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T24 HRAS transformed NIH/3T3 mouse cells (GhrasT-NIH/3T3) in serial tumorigenic in vitro/in vivo passages give rise to increasingly aggressive tumorigenic cell lines T1-A and T2-A and metastatic cell lines T3-HA and T4-PA

Experimental cell research, 2016-01, Vol.340 (1), p.1-11

2016

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Interactions between spider silk and cells--NIH/3T3 fibroblasts seeded on miniature weaving frames

PloS one, 2010-08, Vol.5 (8), p.e12032

2010

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Our Unique Cell Lines Derived Serially After Injection With Human T24 HRAS Transformed NIH/3T3 Mouse Cells (GhrasT‐NIH/3T3) Showed The Highly Tumorigenic T1‐A Cells Cultured From The First Primary Tumor Had A Six‐Fold Higher LD50 For Resveratrol Killing Than The LD50 For Highly Metastatic Lung T4‐PA Cells Derived After Three Subsequent In Vivo / In Vitro Serial Manipulations

The FASEB journal, 2019-04, Vol.33 (S1), p.496.9-496.9

2019

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Delivery of the reduced form of vitamin K 2(20) to NIH/3T3 cells partially protects against rotenone induced cell death

Scientific reports, 2022-11, Vol.12 (1), p.19878

2022

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Down‐regulation of dual‐specificity phosphatase 6, a negative regulator of oncogenic ERK signaling, by ACA‐28 induces apoptosis in NIH/3T3 cells overexpressing HER2/ErbB2

Genes to cells : devoted to molecular & cellular mechanisms, 2021-02, Vol.26 (2), p.109-116

2021

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Both v-Ha-Ras and v-Raf Stimulate Expression of the Vascular Endothelial Growth Factor in NIH 3T3 Cells (∗)

The Journal of biological chemistry, 1995-10, Vol.270 (43), p.25915-25919

1995

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The apoptotic effect of nanosilver is mediated by a ROS- and JNK-dependent mechanism involving the mitochondrial pathway in NIH3T3 cells

Toxicology letters, 2008-07, Vol.179 (3), p.130-139

2008

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Self-Targeting of Carbon Dots into the Cell Nucleus: Diverse Mechanisms of Toxicity in NIH/3T3 and L929 Cells

International journal of molecular sciences, 2021-05, Vol.22 (11), p.5608

2021

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Disulfide‐bond‐containing agamatine‐cystaminebisacrylamide polymer demonstrates better transfection efficiency and lower cytotoxicity than polyethylenimine in NIH/3T3 cells

Journal of cellular biochemistry, 2018-02, Vol.119 (2), p.1767-1779

2018

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Effects of Agaricus blazei polypeptide on cell senescence by regulation of Keap1/Nrf2/ARE and TLR4/NF-κBp65 signaling pathways and its mechanism in D-gal-induced NIH/3T3 cells

Journal of functional foods, 2020-09, Vol.72, p.104037, Article 104037

2020

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Abrogation of translation initiation factor eIF‐2 phosphorylation causes malignant transformation of NIH 3T3 cells

The EMBO journal, 1995-08, Vol.14 (15), p.3828-3834

1995

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Antimetastatic Effect of Epigenetic Drugs, Hydralazine and Valproic Acid, in Ras-Transformed NIH 3T3 Cells [Corrigendum]

OncoTargets and therapy, 2022-08, Vol.15, p.845-846

2022

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Molecularly Characterized Solvent Extracts and Saponins from Polygonum hydropiper L. Show High Anti-Angiogenic, Anti-Tumor, Brine Shrimp, and Fibroblast NIH/3T3 Cell Line Cytotoxicity

Frontiers in pharmacology, 2016-03, Vol.7, p.74-74

2016

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Inorganic Phosphate Export by the Retrovirus Receptor XPR1 in Metazoans

Cell reports (Cambridge), 2013-06, Vol.3 (6), p.1866-1873

2013

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Akt activation‐dependent protective effect of wild ginseng adventitious root protein against UVA‐induced NIH‐3T3 cell damage

Wound repair and regeneration, 2021-11, Vol.29 (6), p.1006-1016

2021

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Retraction notice to “Curcumin inhibits proliferation and soluble collagen synthesis of NIH/3T3 cell line by modulation of miR-29a and via ERK1/2 and β-catenin pathways” [Mol. Immunol. 116 (2019) 191–198]

Molecular immunology, 2022-10, Vol.150, p.245-245

2022

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Retraction notice to "Curcumin inhibits proliferation and soluble collagen synthesis of NIH/3T3 cell line by modulation of miR-29a and via ERK1/2 and β-catenin pathways" [Mol. Immunol. 116 (2019) 191–198]

Molecular immunology, 2022-06, Vol.146, p.90-90

2022

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Tripterine emerges as a potential anti-scarring agent in NIH/3T3 cells by repressing ANRIL

General physiology and biophysics, 2020-01, Vol.39 (4), p.355-362

2020

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The P34G mutation reduces the transforming activity of K-Ras and N-Ras in NIH 3T3 cells but not of H-Ras

