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A new long term in vitro model of myelination

Experimental cell research, 2011-10, Vol.317 (16), p.2374-2383

2011

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Aβ oligomers reduce neuronal plasticity in an aged animal model of Alzheimer’s disease

Alzheimer's & dementia, 2023-12, Vol.19 (S13)

2023

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SITE-SPECIFIC HYPERPHOSPHORYLATION OF TAU BY Aβ PEPTIDE AND GLUTAMATE IN A PRIMARY CULTURE OF CORTICAL NEURONS

Alzheimer's & dementia, 2019-07, Vol.15 (7), p.P1335-P1335

2019

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P4‐159: SITE‐SPECIFIC HYPERPHOSPHORYLATION OF TAU BY Aβ PEPTIDE AND GLUTAMATE IN A PRIMARY CULTURE OF CORTICAL NEURONS

Alzheimer's & dementia, 2019-07, Vol.15 (7S_Part_26), p.P1335-P1335

2019

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Neurotoxicity and hyperphosphorylation of tau protein induced by the amyloid peptide oligomers involve different glutamate dependent and independent mechanisms

Alzheimer's & dementia, 2020-12, Vol.16, p.n/a

2020

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Neurotoxicity and hyperphosphorylation of tau protein induced by the amyloid peptide oligomers involve different glutamate dependent and independent mechanisms: Nonhuman/Target identification and validation studies: Amyloid

Alzheimer's & dementia, 2020-12, Vol.16 (S9)

2020

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Neuroinflammation and microglial activation as target for therapeutic strategy: in vitro and in vivo models of study

Alzheimer's & dementia, 2022-12, Vol.18 (S3), p.n/a

2022

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Glutamate protects neuromuscular junctions from deleterious effects of β-amyloid peptide and conversely: An in vitro study in a nerve-muscle coculture

Journal of neuroscience research, 2015-04, Vol.93 (4), p.633-643

2015

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Neuroinflammation induced by amyloid beta 1‐42 oligomers contributes to neuronal loss in an in vitro model of Alzheimer’s disease

Alzheimer's & dementia, 2021-12, Vol.17, p.e054212-n/a

2021

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Glutamate protects neuromuscular junctions from deleterious effects of β-amyloid peptide and conversely: An in vitro study in a nerve-muscle coculture: Interconnection Between Glutamate and Aβ on NMJs

Journal of neuroscience research, 2015-04, Vol.93 (4), p.633-643

2015

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Glutamate protects neuromuscular junctions from deleterious effects of [beta]-amyloid peptide and conversely: An in vitro study in a nerve-muscle coculture

Journal of neuroscience research, 2015-04, Vol.93 (4), p.633

2015

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Uptake of Aβ 1–40- and Aβ 1–42-coated yeast by microglial cells: a role for LRP

Journal of leukocyte biology, 2004-08, Vol.76 (2), p.451-461

2004

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Operational dissection of [beta]-amyloid cytopathic effects on cultured neurons

Journal of neuroscience research, 2013-05, Vol.91 (5), p.706-716

2013

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Operational dissection of β-amyloid cytopathic effects on cultured neurons

Journal of neuroscience research, 2013-05, Vol.91 (5), p.706-716

2013

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EFFECTS OF CIRCULATING AMYLOID PEPTIDE ON BBB INTEGRITY, CONSEQUENCES ON THE NEURODEGENERATION PROCESS

Alzheimer's & dementia, 2017-07, Vol.13 (7), p.P315-P316

2017

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Progressive motor neuron impairment in an animal model of familial amyotrophic lateral sclerosis

Muscle & nerve, 1997-01, Vol.20 (1), p.45-51

1997

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Cytotoxicity of peritoneal murine macrophages against encapsulated pancreatic rat islets: in vivo and in vitro studies

Journal of leukocyte biology, 1996-12, Vol.60 (6), p.729-736

1996

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Modulation by prednisolone of calcium handling in skeletal muscle cells

British journal of pharmacology, 1995-12, Vol.116 (7), p.2811-2816

1995

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Enhancement of Mouse Sciatic Nerve Regeneration by the Long Chain Fatty Alcohol,N-Hexacosanol

Experimental neurology, 1996-04, Vol.138 (2), p.189-197

1996

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Complement receptor 3 (CD11b/CD18) is implicated in the elimination of β-amyloid peptides

