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Succinylation Links Metabolism to Protein Functions

Neurochemical research, 2019-10, Vol.44 (10), p.2346-2359

2019

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Vitamin B1 (thiamine) and dementia

Annals of the New York Academy of Sciences, 2016-03, Vol.1367 (1), p.21-30

2016

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Altered succinylation of mitochondrial proteins, APP and tau in Alzheimer’s disease

Nature communications, 2022-01, Vol.13 (1), p.159-19, Article 159

2022

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Benfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy

Human molecular genetics, 2018-08, Vol.27 (16), p.2874-2892

2018

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Cause and consequence: Mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases

Biochimica et biophysica acta, 2010-01, Vol.1802 (1), p.122-134

2010

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Benfotiamine and Cognitive Decline in Alzheimer's Disease: Results of a Randomized Placebo-Controlled Phase IIa Clinical Trial

Journal of Alzheimer's disease, 2020-01, Vol.78 (3), p.989-1010

2020

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Mitochondrial abnormalities in Alzheimer brain: Mechanistic implications

Annals of neurology, 2005-05, Vol.57 (5), p.695-703

2005

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Obituary: In Memory of Myron Flint Beal, MD, November 6, 1950–June 12, 2021

Movement disorders, 2021-09, Vol.36 (9), p.2003-2004

2021

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Interactions of Mitochondria/Metabolism and Calcium Regulation in Alzheimer’s Disease: A Calcinist Point of View

Neurochemical research, 2017-06, Vol.42 (6), p.1636-1648

2017

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Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

Neurochemical research, 2010-12, Vol.35 (12), p.2107-2116

2010

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Changes in inflammatory processes associated with selective vulnerability following mild impairment of oxidative metabolism

Neurobiology of disease, 2007-05, Vol.26 (2), p.353-362

2007

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A mitocentric view of Alzheimer's disease suggests multi-faceted treatments

Journal of Alzheimer's disease, 2010-01, Vol.20 Suppl 2 (s2), p.S591-S607

2010

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Mitochondria/metabolic reprogramming in the formation of neurons from peripheral cells: Cause or consequence and the implications to their utility

Neurochemistry international, 2018-07, Vol.117, p.65-76

2018

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Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart

Basic research in cardiology, 2020-09, Vol.115 (5), p.53, Article 53

2020

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The RNA-binding protein HuR is essential for the B cell antibody response

Nature immunology, 2015-04, Vol.16 (4), p.415-425

2015

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Abnormal Glucose Metabolism in Alzheimer’s Disease: Relation to Autophagy/Mitophagy and Therapeutic Approaches

Neurochemical research, 2015-12, Vol.40 (12), p.2557-2569

2015

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Mild metabolic perturbations alter succinylation of mitochondrial proteins

Journal of neuroscience research, 2017-11, Vol.95 (11), p.2244-2252

2017

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Presenilins Are Enriched in Endoplasmic Reticulum Membranes Associated with Mitochondria

The American journal of pathology, 2009-11, Vol.175 (5), p.1810-1816

2009

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Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease

Neurochemistry international, 2009-02, Vol.54 (2), p.111-118

2009

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Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Journal of neurochemistry, 2016-12, Vol.139 (5), p.823

2016

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Abnormal thiamine-dependent processes in Alzheimer's Disease. Lessons from diabetes

Molecular and cellular neuroscience, 2013-07, Vol.55, p.17-25

2013

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Mitochondrial enzymes and endoplasmic reticulum calcium stores as targets of oxidative stress in neurodegenerative diseases

Journal of bioenergetics and biomembranes, 2004-08, Vol.36 (4), p.335-340

2004

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Reversal of Thiamine Deficiency-Induced Neurodegeneration

Journal of neuropathology and experimental neurology, 2003-02, Vol.62 (2), p.195-207

2003

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Nutritional metabolism and cerebral bioenergetics in Alzheimer's disease and related dementias

Alzheimer's & dementia, 2023-03, Vol.19 (3), p.1041-1066

2023

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Abnormalities in the Tricarboxylic Acid Cycle in Huntington Disease and in a Huntington Disease Mouse Model

Journal of neuropathology and experimental neurology, 2015-06, Vol.74 (6), p.527-537

2015

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Succinylation Links Metabolism to Protein Functions

Vitamin B1 (thiamine) and dementia

Altered succinylation of mitochondrial proteins, APP and tau in Alzheimer’s disease

Benfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy

Cause and consequence: Mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases

Benfotiamine and Cognitive Decline in Alzheimer's Disease: Results of a Randomized Placebo-Controlled Phase IIa Clinical Trial

Mitochondrial abnormalities in Alzheimer brain: Mechanistic implications

Obituary: In Memory of Myron Flint Beal, MD, November 6, 1950–June 12, 2021

Interactions of Mitochondria/Metabolism and Calcium Regulation in Alzheimer’s Disease: A Calcinist Point of View

Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

Changes in inflammatory processes associated with selective vulnerability following mild impairment of oxidative metabolism

A mitocentric view of Alzheimer's disease suggests multi-faceted treatments

Mitochondria/metabolic reprogramming in the formation of neurons from peripheral cells: Cause or consequence and the implications to their utility

Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart

The RNA-binding protein HuR is essential for the B cell antibody response

Abnormal Glucose Metabolism in Alzheimer’s Disease: Relation to Autophagy/Mitophagy and Therapeutic Approaches

Mild metabolic perturbations alter succinylation of mitochondrial proteins

Presenilins Are Enriched in Endoplasmic Reticulum Membranes Associated with Mitochondria

Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease

Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Abnormal thiamine-dependent processes in Alzheimer's Disease. Lessons from diabetes

Mitochondrial enzymes and endoplasmic reticulum calcium stores as targets of oxidative stress in neurodegenerative diseases

Reversal of Thiamine Deficiency-Induced Neurodegeneration

Nutritional metabolism and cerebral bioenergetics in Alzheimer's disease and related dementias

Abnormalities in the Tricarboxylic Acid Cycle in Huntington Disease and in a Huntington Disease Mouse Model

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