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Allele-selective lowering of mutant HTT protein by HTT-LC3 linker compounds

Nature (London), 2019-11, Vol.575 (7781), p.203-209

2019

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Nucleation Inhibition of Huntingtin Protein (htt) by Polyproline PPII Helices: A Potential Interaction with the N‑Terminal α‑Helical Region of Htt

Biochemistry (Easton), 2020-02, Vol.59 (4), p.436-449

2020

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Revisiting the outcome of adult wild-type Htt inactivation in the context of HTT-lowering strategies for Huntington’s disease

Brain communications, 2023, Vol.5 (6), p.fcad344-fcad344

2023

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Hydrothermal Treatment (HTT) of Microalgae: Detailed Molecular Characterization of HTT Oil in View of HTT Mechanism Elucidation

Energy & fuels, 2012-01, Vol.26 (1), p.658-671

2012

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Complete suppression of Htt fibrilization and disaggregation of Htt fibrils by a trimeric chaperone complex

The EMBO journal, 2018-01, Vol.37 (2), p.282-299

2018

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Allele-selective transcriptional repression of mutant HTT for the treatment of Huntington's disease

Nature medicine, 2019-07, Vol.25 (7), p.1131-1142

2019

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Complete suppression of Htt fibrillization and disaggregation of Htt fibrils by a trimeric chaperone complex

The EMBO journal, 2021-10, Vol.40 (19), p.e109413-n/a

2021

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N-terminal Huntingtin (Htt) phosphorylation is a molecular switch regulating Htt aggregation, helical conformation, internalization, and nuclear targeting

The Journal of biological chemistry, 2018-11, Vol.293 (48), p.18540-18558

2018

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FAN1 modifies Huntington's disease progression by stabilizing the expanded HTT CAG repeat

Human molecular genetics, 2019-02, Vol.28 (4), p.650-661

2019

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HTT-lowering reverses Huntington's disease immune dysfunction caused by NFκB pathway dysregulation

Brain (London, England : 1878), 2014-03, Vol.137 (Pt 3), p.819-833

2014

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Identification of a HTT-specific binding motif in DNAJB1 essential for suppression and disaggregation of HTT

Nature communications, 2022-08, Vol.13 (1), p.4692-4692, Article 4692

2022

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Alternative processing of human HTT mRNA with implications for Huntington's disease therapeutics

Brain (London, England : 1878), 2022-12, Vol.145 (12), p.4409-4424

2022

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Small molecule splicing modifiers with systemic HTT-lowering activity

Nature communications, 2021-12, Vol.12 (1), p.7299-7299, Article 7299

2021

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The pathogenic exon 1 HTT protein is produced by incomplete splicing in Huntington's disease patients

Scientific reports, 2017-05, Vol.7 (1), p.1307-10, Article 1307

2017

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Mutant HTT (huntingtin) impairs mitophagy in a cellular model of Huntington disease

Autophagy, 2021-03, Vol.17 (3), p.672-689

2021

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Cognitive Assessment During Heat Tolerance Test (HTT): 645

Medicine and science in sports and exercise, 2022-09, Vol.54 (9S), p.160-160

2022

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Epidemiology of Huntington disease: first post-HTT gene analysis of prevalence in Italy

Clinical genetics, 2016-03, Vol.89 (3), p.367-370

2016

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Energy Efficiency for RF-Powered Backscatter Networks Using HTT Protocol

IEEE transactions on vehicular technology, 2020-11, Vol.69 (11), p.13932-13936

2020

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Aberrant splicing of HTT generates the pathogenic exon 1 protein in Huntington disease

Proceedings of the National Academy of Sciences - PNAS, 2013-02, Vol.110 (6), p.2366-2370

2013

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Modulating FKBP5/FKBP51 and autophagy lowers HTT (huntingtin) levels

Autophagy, 2021-12, Vol.17 (12), p.4119-4140

2021

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Detailed analysis of HTT repeat elements in human blood using targeted amplification‐free long‐read sequencing

Human mutation, 2018-09, Vol.39 (9), p.1262-1272

2018

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Comment on "A series of cases with Huntington-like phenotype and intermediate repeats in HTT" by Acuña and colleagues

Journal of the neurological sciences, 2022-11, Vol.442, p.120409-120409, Article 120409

2022

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Targeting Gpr52 lowers mutant HTT levels and rescues Huntington's disease-associated phenotypes

Brain (London, England : 1878), 2018-06, Vol.141 (6), p.1782-1798

2018

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A SNP in the HTT promoter alters NF-κB binding and is a bidirectional genetic modifier of Huntington disease

Nature neuroscience, 2015-06, Vol.18 (6), p.807-816

2015

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Allele-selective lowering of mutant HTT protein by HTT-LC3 linker compounds

Nucleation Inhibition of Huntingtin Protein (htt) by Polyproline PPII Helices: A Potential Interaction with the N‑Terminal α‑Helical Region of Htt

Revisiting the outcome of adult wild-type Htt inactivation in the context of HTT-lowering strategies for Huntington’s disease

Hydrothermal Treatment (HTT) of Microalgae: Detailed Molecular Characterization of HTT Oil in View of HTT Mechanism Elucidation

Complete suppression of Htt fibrilization and disaggregation of Htt fibrils by a trimeric chaperone complex

Allele-selective transcriptional repression of mutant HTT for the treatment of Huntington's disease

Complete suppression of Htt fibrillization and disaggregation of Htt fibrils by a trimeric chaperone complex

Intermediate and Expanded HTT Alleles and the Risk for α‐Synucleinopathies

N-terminal Huntingtin (Htt) phosphorylation is a molecular switch regulating Htt aggregation, helical conformation, internalization, and nuclear targeting

FAN1 modifies Huntington's disease progression by stabilizing the expanded HTT CAG repeat

HTT-lowering reverses Huntington's disease immune dysfunction caused by NFκB pathway dysregulation

Identification of a HTT-specific binding motif in DNAJB1 essential for suppression and disaggregation of HTT

Alternative processing of human HTT mRNA with implications for Huntington's disease therapeutics

Small molecule splicing modifiers with systemic HTT-lowering activity

The pathogenic exon 1 HTT protein is produced by incomplete splicing in Huntington's disease patients

Mutant HTT (huntingtin) impairs mitophagy in a cellular model of Huntington disease

Cognitive Assessment During Heat Tolerance Test (HTT): 645

Epidemiology of Huntington disease: first post-HTT gene analysis of prevalence in Italy

Energy Efficiency for RF-Powered Backscatter Networks Using HTT Protocol

Aberrant splicing of HTT generates the pathogenic exon 1 protein in Huntington disease

Modulating FKBP5/FKBP51 and autophagy lowers HTT (huntingtin) levels

Detailed analysis of HTT repeat elements in human blood using targeted amplification‐free long‐read sequencing

Comment on "A series of cases with Huntington-like phenotype and intermediate repeats in HTT" by Acuña and colleagues

Targeting Gpr52 lowers mutant HTT levels and rescues Huntington's disease-associated phenotypes

A SNP in the HTT promoter alters NF-κB binding and is a bidirectional genetic modifier of Huntington disease

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