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Propionate and Alzheimer's Disease

Frontiers in aging neuroscience, 2021-01, Vol.12, p.580001-580001

2021

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The Immune System as a Therapeutic Target for Alzheimer’s Disease

Life (Basel, Switzerland), 2022-09, Vol.12 (9), p.1440

2022

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Luteolin reduces Alzheimer's disease pathologies induced by traumatic brain injury

International journal of molecular sciences, 2014-01, Vol.15 (1), p.895-904

2014

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Retraction Note to: LISPRO mitigates β-amyloid and associated pathologies in Alzheimer's mice

Cell death & disease, 2021-12, Vol.12 (12), p.1142-1142, Article 1142

2021

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Biodistribution of Infused Human Umbilical Cord Blood Cells in Alzheimer's Disease-Like Murine Model

Cell transplantation, 2016-01, Vol.25 (1), p.195-199

2016

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Human Cord Blood Serum-Derived APP α-Secretase Cleavage Activity is Mediated by C1 Complement

Cell transplantation, 2018-04, Vol.27 (4), p.666-676

2018

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Vasoactive intestinal peptide : cardiovascular effects

Cardiovascular research, 2001, Vol.49 (1), p.27-37

2001

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High-energy compounds promote physiological processing of Alzheimer's amyloid-β precursor protein and boost cell survival in culture

Journal of neurochemistry, 2012-11, Vol.123 (4), p.525-531

2012

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Eukaryotic initiation factor 3, subunit C silencing inhibits cell proliferation and promotes apoptosis in human ovarian cancer cells

Bioscience reports, 2019-08, Vol.39 (8), p.1

2019

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Therapeutic Cocktail Approach for Treatment of Hyperhomocysteinemia in Alzheimer’s Disease

Cell Medicine, 2018-01, Vol.10, p.215517901772228-2155179017722280

2018

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Human Umbilical Cord Blood-Derived Monocytes Improve Cognitive Deficits and Reduce Amyloid-β Pathology in PSAPP Mice

Cell transplantation, 2015-01, Vol.24 (11), p.2237-2250

2015

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Scientific truth or false hope? Understanding Alzheimer's disease from an aging perspective

Journal of Alzheimer's disease, 2011-01, Vol.24 (1), p.3

2011

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Human Umbilical Cord Blood Serum–derived α-Secretase

Cell transplantation, 2018-03, Vol.27 (3), p.438-455

2018

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RETRACTED ARTICLE:LISPRO mitigates β-amyloid and associated pathologies in Alzheimer’s mice

Cell death & disease, 2017-06, Vol.8 (6), p.e2880-e2880

2017

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Beneficial effects of a pyrroloquinolinequinone-containing dietary formulation on motor deficiency, cognitive decline and mitochondrial dysfunction in a mouse model of Alzheimer’s disease

Heliyon, 2017-04, Vol.3 (4), p.e00279-e00279, Article e00279

2017

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Gallic acid is a dual α/β-secretase modulator that reverses cognitive impairment and remediates pathology in Alzheimer mice

The Journal of biological chemistry, 2020-11, Vol.295 (48), p.16251-16266

2020

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Targeting apolipoprotein E and N-terminal amyloid β-protein precursor interaction improves cognition and reduces amyloid pathology in Alzheimer’s mice

The Journal of biological chemistry, 2023-07, Vol.299 (7), p.104846-104846, Article 104846

2023

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Restoring Soluble Amyloid Precursor Protein α Functions as a Potential Treatment for A lzheimer's Disease

Journal of neuroscience research, 2017-04, Vol.95 (4), p.973-991

2017

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Restoring Soluble Amyloid Precursor Protein [alpha] Functions as a Potential Treatment for Alzheimer's Disease

Journal of neuroscience research, 2017-04, Vol.95 (4), p.973

2017

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Autism, heparan sulfate and potential interventions

Experimental neurology, 2022-07, Vol.353, p.114050-114050, Article 114050

2022

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Restoring sAPPα functions as a potential treatment for Alzheimer’s disease

Journal of neuroscience research, 2016-08, Vol.95 (4), p.973-991

2016

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C1q/TNF‐related protein 9: A novel therapeutic target in ischemic stroke?

