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Editorial: Methods and applications in cardiac electrophysiology-application to inherited arrhythmias

Frontiers in Physiology, 2024-03, Vol.15, p.1388433-1388433

2024

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Delayed rectifier K+ currents and cardiac repolarization

Journal of molecular and cellular cardiology, 2010-01, Vol.48 (1), p.37-44

2010

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Mutation in the KCNQ1 gene leading to the short QT-interval syndrome

Circulation (New York, N.Y.), 2004-05, Vol.109 (20), p.2394-2397

2004

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Gap-134, a Connexin43 activator, prevents age-related development of ventricular fibrosis in Scn5a+/− mice

Pharmacological research, 2020-09, Vol.159, p.104922-104922, Article 104922

2020

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Dual Effect of Phosphatidyl (4,5)-Bisphosphate PIP2 on Shaker K+ Channels

The Journal of biological chemistry, 2012-10, Vol.287 (43), p.36158-36167

2012

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Impaired KCNQ1–KCNE1 and Phosphatidylinositol-4,5-Bisphosphate Interaction Underlies the Long QT Syndrome

Circulation research, 2005-04, Vol.96 (7), p.730-739

2005

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14-3-3 Is a Regulator of the Cardiac Voltage-Gated Sodium Channel Nav1.5

Circulation research, 2006-06, Vol.98 (12), p.1538-1546

2006

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Computer modeling of whole-cell voltage-clamp analyses to delineate guidelines for good practice of manual and automated patch-clamp

Scientific reports, 2021-02, Vol.11 (1), p.3282-3282, Article 3282

2021

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Arrhythmias precede cardiomyopathy and remodeling of Ca2+ handling proteins in a novel model of long QT syndrome

Journal of molecular and cellular cardiology, 2018-10, Vol.123, p.13-25

2018

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Kv7.1 (KCNQ1) properties and channelopathies

The Journal of physiology, 2008-04, Vol.586 (7), p.1785-1789

2008

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Phosphatidylinositol-4,5-Bisphosphate (PIP2) Stabilizes the Open Pore Conformation of the Kv11.1 (hERG) Channel

Biophysical journal, 2010-08, Vol.99 (4), p.1110-1118

2010

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LQT1-associated Mutations Increase KCNQ1 Proteasomal Degradation Independently of Derlin-1

The Journal of biological chemistry, 2009-02, Vol.284 (8), p.5250-5256

2009

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Mouse Models of SCN5A-Related Cardiac Arrhythmias

Frontiers in physiology, 2012-01, Vol.3, p.210-210

2012

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KCNQ1 Channels Voltage Dependence through a Voltage-dependent Binding of the S4-S5 Linker to the Pore Domain

The Journal of biological chemistry, 2011-01, Vol.286 (1), p.707-716

2011

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Neural modulation of ion channels in cardiac arrhythmias: Clinical implications and future investigations

Heart rhythm, 2010-06, Vol.7 (6), p.847-849

2010

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Dysfunction of the Voltage‐Gated K+ Channel β2 Subunit in a Familial Case of Brugada Syndrome

Journal of the American Heart Association, 2016-06, Vol.5 (6), p.n/a

2016

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Haploinsufficiency in combination with aging causes SCN5A-linked hereditary Lenègre disease

Journal of the American College of Cardiology, 2003-02, Vol.41 (4), p.643-652

2003

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Dual effect of phosphatidylinositol (4,5)-bisphosphate PIP2 on Shaker K+ channels

The Journal of biological chemistry, 2013-04, Vol.288 (15), p.10951-10951

2013

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KCNE1–KCNQ1 osmoregulation by interaction of phosphatidylinositol‐4,5‐bisphosphate with Mg2+ and polyamines

The Journal of physiology, 2010-09, Vol.588 (18), p.3471-3483

2010

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Microarray Analysis Reveals Complex Remodeling of Cardiac Ion Channel Expression With Altered Thyroid Status: Relation to Cellular and Integrated Electrophysiology

