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Chronic cardiac structural damage, diastolic and systolic dysfunction following acute myocardial injury due to bromine exposure in rats

Archives of toxicology, 2021-01, Vol.95 (1), p.179-193

2021

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5-, 12- and 15-Hydroxyeicosatetraenoic acids induce cellular hypertrophy in the human ventricular cardiomyocyte, RL-14 cell line, through MAPK- and NF-κB-dependent mechanism

Archives of toxicology, 2016-02, Vol.90 (2), p.359-373

2016

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Development of cardiac hypertrophy by sunitinib in vivo and in vitro rat cardiomyocytes is influenced by the aryl hydrocarbon receptor signaling pathway

Archives of toxicology, 2014-03, Vol.88 (3), p.725-738

2014

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The Effects of Angiotensin II and Specific Angiotensin Receptor Blockers on Embryonic Cardiac Development and Looping Patterns

Developmental biology, 1997-12, Vol.192 (2), p.572-584

1997

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Evaluation of the cardiac isoform of alpha2-macroglobulin as a factor inducing cardiac hypertrophy

Methods in molecular medicine, 2005, Vol.112, p.261

2005

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Delineation of sequences essential for specific promoter activation during pressure overloaded hypertrophy or factor-induced hypertrophy

Methods in molecular medicine, 2005, Vol.112, p.251

2005

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Embryotoxic effects of L-691,121, a class III antiarrhythmic agent, in rats

Archives of toxicology, 1994-11, Vol.69 (1), p.65-71

1994

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Cardiac ultrastructural changes induced by daunorubicin therapy

Cancer, 1973-10, Vol.32 (4), p.771-788

1973

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Cardiac hypertrophy in the dog and rat induced by oxfenicine, an agent which modifies muscle metabolism

Archives of toxicology. Supplement. = Archiv fur Toxikologie. Supplement, 1984, Vol.7, p.488

1984

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Restriction of beta-adrenergic myocardial reactions (45 Ca-uptake, ATP utilization) in cardiomegaly induced by isoproterenol

Verhandlungen der Deutschen Gesellschaft für Kreislaufforschung, 1973, Vol.39, p.161

1973

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Rat models of cardiovascular diseases

Methods in molecular biology (Clifton, N.J.), 2010-01, Vol.597, p.403

Bader, Michael

2010

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Successful long‐term control of idiopathic hypereosinophilic syndrome with etoposide

Cancer, 1991-06, Vol.67 (11), p.2826-2827

1991

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Effects of tryptophan on the development of deoxycorticosterone acetate (DOCA)-induced hypertension in rats

Advances in experimental medicine and biology, 1991, Vol.294, p.619

Fregly, M J

1991

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Comparison of hemodynamic and vectorcardiographic parameters of right cardiac hypertrophy in primary pulmonary hypertension. Analysis of 77 cases

Verhandlungen der Deutschen Gesellschaft für Kreislaufforschung, 1972, Vol.38, p.130

1972

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Chronic cardiac structural damage, diastolic and systolic dysfunction following acute myocardial injury due to bromine exposure in rats

5-, 12- and 15-Hydroxyeicosatetraenoic acids induce cellular hypertrophy in the human ventricular cardiomyocyte, RL-14 cell line, through MAPK- and NF-κB-dependent mechanism

Development of cardiac hypertrophy by sunitinib in vivo and in vitro rat cardiomyocytes is influenced by the aryl hydrocarbon receptor signaling pathway

The Effects of Angiotensin II and Specific Angiotensin Receptor Blockers on Embryonic Cardiac Development and Looping Patterns

Evaluation of the cardiac isoform of alpha2-macroglobulin as a factor inducing cardiac hypertrophy

Delineation of sequences essential for specific promoter activation during pressure overloaded hypertrophy or factor-induced hypertrophy

Embryotoxic effects of L-691,121, a class III antiarrhythmic agent, in rats

Cardiac ultrastructural changes induced by daunorubicin therapy

Cardiac hypertrophy in the dog and rat induced by oxfenicine, an agent which modifies muscle metabolism

Restriction of beta-adrenergic myocardial reactions (45 Ca-uptake, ATP utilization) in cardiomegaly induced by isoproterenol

Rat models of cardiovascular diseases

Successful long‐term control of idiopathic hypereosinophilic syndrome with etoposide

Effects of tryptophan on the development of deoxycorticosterone acetate (DOCA)-induced hypertension in rats

Comparison of hemodynamic and vectorcardiographic parameters of right cardiac hypertrophy in primary pulmonary hypertension. Analysis of 77 cases

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