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Pathways of mammalian replication fork restart

Nature reviews. Molecular cell biology, 2010-10, Vol.11 (10), p.683-687

2010

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Increased global transcription activity as a mechanism of replication stress in cancer

Nature communications, 2016-10, Vol.7 (1), p.13087-13087, Article 13087

2016

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DNA repair pathways as targets for cancer therapy

Nature reviews. Cancer, 2008-03, Vol.8 (3), p.193-204

2008

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ATR inhibition induces synthetic lethality and overcomes chemoresistance in TP53- or ATM-defective chronic lymphocytic leukemia cells

Blood, 2016-02, Vol.127 (5), p.582-595

2016

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Replication fork dynamics and the DNA damage response

Biochemical journal, 2012-04, Vol.443 (1), p.13-26

2012

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PARP is activated at stalled forks to mediate Mre11-dependent replication restart and recombination

The EMBO journal, 2009-09, Vol.28 (17), p.2601-2615

2009

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BET Inhibition Induces HEXIM1- and RAD51-Dependent Conflicts between Transcription and Replication

Cell reports (Cambridge), 2018-11, Vol.25 (8), p.2061-2069.e4

2018

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Essential function of Chk1 can be uncoupled from DNA damage checkpoint and replication control

Proceedings of the National Academy of Sciences - PNAS, 2008-12, Vol.105 (52), p.20752-20757

2008

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Conflicts with transcription make early replication late

Molecular cell, 2022-09, Vol.82 (18), p.3315-3317

Petermann, Eva

2022

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PrimPol-dependent single-stranded gap formation mediates homologous recombination at bulky DNA adducts

Nature communications, 2020-11, Vol.11 (1), p.5863-14, Article 5863

2020

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WDR82/PNUTS-PP1 Prevents Transcription-Replication Conflicts by Promoting RNA Polymerase II Degradation on Chromatin

Cell reports (Cambridge), 2020-12, Vol.33 (9), p.108469-108469, Article 108469

2020

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ATR-dependent phosphorylation and activation of ATM in response to UV treatment or replication fork stalling

The EMBO journal, 2006-12, Vol.25 (24), p.5775-5782

2006

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Evidence That the ATR/Chk1 Pathway Maintains Normal Replication Fork Progression during Unperturbed S Phase

Cell cycle (Georgetown, Tex.), 2006-10, Vol.5 (19), p.2203-2209

2006

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Claspin promotes normal replication fork rates in human cells

Molecular biology of the cell, 2008-06, Vol.19 (6), p.2373-2378

2008

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Human single-stranded DNA binding protein 1 (hSSB1/NABP2) is required for the stability and repair of stalled replication forks

Nucleic acids research, 2014-06, Vol.42 (10), p.6326-6336

2014

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Loss of p53 suppresses replication-stress-induced DNA breakage in G1/S checkpoint deficient cells

eLife, 2018-10, Vol.7

2018

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BRCA2 and RAD51 promote double-strand break formation and cell death in response to gemcitabine

Molecular cancer therapeutics, 2014-10, Vol.13 (10), p.2412-2421

2014

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Chk1 Requirement for High Global Rates of Replication Fork Progression during Normal Vertebrate S Phase

Molecular and Cellular Biology, 2006-04, Vol.26 (8), p.3319-3326

2006

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Diminished origin-licensing capacity specifically sensitizes tumor cells to replication stress

Molecular cancer research, 2013-04, Vol.11 (4), p.370-380

2013

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Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place

Cancer discovery, 2018-05, Vol.8 (5), p.537-555

2018

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Human PIF1 helicase supports DNA replication and cell growth under oncogenic-stress

Oncotarget, 2014-11, Vol.5 (22), p.11381-11398

2014

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Pathways of mammalian replication fork restart

Increased global transcription activity as a mechanism of replication stress in cancer

DNA repair pathways as targets for cancer therapy

ATR inhibition induces synthetic lethality and overcomes chemoresistance in TP53- or ATM-defective chronic lymphocytic leukemia cells

Replication fork dynamics and the DNA damage response

PARP is activated at stalled forks to mediate Mre11-dependent replication restart and recombination

BET Inhibition Induces HEXIM1- and RAD51-Dependent Conflicts between Transcription and Replication

Essential function of Chk1 can be uncoupled from DNA damage checkpoint and replication control

Conflicts with transcription make early replication late

PrimPol-dependent single-stranded gap formation mediates homologous recombination at bulky DNA adducts

WDR82/PNUTS-PP1 Prevents Transcription-Replication Conflicts by Promoting RNA Polymerase II Degradation on Chromatin

ATR-dependent phosphorylation and activation of ATM in response to UV treatment or replication fork stalling

Evidence That the ATR/Chk1 Pathway Maintains Normal Replication Fork Progression during Unperturbed S Phase

Claspin promotes normal replication fork rates in human cells

Human single-stranded DNA binding protein 1 (hSSB1/NABP2) is required for the stability and repair of stalled replication forks

Loss of p53 suppresses replication-stress-induced DNA breakage in G1/S checkpoint deficient cells

BRCA2 and RAD51 promote double-strand break formation and cell death in response to gemcitabine

Chk1 Requirement for High Global Rates of Replication Fork Progression during Normal Vertebrate S Phase

Diminished origin-licensing capacity specifically sensitizes tumor cells to replication stress

Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place

Human PIF1 helicase supports DNA replication and cell growth under oncogenic-stress

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