Circulation research, 2003-02, Vol.92 (2), p.234-242

2003

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Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: Evidence for clinically significant I Kr-I Ks α-subunit interaction

Heart rhythm, 2009-12, Vol.6 (12), p.1792-1801

2009

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Toward Personalized Medicine: Using Cardiomyocytes Differentiated From Urine-Derived Pluripotent Stem Cells to Recapitulate Electrophysiological Characteristics of Type 2 Long QT Syndrome

Journal of the American Heart Association, 2015-09, Vol.4 (9), p.e002159-e002159

2015

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Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: Evidence for clinically significant IKr -IKs α-subunit interaction

Heart rhythm, 2009-12, Vol.6 (12), p.1792-1801

2009

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HIV-Tat induces a decrease in I Kr and I Ks via reduction in phosphatidylinositol-(4,5)-bisphosphate availability

Journal of molecular and cellular cardiology, 2016-10, Vol.99, p.1-13

2016

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Torsades de pointes complicating atrioventricular block: Evidence for a genetic predisposition

Heart rhythm, 2007-02, Vol.4 (2), p.170-174

2007

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Editorial: Methods and applications in cardiac electrophysiology-application to inherited arrhythmias

Delayed rectifier K+ currents and cardiac repolarization

Mutation in the KCNQ1 gene leading to the short QT-interval syndrome

Gap-134, a Connexin43 activator, prevents age-related development of ventricular fibrosis in Scn5a+/− mice

Dual Effect of Phosphatidyl (4,5)-Bisphosphate PIP2 on Shaker K+ Channels

Impaired KCNQ1–KCNE1 and Phosphatidylinositol-4,5-Bisphosphate Interaction Underlies the Long QT Syndrome

14-3-3 Is a Regulator of the Cardiac Voltage-Gated Sodium Channel Nav1.5

Computer modeling of whole-cell voltage-clamp analyses to delineate guidelines for good practice of manual and automated patch-clamp

Arrhythmias precede cardiomyopathy and remodeling of Ca2+ handling proteins in a novel model of long QT syndrome

Kv7.1 (KCNQ1) properties and channelopathies

Phosphatidylinositol-4,5-Bisphosphate (PIP2) Stabilizes the Open Pore Conformation of the Kv11.1 (hERG) Channel

LQT1-associated Mutations Increase KCNQ1 Proteasomal Degradation Independently of Derlin-1

Mouse Models of SCN5A-Related Cardiac Arrhythmias

KCNQ1 Channels Voltage Dependence through a Voltage-dependent Binding of the S4-S5 Linker to the Pore Domain

Neural modulation of ion channels in cardiac arrhythmias: Clinical implications and future investigations

Dysfunction of the Voltage‐Gated K+ Channel β2 Subunit in a Familial Case of Brugada Syndrome

Haploinsufficiency in combination with aging causes SCN5A-linked hereditary Lenègre disease

Dual effect of phosphatidylinositol (4,5)-bisphosphate PIP2 on Shaker K+ channels

KCNE1–KCNQ1 osmoregulation by interaction of phosphatidylinositol‐4,5‐bisphosphate with Mg2+ and polyamines

Microarray Analysis Reveals Complex Remodeling of Cardiac Ion Channel Expression With Altered Thyroid Status: Relation to Cellular and Integrated Electrophysiology

Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: Evidence for clinically significant I Kr-I Ks α-subunit interaction

Toward Personalized Medicine: Using Cardiomyocytes Differentiated From Urine-Derived Pluripotent Stem Cells to Recapitulate Electrophysiological Characteristics of Type 2 Long QT Syndrome

Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: Evidence for clinically significant IKr -IKs α-subunit interaction

HIV-Tat induces a decrease in I Kr and I Ks via reduction in phosphatidylinositol-(4,5)-bisphosphate availability

Torsades de pointes complicating atrioventricular block: Evidence for a genetic predisposition

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