Fundamental & clinical pharmacology, 2011-02, Vol.25 (1), p.115-122

2011

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Complement receptor 3 (CD11b/CD18) is implicated in the elimination of β-amyloid peptides: CR3 and β-amyloid peptides phagocytosis

Fundamental & clinical pharmacology, 2011-02, Vol.25 (1), p.115-122

2011

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Comparing reagents for efficient transfection of human primary myoblasts: FuGENE 6, Effectene and ExGen 500

Fundamental & clinical pharmacology, 2006-02, Vol.20 (1), p.81-89

2006

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Progressive motor neuron impairment in an animal model of familial amyotrophic lateral sclerosis

Muscle & nerve, 1997-01, Vol.20 (1), p.45-51

1997

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Possible pathogenic role of muscle cell dysfunction in motor neuron death in spinal muscular atrophy

Muscle & nerve, 2002-05, Vol.25 (5), p.700-708

2002

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Uptake of Abeta 1-40- and Abeta 1-42-coated yeast by microglial cells: a role for LRP

Journal of leukocyte biology, 2004-08, Vol.76 (2), p.451-461

2004

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A new long term in vitro model of myelination

Aβ oligomers reduce neuronal plasticity in an aged animal model of Alzheimer’s disease

SITE-SPECIFIC HYPERPHOSPHORYLATION OF TAU BY Aβ PEPTIDE AND GLUTAMATE IN A PRIMARY CULTURE OF CORTICAL NEURONS

P4‐159: SITE‐SPECIFIC HYPERPHOSPHORYLATION OF TAU BY Aβ PEPTIDE AND GLUTAMATE IN A PRIMARY CULTURE OF CORTICAL NEURONS

Neurotoxicity and hyperphosphorylation of tau protein induced by the amyloid peptide oligomers involve different glutamate dependent and independent mechanisms

Neurotoxicity and hyperphosphorylation of tau protein induced by the amyloid peptide oligomers involve different glutamate dependent and independent mechanisms: Nonhuman/Target identification and validation studies: Amyloid

Neuroinflammation and microglial activation as target for therapeutic strategy: in vitro and in vivo models of study

Glutamate protects neuromuscular junctions from deleterious effects of β-amyloid peptide and conversely: An in vitro study in a nerve-muscle coculture

Neuroinflammation induced by amyloid beta 1‐42 oligomers contributes to neuronal loss in an in vitro model of Alzheimer’s disease

Glutamate protects neuromuscular junctions from deleterious effects of β-amyloid peptide and conversely: An in vitro study in a nerve-muscle coculture: Interconnection Between Glutamate and Aβ on NMJs

Glutamate protects neuromuscular junctions from deleterious effects of [beta]-amyloid peptide and conversely: An in vitro study in a nerve-muscle coculture

Uptake of Aβ 1–40- and Aβ 1–42-coated yeast by microglial cells: a role for LRP

Operational dissection of [beta]-amyloid cytopathic effects on cultured neurons

Operational dissection of β-amyloid cytopathic effects on cultured neurons

EFFECTS OF CIRCULATING AMYLOID PEPTIDE ON BBB INTEGRITY, CONSEQUENCES ON THE NEURODEGENERATION PROCESS

Progressive motor neuron impairment in an animal model of familial amyotrophic lateral sclerosis

Cytotoxicity of peritoneal murine macrophages against encapsulated pancreatic rat islets: in vivo and in vitro studies

Modulation by prednisolone of calcium handling in skeletal muscle cells

Enhancement of Mouse Sciatic Nerve Regeneration by the Long Chain Fatty Alcohol,N-Hexacosanol

Complement receptor 3 (CD11b/CD18) is implicated in the elimination of β-amyloid peptides

Complement receptor 3 (CD11b/CD18) is implicated in the elimination of β-amyloid peptides: CR3 and β-amyloid peptides phagocytosis

Comparing reagents for efficient transfection of human primary myoblasts: FuGENE 6, Effectene and ExGen 500

Progressive motor neuron impairment in an animal model of familial amyotrophic lateral sclerosis

Possible pathogenic role of muscle cell dysfunction in motor neuron death in spinal muscular atrophy

Uptake of Abeta 1-40- and Abeta 1-42-coated yeast by microglial cells: a role for LRP

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