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Propionate and Alzheimer's Disease

Frontiers in aging neuroscience, 2021-01, Vol.12, p.580001-580001

2021

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The Immune System as a Therapeutic Target for Alzheimer’s Disease

Life (Basel, Switzerland), 2022-09, Vol.12 (9), p.1440

2022

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Luteolin reduces Alzheimer's disease pathologies induced by traumatic brain injury

International journal of molecular sciences, 2014-01, Vol.15 (1), p.895-904

2014

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Biodistribution of Infused Human Umbilical Cord Blood Cells in Alzheimer's Disease-Like Murine Model

Cell transplantation, 2016-01, Vol.25 (1), p.195-199

2016

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Human Cord Blood Serum-Derived APP α-Secretase Cleavage Activity is Mediated by C1 Complement

Cell transplantation, 2018-04, Vol.27 (4), p.666-676

2018

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Vasoactive intestinal peptide : cardiovascular effects

Cardiovascular research, 2001, Vol.49 (1), p.27-37

2001

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High-energy compounds promote physiological processing of Alzheimer's amyloid-β precursor protein and boost cell survival in culture

Journal of neurochemistry, 2012-11, Vol.123 (4), p.525-531

2012

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Eukaryotic initiation factor 3, subunit C silencing inhibits cell proliferation and promotes apoptosis in human ovarian cancer cells

Bioscience reports, 2019-08, Vol.39 (8)

2019

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Elevated [Ca2+]i levels occur with decreased calpain activity in aged fibroblasts and their reversal by energy-rich compounds: new paradigm for Alzheimer's disease prevention

Journal of Alzheimer's disease, 2013, Vol.37 (4), p.835

2013

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Octyl gallate markedly promotes anti-amyloidogenic processing of APP through estrogen receptor-mediated ADAM10 activation

PloS one, 2013-08, Vol.8 (8), p.e71913-e71913

2013

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Restoring Soluble Amyloid Precursor Protein α Functions as a Potential Treatment for Alzheimer's Disease

Journal of neuroscience research, 2017-04, Vol.95 (4), p.973-991

2017

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Therapeutic Cocktail Approach for Treatment of Hyperhomocysteinemia in Alzheimer’s Disease

Cell Medicine, 2018-01, Vol.10, p.215517901772228-2155179017722280

2018

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Scientific truth or false hope? Understanding Alzheimer's disease from an aging perspective

Journal of Alzheimer's disease, 2011-01, Vol.24 (1), p.3

2011

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Human Umbilical Cord Blood-Derived Monocytes Improve Cognitive Deficits and Reduce Amyloid-β Pathology in PSAPP Mice

Cell transplantation, 2015-01, Vol.24 (11), p.2237-2250

2015

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Human Umbilical Cord Blood Serum–derived α-Secretase

Cell transplantation, 2018-03, Vol.27 (3), p.438-455

2018

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Evidence supporting the role of calpain in the α-processing of amyloid-β precursor protein

Biochemical and biophysical research communications, 2012-04, Vol.420 (3), p.530-535

2012

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Beneficial effects of a pyrroloquinolinequinone-containing dietary formulation on motor deficiency, cognitive decline and mitochondrial dysfunction in a mouse model of Alzheimer’s disease

Heliyon, 2017-04, Vol.3 (4), p.e00279-e00279, Article e00279

2017

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Gallic acid is a dual α/β-secretase modulator that reverses cognitive impairment and remediates pathology in Alzheimer mice

The Journal of biological chemistry, 2020-11, Vol.295 (48), p.16251-16266

2020

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What to look for beyond "pathogenic" factors in senile dementia? A functional deficiency of Ca²⁺ signaling

Journal of Alzheimer's disease, 2011-01, Vol.27 (4), p.679

2011

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Targeting apolipoprotein E and N-terminal amyloid β-protein precursor interaction improves cognition and reduces amyloid pathology in Alzheimer’s mice

The Journal of biological chemistry, 2023-07, Vol.299 (7), p.104846-104846, Article 104846

2023

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Restoring Soluble Amyloid Precursor Protein α Functions as a Potential Treatment for A lzheimer's Disease

Journal of neuroscience research, 2017-04, Vol.95 (4), p.973-991

2017

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Restoring Soluble Amyloid Precursor Protein [alpha] Functions as a Potential Treatment for Alzheimer's Disease

Journal of neuroscience research, 2017-04, Vol.95 (4), p.973

2017

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Autism, heparan sulfate and potential interventions

Experimental neurology, 2022-07, Vol.353, p.114050-114050, Article 114050

2022

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C1q/TNF‐related protein 9: A novel therapeutic target in ischemic stroke?

Journal of neuroscience research, 2019-02, Vol.97 (2), p.128-136

2019

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Restoring sAPPα functions as a potential treatment for Alzheimer’s disease

Journal of neuroscience research, 2016-08, Vol.95 (4), p.973-991

2016

Volltextzugriff (PDF)

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Propionate and Alzheimer's Disease

The Immune System as a Therapeutic Target for Alzheimer’s Disease

Luteolin reduces Alzheimer's disease pathologies induced by traumatic brain injury

Biodistribution of Infused Human Umbilical Cord Blood Cells in Alzheimer's Disease-Like Murine Model

Human Cord Blood Serum-Derived APP α-Secretase Cleavage Activity is Mediated by C1 Complement

Vasoactive intestinal peptide : cardiovascular effects

High-energy compounds promote physiological processing of Alzheimer's amyloid-β precursor protein and boost cell survival in culture

Eukaryotic initiation factor 3, subunit C silencing inhibits cell proliferation and promotes apoptosis in human ovarian cancer cells

Elevated [Ca2+]i levels occur with decreased calpain activity in aged fibroblasts and their reversal by energy-rich compounds: new paradigm for Alzheimer's disease prevention

Octyl gallate markedly promotes anti-amyloidogenic processing of APP through estrogen receptor-mediated ADAM10 activation

Restoring Soluble Amyloid Precursor Protein α Functions as a Potential Treatment for Alzheimer's Disease

Therapeutic Cocktail Approach for Treatment of Hyperhomocysteinemia in Alzheimer’s Disease

Scientific truth or false hope? Understanding Alzheimer's disease from an aging perspective

Human Umbilical Cord Blood-Derived Monocytes Improve Cognitive Deficits and Reduce Amyloid-β Pathology in PSAPP Mice

Human Umbilical Cord Blood Serum–derived α-Secretase

Evidence supporting the role of calpain in the α-processing of amyloid-β precursor protein

Beneficial effects of a pyrroloquinolinequinone-containing dietary formulation on motor deficiency, cognitive decline and mitochondrial dysfunction in a mouse model of Alzheimer’s disease

Gallic acid is a dual α/β-secretase modulator that reverses cognitive impairment and remediates pathology in Alzheimer mice

What to look for beyond "pathogenic" factors in senile dementia? A functional deficiency of Ca²⁺ signaling

Targeting apolipoprotein E and N-terminal amyloid β-protein precursor interaction improves cognition and reduces amyloid pathology in Alzheimer’s mice

Restoring Soluble Amyloid Precursor Protein α Functions as a Potential Treatment for A lzheimer's Disease

Restoring Soluble Amyloid Precursor Protein [alpha] Functions as a Potential Treatment for Alzheimer's Disease

Autism, heparan sulfate and potential interventions

C1q/TNF‐related protein 9: A novel therapeutic target in ischemic stroke?

Restoring sAPPα functions as a potential treatment for Alzheimer’s disease

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