Journal of neuroscience research, 2019-02, Vol.97 (2), p.128-136

2019

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Soluble amyloid precursor protein alpha inhibits tau phosphorylation through modulation of GSK3[beta] signaling pathway

Journal of neurochemistry, 2015-11, Vol.135 (3), p.630

2015

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Diosmin reduces cerebral Aβ levels, tau hyperphosphorylation, neuroinflammation, and cognitive impairment in the 3xTg-AD mice

Journal of neuroimmunology, 2016-10, Vol.299, p.98-106

2016

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A Novel Apolipoprotein E Antagonist Functionally Blocks Apolipoprotein E Interaction With N-terminal Amyloid Precursor Protein, Reduces β-Amyloid-Associated Pathology, and Improves Cognition

Biological psychiatry (1969), 2019-08, Vol.86 (3), p.208-220

2019

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Propionate and Alzheimer's Disease

The Immune System as a Therapeutic Target for Alzheimer’s Disease

Luteolin reduces Alzheimer's disease pathologies induced by traumatic brain injury

Retraction Note to: LISPRO mitigates β-amyloid and associated pathologies in Alzheimer's mice

Biodistribution of Infused Human Umbilical Cord Blood Cells in Alzheimer's Disease-Like Murine Model

Human Cord Blood Serum-Derived APP α-Secretase Cleavage Activity is Mediated by C1 Complement

Vasoactive intestinal peptide : cardiovascular effects

High-energy compounds promote physiological processing of Alzheimer's amyloid-β precursor protein and boost cell survival in culture

Eukaryotic initiation factor 3, subunit C silencing inhibits cell proliferation and promotes apoptosis in human ovarian cancer cells

Therapeutic Cocktail Approach for Treatment of Hyperhomocysteinemia in Alzheimer’s Disease

Human Umbilical Cord Blood-Derived Monocytes Improve Cognitive Deficits and Reduce Amyloid-β Pathology in PSAPP Mice

Scientific truth or false hope? Understanding Alzheimer's disease from an aging perspective

Human Umbilical Cord Blood Serum–derived α-Secretase

RETRACTED ARTICLE:LISPRO mitigates β-amyloid and associated pathologies in Alzheimer’s mice

Beneficial effects of a pyrroloquinolinequinone-containing dietary formulation on motor deficiency, cognitive decline and mitochondrial dysfunction in a mouse model of Alzheimer’s disease

Gallic acid is a dual α/β-secretase modulator that reverses cognitive impairment and remediates pathology in Alzheimer mice

Targeting apolipoprotein E and N-terminal amyloid β-protein precursor interaction improves cognition and reduces amyloid pathology in Alzheimer’s mice

Restoring Soluble Amyloid Precursor Protein α Functions as a Potential Treatment for A lzheimer's Disease

Restoring Soluble Amyloid Precursor Protein [alpha] Functions as a Potential Treatment for Alzheimer's Disease

Autism, heparan sulfate and potential interventions

Restoring sAPPα functions as a potential treatment for Alzheimer’s disease

C1q/TNF‐related protein 9: A novel therapeutic target in ischemic stroke?

Soluble amyloid precursor protein alpha inhibits tau phosphorylation through modulation of GSK3[beta] signaling pathway

Diosmin reduces cerebral Aβ levels, tau hyperphosphorylation, neuroinflammation, and cognitive impairment in the 3xTg-AD mice

A Novel Apolipoprotein E Antagonist Functionally Blocks Apolipoprotein E Interaction With N-terminal Amyloid Precursor Protein, Reduces β-Amyloid-Associated Pathology, and Improves Cognition

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