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Helicobacter pylori Adapts to Chronic Infection and Gastric Disease via pH-Responsive BabA-Mediated Adherence

Cell host & microbe, 2017-03, Vol.21 (3), p.376-389

2017

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Helicobacter pylori Activates Matrix Metalloproteinase 10 in Gastric Epithelial Cells via EGFR and ERK-mediated Pathways

The Journal of infectious diseases, 2016-06, Vol.213 (11), p.1767-1776

2016

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Docosahexaenoic acid inhibits Helicobacter pylori growth in vitro and mice gastric mucosa colonization

PloS one, 2012-04, Vol.7 (4), p.e35072-e35072

2012

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Myeloid HIF-1 is protective in Helicobacter pylori-mediated gastritis

The Journal of immunology (1950), 2015-04, Vol.194 (7), p.3259-3266

2015

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Helicobacter pylori CagA protein induces factors involved in the epithelial to mesenchymal transition (EMT) in infected gastric epithelial cells in an EPIYA‐ phosphorylation‐dependent manner

The FEBS journal, 2016-01, Vol.283 (2), p.206-220

2016

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Functional Adaptation of BabA, the H. pylori ABO Blood Group Antigen Binding Adhesin

Science (American Association for the Advancement of Science), 2004-07, Vol.305 (5683), p.519-522

2004

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Gastric microbial community profiling reveals a dysbiotic cancer-associated microbiota

Gut, 2018-02, Vol.67 (2), p.226

2018

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SabA is the H. pylori hemagglutinin and is polymorphic in binding to sialylated glycans

PLoS pathogens, 2006-10, Vol.2 (10), p.e110-e110

2006

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Effect of Helicobacter pylori on gastrointestinal microbiota: a population-based study in Linqu, a high-risk area of gastric cancer

Gut, 2020-09, Vol.69 (9), p.1598

2020

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Mucosal microbiome dysbiosis in gastric carcinogenesis

Gut, 2018-06, Vol.67 (6), p.1024

2018

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Proton pump inhibitors and dysbiosis: Current knowledge and aspects to be clarified

World journal of gastroenterology : WJG, 2019-06, Vol.25 (22), p.2706-2719

2019

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Helicobacter pylori BabA Expression, Gastric Mucosal Injury, and Clinical Outcome

Clinical gastroenterology and hepatology, 2007-01, Vol.5 (1), p.49-58

2007

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Studies of mucus in mouse stomach, small intestine, and colon. I. Gastrointestinal mucus layers have different properties depending on location as well as over the Peyer's patches

American journal of physiology: Gastrointestinal and liver physiology, 2013-09, Vol.305 (5), p.G341-G347

2013

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Pathobiology of Helicobacter pylori –Induced Gastric Cancer

Gastroenterology (New York, N.Y. 1943), 2016-01, Vol.150 (1), p.64-78

2016

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HpaA Is Essential for Helicobacter pylori Colonization in Mice

Infection and Immunity, 2006-02, Vol.74 (2), p.920-926

2006

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Helicobacter pylori -induced gastric inflammation and gastric cancer

Cancer letters, 2014-04, Vol.345 (2), p.196-202

2014

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Mucin dynamics and enteric pathogens

Nature reviews. Microbiology, 2011-04, Vol.9 (4), p.265-278

2011

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Gastric Mucosal Recognition of Helicobacter pylori Is Independent of Toll-Like Receptor 4

The Journal of infectious diseases, 2003-03, Vol.187 (5), p.829-836

2003

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Effects of pH on Helicobacter pylori binding to human gastric mucins: identification of binding to non-MUC5AC mucins

Biochemical journal, 2004-12, Vol.384 (Pt 2), p.263-270

2004

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Life in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori

Nature reviews. Microbiology, 2013-06, Vol.11 (6), p.385-399

2013

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Epithelial-cell recognition of commensal bacteria and maintenance of immune homeostasis in the gut

Nature reviews. Immunology, 2008-06, Vol.8 (6), p.411-420

Artis, David

2008

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Pathogenesis and clinical management of Helicobacter pylori gastric infection

World journal of gastroenterology : WJG, 2019-10, Vol.25 (37), p.5578-5589

2019

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In vivo activation of pH-responsive oxidase-like graphitic nanozymes for selective killing of Helicobacter pylori

Nature communications, 2021-03, Vol.12 (1), p.2002-2002, Article 2002

2021

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Helicobacter pylori Adapts to Chronic Infection and Gastric Disease via pH-Responsive BabA-Mediated Adherence

Helicobacter pylori Activates Matrix Metalloproteinase 10 in Gastric Epithelial Cells via EGFR and ERK-mediated Pathways

Docosahexaenoic acid inhibits Helicobacter pylori growth in vitro and mice gastric mucosa colonization

Myeloid HIF-1 is protective in Helicobacter pylori-mediated gastritis

Helicobacter pylori CagA protein induces factors involved in the epithelial to mesenchymal transition (EMT) in infected gastric epithelial cells in an EPIYA‐ phosphorylation‐dependent manner

Functional Adaptation of BabA, the H. pylori ABO Blood Group Antigen Binding Adhesin

Gastric microbial community profiling reveals a dysbiotic cancer-associated microbiota

SabA is the H. pylori hemagglutinin and is polymorphic in binding to sialylated glycans

Effect of Helicobacter pylori on gastrointestinal microbiota: a population-based study in Linqu, a high-risk area of gastric cancer

Mucosal microbiome dysbiosis in gastric carcinogenesis

Personalized Gut Mucosal Colonization Resistance to Empiric Probiotics Is Associated with Unique Host and Microbiome Features

Proton pump inhibitors and dysbiosis: Current knowledge and aspects to be clarified

Helicobacter pylori BabA Expression, Gastric Mucosal Injury, and Clinical Outcome

Studies of mucus in mouse stomach, small intestine, and colon. I. Gastrointestinal mucus layers have different properties depending on location as well as over the Peyer's patches

Pathobiology of Helicobacter pylori –Induced Gastric Cancer

HpaA Is Essential for Helicobacter pylori Colonization in Mice

Helicobacter pylori -induced gastric inflammation and gastric cancer

Mucin dynamics and enteric pathogens

Genomic and Epigenomic Profiling of High-Risk Intestinal Metaplasia Reveals Molecular Determinants of Progression to Gastric Cancer

Gastric Mucosal Recognition of Helicobacter pylori Is Independent of Toll-Like Receptor 4

Effects of pH on Helicobacter pylori binding to human gastric mucins: identification of binding to non-MUC5AC mucins

Life in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori

Epithelial-cell recognition of commensal bacteria and maintenance of immune homeostasis in the gut

Pathogenesis and clinical management of Helicobacter pylori gastric infection

In vivo activation of pH-responsive oxidase-like graphitic nanozymes for selective killing of Helicobacter pylori

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