The Journal of biological chemistry, 2018-07, Vol.293 (29), p.11651-11651

2018

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Statins modulate transcriptional activity of heme-oxygenase-1 promoter in NIH 3T3 Cells

Journal of cellular biochemistry, 2012-11, Vol.113 (11), p.3466-3475

2012

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Expression of Concern "Curcumin inhibits proliferation and soluble collagen synthesis of NIH/3T3 cell line by modulation of miR-29a and via ERK1/2 and β-catenin pathways" [Mol. Immunol. 116 (2019) 191–198]

Molecular immunology, 2022-01, Vol.141, p.345-345

2022

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Prolonged induction activates Cebpα independent adipogenesis in NIH/3T3 cells

PloS one, 2013, Vol.8 (1), p.e51459

2013

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Quercetin inhibits radiation-induced skin fibrosis

Radiation research, 2013-08, Vol.180 (2), p.205

2013

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Segmenting time‐lapse phase contrast images of adjacent NIH 3T3 cells

Journal of microscopy (Oxford), 2013-01, Vol.249 (1), p.41-52

2013

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Nickel‐smelting fumes increased the expression of HIF‐1α through PI3K/ERK pathway in NIH/3T3 cells

Journal of occupational health, 2016-09, Vol.58 (5), p.413-424

2016

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T24 HRAS transformed NIH/3T3 mouse cells (GhrasT-NIH/3T3) in serial tumorigenic in vitro/in vivo passages give rise to increasingly aggressive tumorigenic cell lines T1-A and T2-A and metastatic cell lines T3-HA and T4-PA

Interactions between spider silk and cells--NIH/3T3 fibroblasts seeded on miniature weaving frames

Delivery of the reduced form of vitamin K 2(20) to NIH/3T3 cells partially protects against rotenone induced cell death

Down‐regulation of dual‐specificity phosphatase 6, a negative regulator of oncogenic ERK signaling, by ACA‐28 induces apoptosis in NIH/3T3 cells overexpressing HER2/ErbB2

Both v-Ha-Ras and v-Raf Stimulate Expression of the Vascular Endothelial Growth Factor in NIH 3T3 Cells (∗)

The apoptotic effect of nanosilver is mediated by a ROS- and JNK-dependent mechanism involving the mitochondrial pathway in NIH3T3 cells

Self-Targeting of Carbon Dots into the Cell Nucleus: Diverse Mechanisms of Toxicity in NIH/3T3 and L929 Cells

Disulfide‐bond‐containing agamatine‐cystaminebisacrylamide polymer demonstrates better transfection efficiency and lower cytotoxicity than polyethylenimine in NIH/3T3 cells

Effects of Agaricus blazei polypeptide on cell senescence by regulation of Keap1/Nrf2/ARE and TLR4/NF-κBp65 signaling pathways and its mechanism in D-gal-induced NIH/3T3 cells

Abrogation of translation initiation factor eIF‐2 phosphorylation causes malignant transformation of NIH 3T3 cells

Antimetastatic Effect of Epigenetic Drugs, Hydralazine and Valproic Acid, in Ras-Transformed NIH 3T3 Cells [Corrigendum]

Molecularly Characterized Solvent Extracts and Saponins from Polygonum hydropiper L. Show High Anti-Angiogenic, Anti-Tumor, Brine Shrimp, and Fibroblast NIH/3T3 Cell Line Cytotoxicity

Inorganic Phosphate Export by the Retrovirus Receptor XPR1 in Metazoans

Akt activation‐dependent protective effect of wild ginseng adventitious root protein against UVA‐induced NIH‐3T3 cell damage

Retraction notice to “Curcumin inhibits proliferation and soluble collagen synthesis of NIH/3T3 cell line by modulation of miR-29a and via ERK1/2 and β-catenin pathways” [Mol. Immunol. 116 (2019) 191–198]

Retraction notice to "Curcumin inhibits proliferation and soluble collagen synthesis of NIH/3T3 cell line by modulation of miR-29a and via ERK1/2 and β-catenin pathways" [Mol. Immunol. 116 (2019) 191–198]

Tripterine emerges as a potential anti-scarring agent in NIH/3T3 cells by repressing ANRIL

The P34G mutation reduces the transforming activity of K-Ras and N-Ras in NIH 3T3 cells but not of H-Ras

Statins modulate transcriptional activity of heme-oxygenase-1 promoter in NIH 3T3 Cells

Expression of Concern "Curcumin inhibits proliferation and soluble collagen synthesis of NIH/3T3 cell line by modulation of miR-29a and via ERK1/2 and β-catenin pathways" [Mol. Immunol. 116 (2019) 191–198]

Prolonged induction activates Cebpα independent adipogenesis in NIH/3T3 cells

Quercetin inhibits radiation-induced skin fibrosis

Segmenting time‐lapse phase contrast images of adjacent NIH 3T3 cells

Nickel‐smelting fumes increased the expression of HIF‐1α through PI3K/ERK pathway in NIH/3T3 cells

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