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Neuroinflammation and M2 microglia: the good, the bad, and the inflamed

Journal of neuroinflammation, 2014-06, Vol.11 (1), p.98-98, Article 98

2014

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Exploiting microglial and peripheral immune cell crosstalk to treat Alzheimer's disease

Journal of neuroinflammation, 2019-04, Vol.16 (1), p.74-13, Article 74

2019

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Galactic cosmic radiation leads to cognitive impairment and increased aβ plaque accumulation in a mouse model of Alzheimer's disease

PloS one, 2012-12, Vol.7 (12), p.e53275

2012

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Osteoarthritis accelerates and exacerbates Alzheimer's disease pathology in mice

Journal of neuroinflammation, 2011-09, Vol.8 (1), p.112-112

2011

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IL-1β-driven amyloid plaque clearance is associated with an expansion of transcriptionally reprogrammed microglia

Journal of neuroinflammation, 2019-12, Vol.16 (1), p.261-261, Article 261

2019

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Gas6 induces inflammation and reduces plaque burden but worsens behavior in a sex-dependent manner in the APP/PS1 model of Alzheimer's disease

Journal of neuroinflammation, 2022-02, Vol.19 (1), p.38-38, Article 38

2022

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Correction: Gas6 induces infammation and reduces plaque burden but worsens behavior in a sex-dependent manner in the APP/PS1 model of Alzheimer’s disease

Journal of neuroinflammation, 2023-11, Vol.20 (1), p.1-254, Article 254

2023

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Inconsistent Effects of Glatiramer Acetate Treatment in the 5xFAD Mouse Model of Alzheimer's Disease

Pharmaceutics, 2023-06, Vol.15 (7), p.1809

2023

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Are "resting" microglia more "m2"?

Frontiers in immunology, 2014, Vol.5, p.594-594

2014

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Aryl hydrocarbon receptor deletion in cerebellar granule neuron precursors impairs neurogenesis

Developmental neurobiology (Hoboken, N.J.), 2016-05, Vol.76 (5), p.533-550

2016

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Characterization of binge-dosed methamphetamine-induced neurotoxicity and neuroinflammation

Neurotoxicology (Park Forest South), 2015-09, Vol.50, p.131-141

2015

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Evaluating the Effect of Interleukin-4 in the 3xTg Mouse Model of Alzheimer’s Disease

Frontiers in neuroscience, 2020-05, Vol.14, p.441-441

2020

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Evaluating Effects of Glatiramer Acetate Treatment on Amyloid Deposition and Tau Phosphorylation in the 3xTg Mouse Model of Alzheimer’s Disease

Frontiers in neuroscience, 2021-10, Vol.15, p.758677-758677

2021

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Neurogenic Effects of Low-Dose Whole-Body HZE (Fe) Ion and Gamma Irradiation

Radiation research, 2016-12, Vol.186 (6), p.614

2016

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Interleukin-1β mediated amyloid plaque clearance is independent of CCR2 signaling in the APP/PS1 mouse model of Alzheimer's disease

Neurobiology of disease, 2014-09, Vol.69, p.124-133

2014

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Sustained interleukin-1β overexpression exacerbates tau pathology despite reduced amyloid burden in an Alzheimer's mouse model

The Journal of neuroscience, 2013-03, Vol.33 (11), p.5053-5064

2013

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Fractionation Spares Mice From Radiation-Induced Reductions in Weight Gain But Does Not Prevent Late Oligodendrocyte Lineage Side Effects

International journal of radiation oncology, biology, physics, 2016-10, Vol.96 (2), p.449-457

2016

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Arginase 1+ microglia reduce Aβ plaque deposition during IL-1β-dependent neuroinflammation

Journal of neuroinflammation, 2015-11, Vol.12 (1), p.203-203, Article 203

2015

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Pharmacologic Manipulation of Complement Receptor 3 Prevents Dendritic Spine Loss and Cognitive Impairment After Acute Cranial Radiation

