosanoid profiles in the mouse hippocampus: potential role in impaired synaptic plasticity and cognitive decline

Cerebral microhemorrhages impair gait coordination in mice

Interaction of obesity and Nrf2 deficiency exacerbates vascular aging: potential role of endothelial senescence

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