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Role of Mitochondria-Associated ER Membranes in Calcium Regulation in Cancer-Specific Settings

Neoplasia (New York, N.Y.), 2018-05, Vol.20 (5), p.510-523

2018

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Dynamic control of mitochondria-associated membranes by kinases and phosphatases in health and disease

Cellular and molecular life sciences : CMLS, 2021-10, Vol.78 (19-20), p.6541-6556

2021

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Endoplasmic Reticulum–Mitochondrial Ca2+ Fluxes Underlying Cancer Cell Survival

Frontiers in oncology, 2017-05, Vol.7, p.70-70

2017

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Recent advances in uncovering the mechanisms contributing to BIRD-2-induced cell death in B-cell cancer cells

Cell death & disease, 2019-01, Vol.10 (2), p.42-42, Article 42

2019

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The AMPK-related kinase NUAK1 controls cortical axons branching by locally modulating mitochondrial metabolic functions

Nature communications, 2024-03, Vol.15 (1), p.2487-2487

2024

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Intracellular BAPTA directly inhibits PFKFB3, thereby impeding mTORC1-driven Mcl-1 translation and killing MCL-1-addicted cancer cells

Cell death & disease, 2023-09, Vol.14 (9), p.600-600, Article 600

2023

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Cytosolic Ca2+ oversees the MASs production of pyruvate for the mitochondrial market

Cell calcium (Edinburgh), 2020-07, Vol.89, p.102223-102223, Article 102223

Kerkhofs, Martijn

2020

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Getting old without type 2 IP3 receptors

Cell calcium (Edinburgh), 2021-09, Vol.98, p.102437-102437, Article 102437

2021

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DLBCL Cells with Acquired Resistance to Venetoclax Are Not Sensitized to BIRD-2 But Can Be Resensitized to Venetoclax through Bcl-XL Inhibition

Biomolecules (Basel, Switzerland), 2020-07, Vol.10 (7), p.1081

2020

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Therapeutic implications of novel peptides targeting ER–mitochondria Ca2+-flux systems

Drug discovery today, 2019-05, Vol.24 (5), p.1092-1103

2019

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Bok joining the “Ca2+ club”

Cell calcium (Edinburgh), 2021-09, Vol.98, p.102438-102438, Article 102438

2021

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Balancing ER-Mitochondrial Ca2+ Fluxes in Health and Disease

Trends in cell biology, 2021-07, Vol.31 (7), p.598-612

2021

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VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like disease

Science (American Association for the Advancement of Science), 2019-12, Vol.366 (6472), p.1531-1536

2019

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Emerging molecular mechanisms in chemotherapy: Ca2+ signaling at the mitochondria-associated endoplasmic reticulum membranes

Cell death & disease, 2018-02, Vol.9 (3), p.1-15, Article 334

2018

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Cancer cell death strategies by targeting Bcl-2's BH4 domain

Biochimica et biophysica acta. Molecular cell research, 2021-04, Vol.1868 (5), p.118983-118983, Article 118983

2021

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Pathophysiological consequences of isoform-specific IP3 receptor mutations

Biochimica et biophysica acta. Molecular cell research, 2018-11, Vol.1865 (11), p.1707-1717

2018

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BIRD-2, a BH4-domain-targeting peptide of Bcl-2, provokes Bax/Bak-independent cell death in B-cell cancers through mitochondrial Ca2+-dependent mPTP opening

Cell calcium (Edinburgh), 2021-03, Vol.94, p.102333-102333, Article 102333

2021

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Endoplasmic Reticulum-Mitochondrial Ca 2+ Fluxes Underlying Cancer Cell Survival

Frontiers in oncology, 2017, Vol.7, p.70

2017

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The alkalinizing, lysosomotropic agent ML-9 induces a pH-dependent depletion of ER Ca2+ stores in cellulo

Biochimica et biophysica acta. Molecular cell research, 2022-10, Vol.1869 (10), p.119308-119308, Article 119308

2022

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The ER-mitochondria interface, where Ca2+ and cell death meet

Cell calcium (Edinburgh), 2023-06, Vol.112, p.102743-102743, Article 102743

2023

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Therapeutic implications of novel peptides targeting ER-mitochondria Ca 2+ -flux systems

Drug discovery today, 2019-05, Vol.24 (5), p.1092

2019

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Pathophysiological consequences of isoform-specific IP 3 receptor mutations

Biochimica et biophysica acta. Molecular cell research, 2018-11, Vol.1865 (11 Pt B), p.1707

2018

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Bcl-2 inhibitors as anti-cancer therapeutics: the impact of and on calcium signaling

Cell calcium (Edinburgh), 2018-03, Vol.70, p.102-116

2018

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Endoplasmic Reticulum-Mitochondria Communication Through Ca2+ Signaling: The Importance of Mitochondria-Associated Membranes (MAMs)

Organelle Contact Sites, 2017, p.49-67

2017

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Role of Mitochondria-Associated ER Membranes in Calcium Regulation in Cancer-Specific Settings

Non-canonical function of IRE1α determines mitochondria-associated endoplasmic reticulum composition to control calcium transfer and bioenergetics

Dynamic control of mitochondria-associated membranes by kinases and phosphatases in health and disease

Endoplasmic Reticulum–Mitochondrial Ca2+ Fluxes Underlying Cancer Cell Survival

Recent advances in uncovering the mechanisms contributing to BIRD-2-induced cell death in B-cell cancer cells

The AMPK-related kinase NUAK1 controls cortical axons branching by locally modulating mitochondrial metabolic functions

Intracellular BAPTA directly inhibits PFKFB3, thereby impeding mTORC1-driven Mcl-1 translation and killing MCL-1-addicted cancer cells

Cytosolic Ca2+ oversees the MASs production of pyruvate for the mitochondrial market

Getting old without type 2 IP3 receptors

DLBCL Cells with Acquired Resistance to Venetoclax Are Not Sensitized to BIRD-2 But Can Be Resensitized to Venetoclax through Bcl-XL Inhibition

Therapeutic implications of novel peptides targeting ER–mitochondria Ca2+-flux systems

Bok joining the “Ca2+ club”

Balancing ER-Mitochondrial Ca2+ Fluxes in Health and Disease

VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like disease

Emerging molecular mechanisms in chemotherapy: Ca2+ signaling at the mitochondria-associated endoplasmic reticulum membranes

Cancer cell death strategies by targeting Bcl-2's BH4 domain

Pathophysiological consequences of isoform-specific IP3 receptor mutations

BIRD-2, a BH4-domain-targeting peptide of Bcl-2, provokes Bax/Bak-independent cell death in B-cell cancers through mitochondrial Ca2+-dependent mPTP opening

Endoplasmic Reticulum-Mitochondrial Ca 2+ Fluxes Underlying Cancer Cell Survival

The alkalinizing, lysosomotropic agent ML-9 induces a pH-dependent depletion of ER Ca2+ stores in cellulo

The ER-mitochondria interface, where Ca2+ and cell death meet

Therapeutic implications of novel peptides targeting ER-mitochondria Ca 2+ -flux systems

Pathophysiological consequences of isoform-specific IP 3 receptor mutations

Bcl-2 inhibitors as anti-cancer therapeutics: the impact of and on calcium signaling

Endoplasmic Reticulum-Mitochondria Communication Through Ca2+ Signaling: The Importance of Mitochondria-Associated Membranes (MAMs)

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