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Myeloproliferative neoplasms: from origins to outcomes

Blood, 2017-12, Vol.130 (23), p.2475-2483

2017

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Combined Single-Cell Functional and Gene Expression Analysis Resolves Heterogeneity within Stem Cell Populations

Cell stem cell, 2015-06, Vol.16 (6), p.712-724

2015

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Molecular determinants of pathogenesis and clinical phenotype in myeloproliferative neoplasms

Haematologica (Roma), 2017-01, Vol.102 (1), p.7-17

2017

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Notch Signaling Mediates Secondary Senescence

Cell reports (Cambridge), 2019-04, Vol.27 (4), p.997-1007.e5

2019

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Genome-wide Analysis of Simultaneous GATA1/2, RUNX1, FLI1, and SCL Binding in Megakaryocytes Identifies Hematopoietic Regulators

Developmental cell, 2011-05, Vol.20 (5), p.597-609

2011

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Subclonal phylogenetic structures in cancer revealed by ultra-deep sequencing

Proceedings of the National Academy of Sciences - PNAS, 2008-09, Vol.105 (35), p.13081-13086

2008

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Self-renewal of single mouse hematopoietic stem cells is reduced by JAK2V617F without compromising progenitor cell expansion

PLoS biology, 2013-06, Vol.11 (6), p.e1001576

2013

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JAK2V617F leads to intrinsic changes in platelet formation and reactivity in a knock-in mouse model of essential thrombocythemia

Blood, 2013-11, Vol.122 (23), p.3787-3797

2013

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Two routes to leukemic transformation after a JAK2 mutation–positive myeloproliferative neoplasm

Blood, 2010-04, Vol.115 (14), p.2891-2900

2010

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MPL mutations in myeloproliferative disorders: analysis of the PT-1 cohort

Blood, 2008-07, Vol.112 (1), p.141-149

2008

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JAK2 V617F impairs hematopoietic stem cell function in a conditional knock-in mouse model of JAK2 V617F–positive essential thrombocythemia

Blood, 2010-09, Vol.116 (9), p.1528-1538

2010

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Proliferation Drives Aging-Related Functional Decline in a Subpopulation of the Hematopoietic Stem Cell Compartment

Cell reports (Cambridge), 2017-05, Vol.19 (8), p.1503-1511

2017

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Inactivating CUX1 mutations promote tumorigenesis

Nature genetics, 2014-01, Vol.46 (1), p.33

2014

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Bone marrow pathology in essential thrombocythemia: interobserver reliability and utility for identifying disease subtypes

Blood, 2008-01, Vol.111 (1), p.60-70

2008

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JAK2V617F homozygosity drives a phenotypic switch in myeloproliferative neoplasms, but is insufficient to sustain disease

Blood, 2014-05, Vol.123 (20), p.3139-3151

2014

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SC3: consensus clustering of single-cell RNA-seq data

Nature methods, 2017-05, Vol.14 (5), p.483

2017

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Single-cell approaches identify the molecular network driving malignant hematopoietic stem cell self-renewal

Blood, 2018-08, Vol.132 (8), p.791-803

2018

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How I treat essential thrombocythemia

Blood, 2011-02, Vol.117 (5), p.1472-1482

2011

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Mouse models of myeloproliferative neoplasms: JAK of all grades

Disease models & mechanisms, 2011-05, Vol.4 (3), p.311-317

2011

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Gata2, Fli1, and Scl form a recursively wired gene-regulatory circuit during early hematopoietic development

Proceedings of the National Academy of Sciences - PNAS, 2007-11, Vol.104 (45), p.17692-17697

2007

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JAK2V617F promotes replication fork stalling with disease-restricted impairment of the intra-S checkpoint response

Proceedings of the National Academy of Sciences - PNAS, 2014-10, Vol.111 (42), p.15190-15195

2014

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Clonal heterogeneity as a driver of disease variability in the evolution of myeloproliferative neoplasms

Experimental hematology, 2014-10, Vol.42 (10), p.841-851

2014

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Clonal approaches to understanding the impact of mutations on hematologic disease development

Blood, 2019-03, Vol.133 (13), p.1436-1445

2019

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RECQL5 Suppresses Oncogenic JAK2-Induced Replication Stress and Genomic Instability

Cell reports (Cambridge), 2015-12, Vol.13 (11), p.2345-2352

2015

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Myeloproliferative neoplasms: from origins to outcomes

Combined Single-Cell Functional and Gene Expression Analysis Resolves Heterogeneity within Stem Cell Populations

Molecular determinants of pathogenesis and clinical phenotype in myeloproliferative neoplasms

A Kinase-Independent Function of CDK6 Links the Cell Cycle to Tumor Angiogenesis

Notch Signaling Mediates Secondary Senescence

Genome-wide Analysis of Simultaneous GATA1/2, RUNX1, FLI1, and SCL Binding in Megakaryocytes Identifies Hematopoietic Regulators

Subclonal phylogenetic structures in cancer revealed by ultra-deep sequencing

Self-renewal of single mouse hematopoietic stem cells is reduced by JAK2V617F without compromising progenitor cell expansion

JAK2V617F leads to intrinsic changes in platelet formation and reactivity in a knock-in mouse model of essential thrombocythemia

Two routes to leukemic transformation after a JAK2 mutation–positive myeloproliferative neoplasm

MPL mutations in myeloproliferative disorders: analysis of the PT-1 cohort

JAK2 V617F impairs hematopoietic stem cell function in a conditional knock-in mouse model of JAK2 V617F–positive essential thrombocythemia

Proliferation Drives Aging-Related Functional Decline in a Subpopulation of the Hematopoietic Stem Cell Compartment

Inactivating CUX1 mutations promote tumorigenesis

Bone marrow pathology in essential thrombocythemia: interobserver reliability and utility for identifying disease subtypes

JAK2V617F homozygosity drives a phenotypic switch in myeloproliferative neoplasms, but is insufficient to sustain disease

SC3: consensus clustering of single-cell RNA-seq data

Single-cell approaches identify the molecular network driving malignant hematopoietic stem cell self-renewal

How I treat essential thrombocythemia

Mouse models of myeloproliferative neoplasms: JAK of all grades

Gata2, Fli1, and Scl form a recursively wired gene-regulatory circuit during early hematopoietic development

JAK2V617F promotes replication fork stalling with disease-restricted impairment of the intra-S checkpoint response

Clonal heterogeneity as a driver of disease variability in the evolution of myeloproliferative neoplasms

Clonal approaches to understanding the impact of mutations on hematologic disease development

RECQL5 Suppresses Oncogenic JAK2-Induced Replication Stress and Genomic Instability

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