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Ca²⁺ release-activated Ca²⁺ channel blockade as a potential tool in antipancreatitis therapy

Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (32), p.13186-13191

2013

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Activation of pancreatic stellate cells attenuates intracellular Ca2+ signals due to downregulation of TRPA1 and protects against cell death induced by alcohol metabolites

Cell death & disease, 2022-08, Vol.13 (8), p.1-14, Article 744

2022

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Calcium Elevation in Mitochondria Is the Main Ca2+ Requirement for Mitochondrial Permeability Transition Pore (mPTP) Opening

The Journal of biological chemistry, 2009-07, Vol.284 (31), p.20796-20803

2009

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A Novel Role for Bcl-2 in Regulation of Cellular Calcium Extrusion

Current biology, 2012-07, Vol.22 (13), p.1241-1246

2012

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The roles of calcium and ATP in the physiology and pathology of the exocrine pancreas

Physiological reviews, 2021-10, Vol.101 (4), p.1691-1744

2021

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Menadione-induced Reactive Oxygen Species Generation via Redox Cycling Promotes Apoptosis of Murine Pancreatic Acinar Cells

The Journal of biological chemistry, 2006-12, Vol.281 (52), p.40485-40492

2006

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Galectin-3 Interaction with Thomsen-Friedenreich Disaccharide on Cancer-associated MUC1 Causes Increased Cancer Cell Endothelial Adhesion

The Journal of biological chemistry, 2007-01, Vol.282 (1), p.773-781

2007

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Pancreatic protease activation by alcohol metabolite depends on Ca²⁺ release via acid store IP₃ receptors

Proceedings of the National Academy of Sciences - PNAS, 2009-06, Vol.106 (26), p.10758-10763

2009

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CRAC channel inhibitors in pancreatic pathologies

The Journal of physiology, 2022-04, Vol.600 (7), p.1597-1598

2022

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Nitric oxide signals are interlinked with calcium signals in normal pancreatic stellate cells upon oxidative stress and inflammation

Open biology, 2016-08, Vol.6 (8), p.160149

2016

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Bile Acids Induce Ca2+ Release from Both the Endoplasmic Reticulum and Acidic Intracellular Calcium Stores through Activation of Inositol Trisphosphate Receptors and Ryanodine Receptors

The Journal of biological chemistry, 2006-12, Vol.281 (52), p.40154-40163

2006

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The role of CFTR in diabetes‐induced pancreatic ductal dysfunction

The Journal of physiology, 2024-03, Vol.602 (6), p.993-994

2024

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Calmodulin protects against alcohol-induced pancreatic trypsinogen activation elicited via Ca²⁺ release through IP₃ receptors

Proceedings of the National Academy of Sciences - PNAS, 2011-04, Vol.108 (14), p.5873-5878

2011

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The role of Ca2+ signalling in the pathology of exocrine pancreas

Cell calcium (Edinburgh), 2023-06, Vol.112, p.102740-102740, Article 102740

2023

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Activation of Trypsinogen in Large Endocytic Vacuoles of Pancreatic Acinar Cells

Proceedings of the National Academy of Sciences - PNAS, 2007-03, Vol.104 (13), p.5674-5679

2007

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The role of Ca2+ in the pathophysiology of pancreatitis

The Journal of physiology, 2014-01, Vol.592 (2), p.269-280

2014

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Ca2+ signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca2+ channel blockade

The Journal of physiology, 2016-01, Vol.594 (2), p.281-293

2016

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Calcium signalling in the acinar environment of the exocrine pancreas: physiology and pathophysiology

The Journal of physiology, 2018-07, Vol.596 (14), p.2663-2678

2018

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Ca(2+) signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca(2+) channel blockade

The Journal of physiology, 2016-01, Vol.594 (2), p.281-293

2016

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Calcium and adenosine triphosphate control of cellular pathology: asparaginase-induced pancreatitis elicited via protease-activated receptor 2

