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Editorial: Methods and applications in cardiac electrophysiology-application to inherited arrhythmias

Frontiers in Physiology, 2024-03, Vol.15, p.1388433-1388433

2024

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Modelling sudden cardiac death risks factors in patients with coronavirus disease of 2019: the hydroxychloroquine and azithromycin case

Europace (London, England), 2021-05, Vol.23 (7), p.1124-1136

2021

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Gap-134, a Connexin43 activator, prevents age-related development of ventricular fibrosis in Scn5a+/− mice

Pharmacological research, 2020-09, Vol.159, p.104922-104922, Article 104922

2020

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Dual Effect of Phosphatidyl (4,5)-Bisphosphate PIP2 on Shaker K+ Channels

The Journal of biological chemistry, 2012-10, Vol.287 (43), p.36158-36167

2012

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Arrhythmias precede cardiomyopathy and remodeling of Ca2+ handling proteins in a novel model of long QT syndrome

Journal of molecular and cellular cardiology, 2018-10, Vol.123, p.13-25

2018

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Mouse Models of SCN5A-Related Cardiac Arrhythmias

Frontiers in physiology, 2012-01, Vol.3, p.210-210

2012

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Dual effect of phosphatidylinositol (4,5)-bisphosphate PIP2 on Shaker K+ channels

The Journal of biological chemistry, 2013-04, Vol.288 (15), p.10951-10951

2013

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Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: Evidence for clinically significant I Kr-I Ks α-subunit interaction

Heart rhythm, 2009-12, Vol.6 (12), p.1792-1801

2009

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Physiological and Pathophysiological Insights of Nav1.4 and Nav1.5 Comparison

Frontiers in pharmacology, 2016-01, Vol.6, p.314-314

2016

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Modeling sudden cardiac death risks factors in COVID-19 patients -the hydroxychloroquine and azithromycin case

Europace (London, England), 2021-07

2021

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Guest Editors' Introduction

Journal of molecular and cellular cardiology, 2010, Vol.48 (1), p.1-1

2010

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Guest Editors' Introduction

Journal of molecular and cellular cardiology, 2010-01, Vol.48 (1), p.1

2010

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Dysfunction of the Voltage-Gated K+ Channel beta 2 Subunit in a Familial Case of Brugada Syndrome

Journal of the American Heart Association, 2016-06, Vol.5 (6)

2016

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HIV-Tat induces a decrease in IKr and IKs via reduction in phosphatidylinositol-(4,5)-bisphosphate availability

Journal of molecular and cellular cardiology, 2016-10, Vol.99, p.1-13

2016

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β3-Adrenoceptor Control the Cystic Fibrosis Transmembrane Conductance Regulator through a cAMP/Protein Kinase A-independent Pathway

The Journal of biological chemistry, 1999-03, Vol.274 (10), p.6107-6113

1999

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Guest Editors' Introduction

Journal of molecular and cellular cardiology, 2009, Vol.48 (1), p.1-1

2009

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A Molecular Substrate for Long QT in HIV Patients: Tat Protein Reduces IKR in Human Induced Pluripotent Stem Cells-Derived Cardiomyocytes

Biophysical journal, 2016-02, Vol.110 (3), p.103a-103a

2016

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HIV-Tat Induces a Decrease in IKr and IKs via Reduction in Phosphatidylinositol-(4,5)-Bisphosphate Availability

Biophysical journal, 2017-02, Vol.112 (3), p.405a-405a

2017

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HIV-Tat Induces a Decrease in I Kr and I Ks via Reduction in Phosphatidylinositol-(4,5)-Bisphosphate Availability

Biophysical journal, 2017-02, Vol.112 (3), p.405

2017

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HIV-Tat induces a decrease in IKr and IKsvia reduction in phosphatidylinositol-(4,5)-bisphosphate availability

Journal of molecular and cellular cardiology, 2016-10, Vol.99, p.1-13

2016

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0417 : SCN5A+/ΔQKP mice present late sodium current associated with long QT syndrome, dilated cardiomyopathy and ranolazine-sensitive spontaneous ventricular arrhythmias

Archives of Cardiovascular Diseases Supplements, 2015-04, Vol.7 (2), p.169-169

2015

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Dual Effect of PIP2 on Shaker K+ Channels

Biophysical journal, 2013-01, Vol.104 (2), p.464a-464a

2013

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In KCNQ1 Channels, a Long QT Mutation Induces a Regulation by Cholesterol Instead of Phosphatidylinositol-4,5-Bisphosphate

Biophysical journal, 2014-01, Vol.106 (2), p.140a-140a

2014

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Opposite Effects of the S4–S5 Linker and PIP2 on Voltage-Gated Channel Function: KCNQ1/KCNE1 and Other Channels

Frontiers in pharmacology, 2012, Vol.3

2012

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Editorial: Methods and applications in cardiac electrophysiology-application to inherited arrhythmias

Modelling sudden cardiac death risks factors in patients with coronavirus disease of 2019: the hydroxychloroquine and azithromycin case

Gap-134, a Connexin43 activator, prevents age-related development of ventricular fibrosis in Scn5a+/− mice

Dual Effect of Phosphatidyl (4,5)-Bisphosphate PIP2 on Shaker K+ Channels

Arrhythmias precede cardiomyopathy and remodeling of Ca2+ handling proteins in a novel model of long QT syndrome

Mouse Models of SCN5A-Related Cardiac Arrhythmias

Dual effect of phosphatidylinositol (4,5)-bisphosphate PIP2 on Shaker K+ channels

Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: Evidence for clinically significant I Kr-I Ks α-subunit interaction

RRAD mutation causes electrical and cytoskeletal defects in cardiomyocytes derived from a familial case of Brugada syndrome

Physiological and Pathophysiological Insights of Nav1.4 and Nav1.5 Comparison

Modeling sudden cardiac death risks factors in COVID-19 patients -the hydroxychloroquine and azithromycin case

Guest Editors' Introduction

Guest Editors' Introduction

Dysfunction of the Voltage-Gated K+ Channel beta 2 Subunit in a Familial Case of Brugada Syndrome

HIV-Tat induces a decrease in IKr and IKs via reduction in phosphatidylinositol-(4,5)-bisphosphate availability

β3-Adrenoceptor Control the Cystic Fibrosis Transmembrane Conductance Regulator through a cAMP/Protein Kinase A-independent Pathway

Guest Editors' Introduction

A Molecular Substrate for Long QT in HIV Patients: Tat Protein Reduces IKR in Human Induced Pluripotent Stem Cells-Derived Cardiomyocytes

HIV-Tat Induces a Decrease in IKr and IKs via Reduction in Phosphatidylinositol-(4,5)-Bisphosphate Availability

HIV-Tat Induces a Decrease in I Kr and I Ks via Reduction in Phosphatidylinositol-(4,5)-Bisphosphate Availability

HIV-Tat induces a decrease in IKr and IKsvia reduction in phosphatidylinositol-(4,5)-bisphosphate availability

0417 : SCN5A+/ΔQKP mice present late sodium current associated with long QT syndrome, dilated cardiomyopathy and ranolazine-sensitive spontaneous ventricular arrhythmias

Dual Effect of PIP2 on Shaker K+ Channels

In KCNQ1 Channels, a Long QT Mutation Induces a Regulation by Cholesterol Instead of Phosphatidylinositol-4,5-Bisphosphate

Opposite Effects of the S4–S5 Linker and PIP2 on Voltage-Gated Channel Function: KCNQ1/KCNE1 and Other Channels

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