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A New Initiative on Precision Medicine

The New England journal of medicine, 2015-02, Vol.372 (9), p.793-795

2015

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New Era in Cancer Research

Science (American Association for the Advancement of Science), 2006-05, Vol.312 (5777), p.1162-1165

Varmus, Harold

2006

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Three decades of Wnts: a personal perspective on how a scientific field developed

The EMBO journal, 2012-06, Vol.31 (12), p.2670-2684

2012

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Toward a Shared Vision for Cancer Genomic Data

The New England journal of medicine, 2016-09, Vol.375 (12), p.1109-1112

2016

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Insurance for broad genomic tests in oncology

Science (American Association for the Advancement of Science), 2017-12, Vol.358 (6367), p.1133-1134

2017

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Of oncogenes and open science: an interview with Harold Varmus

Disease models & mechanisms, 2019-03, Vol.12 (3)

Varmus, Harold

2019

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Rescuing US biomedical research from its systemic flaws

Proceedings of the National Academy of Sciences - PNAS, 2014-04, Vol.111 (16), p.5773-5777

2014

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Sensitivity of human lung adenocarcinoma cell lines to targeted inhibition of BET epigenetic signaling proteins

Proceedings of the National Academy of Sciences - PNAS, 2012-11, Vol.109 (47), p.19408-19413

2012

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KRAS mutations and primary resistance of lung adenocarcinomas to gefitinib or erlotinib

PLoS medicine, 2005-01, Vol.2 (1), p.e17-e17

2005

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Lung Cancer Signatures in Plasma Based on Proteome Profiling of Mouse Tumor Models

Cancer cell, 2011-09, Vol.20 (3), p.289-299

2011

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Evidence that synthetic lethality underlies the mutual exclusivity of oncogenic KRAS and EGFR mutations in lung adenocarcinoma

eLife, 2015-06, Vol.4, p.e06907-e06907

2015

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Hyperactivation of ERK by multiple mechanisms is toxic to RTK-RAS mutation-driven lung adenocarcinoma cells

eLife, 2018-11, Vol.7

2018

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Arlen Specter: A retrospective

Proceedings of the National Academy of Sciences - PNAS, 2012-12, Vol.109 (52), p.21178-21179

Varmus, Harold

2012

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Epstein-Barr virus: an important vaccine target for cancer prevention

Science translational medicine, 2011-11, Vol.3 (107), p.107fs7

2011

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Generation of pulmonary neuroendocrine cells and SCLC-like tumors from human embryonic stem cells

The Journal of experimental medicine, 2019-03, Vol.216 (3), p.674-687

2019

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Improving support for young biomedical scientists

Science (American Association for the Advancement of Science), 2018-05, Vol.360 (6390), p.716-718

2018

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Lung adenocarcinomas induced in mice by mutant EGF receptors found in human lung cancers respond to a tyrosine kinase inhibitor or to down-regulation of the receptors

Genes & development, 2006-06, Vol.20 (11), p.1496-1510

2006

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Superoxide dismutase 1 (SOD1) is a target for a small molecule identified in a screen for inhibitors of the growth of lung adenocarcinoma cell lines

Proceedings of the National Academy of Sciences - PNAS, 2011-09, Vol.108 (39), p.16375-16380

2011

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Wild-Type U2AF1 Antagonizes the Splicing Program Characteristic of U2AF1-Mutant Tumors and Is Required for Cell Survival

PLoS genetics, 2016-10, Vol.12 (10), p.e1006384-e1006384

2016

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Seeding and Propagation of Untransformed Mouse Mammary Cells in the Lung

Science (American Association for the Advancement of Science), 2008-09, Vol.321 (5897), p.1841-1844

2008

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Opinion: Addressing systemic problems in the biomedical research enterprise

Proceedings of the National Academy of Sciences - PNAS, 2015-02, Vol.112 (7), p.1912-1913

2015

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Oncogene cooperation in tumor maintenance and tumor recurrence in mouse mammary tumors induced by Myc and mutant Kras

Proceedings of the National Academy of Sciences - PNAS, 2008-04, Vol.105 (13), p.5242-5247

2008

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Acquired resistance of lung adenocarcinomas to gefitinib or erlotinib is associated with a second mutation in the EGFR kinase domain

PLoS medicine, 2005-03, Vol.2 (3), p.e73-e73

2005

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Threatening the Global AIDS Response — Obstacles to PEPFAR’s Reauthorization

The New England journal of medicine, 2023-09, Vol.389 (13), p.1159-1161

2023

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EGF Receptor Gene Mutations Are Common in Lung Cancers from "Never Smokers" and Are Associated with Sensitivity of Tumors to Gefitinib and Erlotinib

Proceedings of the National Academy of Sciences - PNAS, 2004-09, Vol.101 (36), p.13306-13311

2004

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A New Initiative on Precision Medicine

New Era in Cancer Research

Three decades of Wnts: a personal perspective on how a scientific field developed

Toward a Shared Vision for Cancer Genomic Data

Insurance for broad genomic tests in oncology

Of oncogenes and open science: an interview with Harold Varmus

Rescuing US biomedical research from its systemic flaws

Sensitivity of human lung adenocarcinoma cell lines to targeted inhibition of BET epigenetic signaling proteins

KRAS mutations and primary resistance of lung adenocarcinomas to gefitinib or erlotinib

Lung Cancer Signatures in Plasma Based on Proteome Profiling of Mouse Tumor Models

Evidence that synthetic lethality underlies the mutual exclusivity of oncogenic KRAS and EGFR mutations in lung adenocarcinoma

Hyperactivation of ERK by multiple mechanisms is toxic to RTK-RAS mutation-driven lung adenocarcinoma cells

Arlen Specter: A retrospective

Epstein-Barr virus: an important vaccine target for cancer prevention

Generation of pulmonary neuroendocrine cells and SCLC-like tumors from human embryonic stem cells

Improving support for young biomedical scientists

Lung adenocarcinomas induced in mice by mutant EGF receptors found in human lung cancers respond to a tyrosine kinase inhibitor or to down-regulation of the receptors

Superoxide dismutase 1 (SOD1) is a target for a small molecule identified in a screen for inhibitors of the growth of lung adenocarcinoma cell lines

Wild-Type U2AF1 Antagonizes the Splicing Program Characteristic of U2AF1-Mutant Tumors and Is Required for Cell Survival

Seeding and Propagation of Untransformed Mouse Mammary Cells in the Lung

Opinion: Addressing systemic problems in the biomedical research enterprise

Oncogene cooperation in tumor maintenance and tumor recurrence in mouse mammary tumors induced by Myc and mutant Kras

Acquired resistance of lung adenocarcinomas to gefitinib or erlotinib is associated with a second mutation in the EGFR kinase domain

Threatening the Global AIDS Response — Obstacles to PEPFAR’s Reauthorization

EGF Receptor Gene Mutations Are Common in Lung Cancers from "Never Smokers" and Are Associated with Sensitivity of Tumors to Gefitinib and Erlotinib

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