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NLRP3 inflammasome activation drives tau pathology

Nature (London), 2019-11, Vol.575 (7784), p.669-673

2019

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Spinal motor neuron protein supersaturation patterns are associated with inclusion body formation in ALS

Proceedings of the National Academy of Sciences - PNAS, 2017-05, Vol.114 (20), p.E3935-E3943

2017

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Huntington’s Disease Pathogenesis Is Modified In Vivo by Alfy/Wdfy3 and Selective Macroautophagy

Neuron (Cambridge, Mass.), 2020-03, Vol.105 (5), p.813-821.e6

2020

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C9orf72 ALS-FTD: recent evidence for dysregulation of the autophagy-lysosome pathway at multiple levels

Autophagy, 2021-11, Vol.17 (11), p.3306-3322

2021

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Liquid–liquid phase separation of amyloid-β oligomers modulates amyloid fibrils formation

The Journal of biological chemistry, 2023-03, Vol.299 (3), p.102926-102926, Article 102926

2023

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Pathophysiological importance of aggregated damaged proteins

Free radical biology & medicine, 2014-06, Vol.71, p.70-89

2014

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Small molecule-mediated stabilization of vesicle-associated helical α-synuclein inhibits pathogenic misfolding and aggregation

Nature communications, 2014-12, Vol.5 (1), p.5857, Article 5857

2014

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Tau neurotoxicity and rescue in animal models of human Tauopathies

Current opinion in neurobiology, 2016-02, Vol.36, p.52-58

2016

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Amyloid β-peptides interfere with mitochondrial preprotein import competence by a coaggregation process

Molecular biology of the cell, 2016-11, Vol.27 (21), p.3257-3272

2016

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TDP-1/TDP-43 potentiates human α-Synuclein (HASN) neurodegeneration in Caenorhabditis elegans

Biochimica et biophysica acta. Molecular basis of disease, 2020-10, Vol.1866 (10), p.165876-165876, Article 165876

2020

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Aberrant enteric neuromuscular system and dysbiosis in amyotrophic lateral sclerosis

Gut microbes, 2021-01, Vol.13 (1), p.1996848-1996848

2021

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Protein misassembly and aggregation as potential convergence points for non-genetic causes of chronic mental illness

Molecular psychiatry, 2019-07, Vol.24 (7), p.936-951

2019

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Influence of fullerenol on hIAPP aggregation: amyloid inhibition and mechanistic aspects

Physical chemistry chemical physics : PCCP, 2019-02, Vol.21 (7), p.4022-4031

2019

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Clusterin ameliorates tau pathology in vivo by inhibiting fibril formation

Acta neuropathologica communications, 2020-12, Vol.8 (1), p.210-210, Article 210

2020

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Synucleinopathies: common features and hippocampal manifestations

Cellular and molecular life sciences : CMLS, 2017-04, Vol.74 (8), p.1485-1501

2017

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Elucidating Critical Proteinopathic Mechanisms and Potential Drug Targets in Neurodegeneration

Cellular and molecular neurobiology, 2020-04, Vol.40 (3), p.313-345

2020

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Transcellular Spreading of Tau in Tauopathies

Chembiochem : a European journal of chemical biology, 2018-12, Vol.19 (23), p.2424-2432

2018

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Proteostasis disturbance in amyotrophic lateral sclerosis

Human molecular genetics, 2017-10, Vol.26 (R2), p.R91-R104

2017

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Inhibition of synucleinopathic seeding by rationally designed inhibitors

eLife, 2020-01, Vol.9

2020

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Differential effects of voluntary treadmill exercise and caloric restriction on tau pathogenesis in a mouse model of Alzheimer's disease-like tau pathology fed with Western diet

Progress in neuro-psychopharmacology & biological psychiatry, 2017-10, Vol.79 (Pt B), p.452-461

2017

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Charcot-Marie-Tooth disease type 2CC due to a frameshift mutation of the neurofilament heavy polypeptide gene in an Austrian family

Neuromuscular disorders : NMD, 2019-05, Vol.29 (5), p.392-397

2019

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Distinct microscopic mechanisms for the accelerated aggregation of pathogenic Tau mutants revealed by kinetic analysis

Physical chemistry chemical physics : PCCP, 2020-04, Vol.22 (14), p.7241-7249

2020

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Beta amyloid aggregates induce sensitised TLR4 signalling causing long-term potentiation deficit and rat neuronal cell death

Communications biology, 2020-02, Vol.3 (1), p.79-79, Article 79

2020

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14-3-3 mitigates alpha-synuclein aggregation and toxicity in the in vivo preformed fibril model

Acta neuropathologica communications, 2021-01, Vol.9 (1), p.13-16, Article 13

2021

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Tau assemblies do not behave like independently acting prion-like particles in mouse neural tissue

Acta neuropathologica communications, 2021-03, Vol.9 (1), p.41-41, Article 41

2021

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NLRP3 inflammasome activation drives tau pathology

Spinal motor neuron protein supersaturation patterns are associated with inclusion body formation in ALS

Huntington’s Disease Pathogenesis Is Modified In Vivo by Alfy/Wdfy3 and Selective Macroautophagy

C9orf72 ALS-FTD: recent evidence for dysregulation of the autophagy-lysosome pathway at multiple levels

Liquid–liquid phase separation of amyloid-β oligomers modulates amyloid fibrils formation

Pathophysiological importance of aggregated damaged proteins

Small molecule-mediated stabilization of vesicle-associated helical α-synuclein inhibits pathogenic misfolding and aggregation

Tau neurotoxicity and rescue in animal models of human Tauopathies

Amyloid β-peptides interfere with mitochondrial preprotein import competence by a coaggregation process

TDP-1/TDP-43 potentiates human α-Synuclein (HASN) neurodegeneration in Caenorhabditis elegans

Aberrant enteric neuromuscular system and dysbiosis in amyotrophic lateral sclerosis

Protein misassembly and aggregation as potential convergence points for non-genetic causes of chronic mental illness

Influence of fullerenol on hIAPP aggregation: amyloid inhibition and mechanistic aspects

Clusterin ameliorates tau pathology in vivo by inhibiting fibril formation

Synucleinopathies: common features and hippocampal manifestations

Elucidating Critical Proteinopathic Mechanisms and Potential Drug Targets in Neurodegeneration

Transcellular Spreading of Tau in Tauopathies

Proteostasis disturbance in amyotrophic lateral sclerosis

Inhibition of synucleinopathic seeding by rationally designed inhibitors

Differential effects of voluntary treadmill exercise and caloric restriction on tau pathogenesis in a mouse model of Alzheimer's disease-like tau pathology fed with Western diet

Charcot-Marie-Tooth disease type 2CC due to a frameshift mutation of the neurofilament heavy polypeptide gene in an Austrian family

Distinct microscopic mechanisms for the accelerated aggregation of pathogenic Tau mutants revealed by kinetic analysis

Beta amyloid aggregates induce sensitised TLR4 signalling causing long-term potentiation deficit and rat neuronal cell death

14-3-3 mitigates alpha-synuclein aggregation and toxicity in the in vivo preformed fibril model

Tau assemblies do not behave like independently acting prion-like particles in mouse neural tissue

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