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2016-08, Vol.371 (1700)

2016

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Galactose protects against cell damage in mouse models of acute pancreatitis

The Journal of clinical investigation, 2018-09, Vol.128 (9), p.3769-3778

2018

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Bile acids induce necrosis in pancreatic stellate cells dependent on calcium entry and sodium‐driven bile uptake

The Journal of physiology, 2016-11, Vol.594 (21), p.6147-6164

2016

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Both RyRs and TPCs are required for NAADP-induced intracellular Ca2+ release

Cell calcium (Edinburgh), 2015-09, Vol.58 (3), p.237-245

2015

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ABT‐199 (Venetoclax), a BH3‐mimetic Bcl‐2 inhibitor, does not cause Ca2+‐signalling dysregulation or toxicity in pancreatic acinar cells

British journal of pharmacology, 2019-11, Vol.176 (22), p.4402-4415

2019

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Reactive Oxygen Species Induced by Bile Acid Induce Apoptosis and Protect Against Necrosis in Pancreatic Acinar Cells

Gastroenterology (New York, N.Y. 1943), 2011-06, Vol.140 (7), p.2116-2125

2011

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Ca²⁺ release-activated Ca²⁺ channel blockade as a potential tool in antipancreatitis therapy

Activation of pancreatic stellate cells attenuates intracellular Ca2+ signals due to downregulation of TRPA1 and protects against cell death induced by alcohol metabolites

Calcium Elevation in Mitochondria Is the Main Ca2+ Requirement for Mitochondrial Permeability Transition Pore (mPTP) Opening

A Novel Role for Bcl-2 in Regulation of Cellular Calcium Extrusion

The roles of calcium and ATP in the physiology and pathology of the exocrine pancreas

Menadione-induced Reactive Oxygen Species Generation via Redox Cycling Promotes Apoptosis of Murine Pancreatic Acinar Cells

Galectin-3 Interaction with Thomsen-Friedenreich Disaccharide on Cancer-associated MUC1 Causes Increased Cancer Cell Endothelial Adhesion

Pancreatic protease activation by alcohol metabolite depends on Ca²⁺ release via acid store IP₃ receptors

CRAC channel inhibitors in pancreatic pathologies

Nitric oxide signals are interlinked with calcium signals in normal pancreatic stellate cells upon oxidative stress and inflammation

Bile Acids Induce Ca2+ Release from Both the Endoplasmic Reticulum and Acidic Intracellular Calcium Stores through Activation of Inositol Trisphosphate Receptors and Ryanodine Receptors

The role of CFTR in diabetes‐induced pancreatic ductal dysfunction

Calmodulin protects against alcohol-induced pancreatic trypsinogen activation elicited via Ca²⁺ release through IP₃ receptors

The role of Ca2+ signalling in the pathology of exocrine pancreas

Activation of Trypsinogen in Large Endocytic Vacuoles of Pancreatic Acinar Cells

The role of Ca2+ in the pathophysiology of pancreatitis

Ca2+ signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca2+ channel blockade

Calcium signalling in the acinar environment of the exocrine pancreas: physiology and pathophysiology

Ca(2+) signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca(2+) channel blockade

Calcium and adenosine triphosphate control of cellular pathology: asparaginase-induced pancreatitis elicited via protease-activated receptor 2

Galactose protects against cell damage in mouse models of acute pancreatitis

Bile acids induce necrosis in pancreatic stellate cells dependent on calcium entry and sodium‐driven bile uptake

Both RyRs and TPCs are required for NAADP-induced intracellular Ca2+ release

ABT‐199 (Venetoclax), a BH3‐mimetic Bcl‐2 inhibitor, does not cause Ca2+‐signalling dysregulation or toxicity in pancreatic acinar cells

Reactive Oxygen Species Induced by Bile Acid Induce Apoptosis and Protect Against Necrosis in Pancreatic Acinar Cells

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