International journal of radiation oncology, biology, physics, 2023-12

2023

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Peripheral blood mononuclear cell infiltration and neuroinflammation in the HexB−/− mouse model of neurodegeneration

Journal of neuroimmunology, 2008-10, Vol.203 (1), p.50-57

2008

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Cranial irradiation mediated spine loss is sex-specific and complement receptor-3 dependent in male mice

Scientific reports, 2019-12, Vol.9 (1), p.18899-12, Article 18899

2019

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Sustained hippocampal IL-1 beta overexpression mediates chronic neuroinflammation and ameliorates Alzheimer plaque pathology

The Journal of clinical investigation, 2007-06, Vol.117 (6), p.1595-1604

2007

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Cranial irradiation acutely and persistently impairs injury-induced microglial proliferation

Brain, behavior, & immunity. Health, 2020-04, Vol.4, p.100057-100057, Article 100057

2020

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Fractionation enhances acute oligodendrocyte progenitor cell radiation sensitivity and leads to long term depletion

Glia, 2018-04, Vol.66 (4), p.846-861

2018

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Cyclooxygenase inhibition as a Strategy to Ameliorate Brain Injury

Journal of neurotrauma, 2002, Vol.19 (1), p.1-15

2002

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Neuroinflammation and M2 microglia: the good, the bad, and the inflamed

Exploiting microglial and peripheral immune cell crosstalk to treat Alzheimer's disease

Galactic cosmic radiation leads to cognitive impairment and increased aβ plaque accumulation in a mouse model of Alzheimer's disease

Osteoarthritis accelerates and exacerbates Alzheimer's disease pathology in mice

IL-1β-driven amyloid plaque clearance is associated with an expansion of transcriptionally reprogrammed microglia

Gas6 induces inflammation and reduces plaque burden but worsens behavior in a sex-dependent manner in the APP/PS1 model of Alzheimer's disease

Correction: Gas6 induces infammation and reduces plaque burden but worsens behavior in a sex-dependent manner in the APP/PS1 model of Alzheimer’s disease

Inconsistent Effects of Glatiramer Acetate Treatment in the 5xFAD Mouse Model of Alzheimer's Disease

Are "resting" microglia more "m2"?

Aryl hydrocarbon receptor deletion in cerebellar granule neuron precursors impairs neurogenesis

Characterization of binge-dosed methamphetamine-induced neurotoxicity and neuroinflammation

Evaluating the Effect of Interleukin-4 in the 3xTg Mouse Model of Alzheimer’s Disease

Evaluating Effects of Glatiramer Acetate Treatment on Amyloid Deposition and Tau Phosphorylation in the 3xTg Mouse Model of Alzheimer’s Disease

Neurogenic Effects of Low-Dose Whole-Body HZE (Fe) Ion and Gamma Irradiation

Interleukin-1β mediated amyloid plaque clearance is independent of CCR2 signaling in the APP/PS1 mouse model of Alzheimer's disease

Sustained interleukin-1β overexpression exacerbates tau pathology despite reduced amyloid burden in an Alzheimer's mouse model

Fractionation Spares Mice From Radiation-Induced Reductions in Weight Gain But Does Not Prevent Late Oligodendrocyte Lineage Side Effects

Arginase 1+ microglia reduce Aβ plaque deposition during IL-1β-dependent neuroinflammation

Pharmacologic Manipulation of Complement Receptor 3 Prevents Dendritic Spine Loss and Cognitive Impairment After Acute Cranial Radiation

Peripheral blood mononuclear cell infiltration and neuroinflammation in the HexB−/− mouse model of neurodegeneration

Cranial irradiation mediated spine loss is sex-specific and complement receptor-3 dependent in male mice

Sustained hippocampal IL-1 beta overexpression mediates chronic neuroinflammation and ameliorates Alzheimer plaque pathology

Cranial irradiation acutely and persistently impairs injury-induced microglial proliferation

Fractionation enhances acute oligodendrocyte progenitor cell radiation sensitivity and leads to long term depletion

Cyclooxygenase inhibition as a Strategy to Ameliorate Brain Injury

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