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Correction to: Comment on "ILC1 drive intestinal epithelial and matrix remodeling"

Mucosal immunology, 2022-03, Vol.15 (3), p.530-530

2022

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Comment on "ILC1 drive intestinal epithelial and matrix remodeling"

Mucosal immunology, 2021-03, Vol.14 (2), p.279-281

2021

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Oncogenetic landscape of lymphomagenesis in coeliac disease

Gut, 2022-03, Vol.71 (3), p.497-508

2022

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Cytotoxic innate intraepithelial lymphocytes control early stages of Cryptosporidium infection

Frontiers in immunology, 2023-09, Vol.14, p.1229406-1229406

2023

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Gastrointestinal Disorder Associated with Olmesartan Mimics Autoimmune Enteropathy

PloS one, 2015-06, Vol.10 (6), p.e0125024-e0125024

2015

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Targeting CCR4 with mogamulizumab in refractory CD3-CD4+ lymphocytic-variant hypereosinophilic syndrome

Haematologica (Roma), 2024-06

2024

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Severe SARS‐CoV‐2 patients develop a higher specific T‐cell response

Clinical & translational immunology, 2020, Vol.9 (12), p.e1217-n/a

2020

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Celiac Disease: An Immunological Jigsaw

Immunity (Cambridge, Mass.), 2012-06, Vol.36 (6), p.907-919

2012

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The role of animal models in unravelling therapeutic targets in coeliac disease

Baillière's best practice & research. Clinical gastroenterology, 2015-06, Vol.29 (3), p.437-450

2015

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Coeliac disease &gluten sensitivity: Epithelial stress enters the dance in coeliac disease

Nature reviews. Gastroenterology & hepatology, 2015-09, Vol.12 (9), p.491

2015

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Refractory celiac disease: from bench to bedside

Seminars in immunopathology, 2012-07, Vol.34 (4), p.601-613

2012

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Epithelial stress enters the dance in coeliac disease

Nature reviews. Gastroenterology & hepatology, 2015-09, Vol.12 (9), p.491-492

2015

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Interleukin-15-Dependent T-Cell-like Innate Intraepithelial Lymphocytes Develop in the Intestine and Transform into Lymphomas in Celiac Disease

Immunity (Cambridge, Mass.), 2016-09, Vol.45 (3), p.610-625

2016

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Mogamulizumab for Refractory CD3-CD4+ Lymphocytic-Variant Hypereosinophilic Syndrome

Blood, 2023-11, Vol.142 (Supplement 1), p.4444-4444

2023

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IL-15 triggers an antiapoptotic pathway in human intraepithelial lymphocytes that is a potential new target in celiac disease-associated inflammation and lymphomagenesis

The Journal of clinical investigation, 2010-06, Vol.120 (6), p.2131-2143

2010

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Interleukin-15, a Master Piece in the Immunological Jigsaw of Celiac Disease

Digestive diseases (Basel), 2015-01, Vol.33 (2), p.122-130

2015

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Interleukin 15 and CD4+ T Cells Cooperate to Promote Small Intestinal Enteropathy in Response to Dietary Antigen

Gastroenterology (New York, N.Y. 1943), 2014-04, Vol.146 (4), p.1017-1027

2014

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Enteropathy associated T cell lymphoma in celiac disease: A large retrospective study

Digestive and liver disease, 2013-05, Vol.45 (5), p.377-384

2013

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Impaired Control of Effector T Cells by Regulatory T Cells: A Clue to Loss of Oral Tolerance and Autoimmunity in Celiac Disease?

The American journal of gastroenterology, 2012-04, Vol.107 (4), p.604-611

2012

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Coordinated Induction by IL15 of a TCR-Independent NKG2D Signaling Pathway Converts CTL into Lymphokine-Activated Killer Cells in Celiac Disease

Immunity (Cambridge, Mass.), 2004-09, Vol.21 (3), p.357-366

2004

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Presentation and Long-Term Follow-up of Refractory Celiac Disease: Comparison of Type I With Type II

Gastroenterology (New York, N.Y. 1943), 2009, Vol.136 (1), p.81-90

2009

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Interleukin-15-Dependent NKp46+ Innate Lymphoid Cells Control Intestinal Inflammation by Recruiting Inflammatory Monocytes

Immunity (Cambridge, Mass.), 2012-07, Vol.37 (1), p.108-121

2012

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Innate T cell responses in human gut

Seminars in immunology, 2009-06, Vol.21 (3), p.121-129

2009

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Inhibition of TGF-β Signaling by IL-15: A New Role for IL-15 in the Loss of Immune Homeostasis in Celiac Disease

Gastroenterology (New York, N.Y. 1943), 2007-03, Vol.132 (3), p.994-1008

2007

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Correction to: Comment on "ILC1 drive intestinal epithelial and matrix remodeling"

Comment on "ILC1 drive intestinal epithelial and matrix remodeling"

Oncogenetic landscape of lymphomagenesis in coeliac disease

Cytotoxic innate intraepithelial lymphocytes control early stages of Cryptosporidium infection

Gastrointestinal Disorder Associated with Olmesartan Mimics Autoimmune Enteropathy

Targeting CCR4 with mogamulizumab in refractory CD3-CD4+ lymphocytic-variant hypereosinophilic syndrome

Severe SARS‐CoV‐2 patients develop a higher specific T‐cell response

Celiac Disease: An Immunological Jigsaw

The role of animal models in unravelling therapeutic targets in coeliac disease

Coeliac disease &gluten sensitivity: Epithelial stress enters the dance in coeliac disease

Refractory celiac disease: from bench to bedside

Epithelial stress enters the dance in coeliac disease

Interleukin-15-Dependent T-Cell-like Innate Intraepithelial Lymphocytes Develop in the Intestine and Transform into Lymphomas in Celiac Disease

Mogamulizumab for Refractory CD3-CD4+ Lymphocytic-Variant Hypereosinophilic Syndrome

IL-15 triggers an antiapoptotic pathway in human intraepithelial lymphocytes that is a potential new target in celiac disease-associated inflammation and lymphomagenesis

Interleukin-15, a Master Piece in the Immunological Jigsaw of Celiac Disease

Interleukin 15 and CD4+ T Cells Cooperate to Promote Small Intestinal Enteropathy in Response to Dietary Antigen

Enteropathy associated T cell lymphoma in celiac disease: A large retrospective study

Impaired Control of Effector T Cells by Regulatory T Cells: A Clue to Loss of Oral Tolerance and Autoimmunity in Celiac Disease?

Coordinated Induction by IL15 of a TCR-Independent NKG2D Signaling Pathway Converts CTL into Lymphokine-Activated Killer Cells in Celiac Disease

Presentation and Long-Term Follow-up of Refractory Celiac Disease: Comparison of Type I With Type II

Interleukin-15-Dependent NKp46+ Innate Lymphoid Cells Control Intestinal Inflammation by Recruiting Inflammatory Monocytes

NKp46 is a diagnostic biomarker and may be a therapeutic target in gastrointestinal T-cell lymphoproliferative diseases: a CELAC study

Innate T cell responses in human gut

Inhibition of TGF-β Signaling by IL-15: A New Role for IL-15 in the Loss of Immune Homeostasis in Celiac Disease

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