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Capillary K + -sensing initiates retrograde hyperpolarization to increase local cerebral blood flow

Nature neuroscience, 2017-05, Vol.20 (5), p.717

2017

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Contractile pericytes determine the direction of blood flow at capillary junctions

Proceedings of the National Academy of Sciences - PNAS, 2020-10, Vol.117 (43), p.27022-27033

2020

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Endothelium-Dependent Cerebral Artery Dilation Mediated by TRPA1 and Ca2+-Activated K+ Channels

Circulation research, 2009-04, Vol.104 (8), p.987-994

2009

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Localized TRPA1 channel Ca2+ signals stimulated by reactive oxygen species promote cerebral artery dilation

Science signaling, 2015-01, Vol.8 (358), p.ra2

2015

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A dietary agonist of transient receptor potential cation channel V3 elicits endothelium-dependent vasodilation

Molecular pharmacology, 2010-04, Vol.77 (4), p.612-620

2010

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Endogenous cytosolic Ca2+ buffering is necessary for TRPM4 activity in cerebral artery smooth muscle cells

Cell calcium (Edinburgh), 2012-01, Vol.51 (1), p.82-93

2012

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STIM1-dependent peripheral coupling governs the contractility of vascular smooth muscle cells

eLife, 2022-02, Vol.11

2022

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Pharmacological inhibition of TRPM4 hyperpolarizes vascular smooth muscle

American Journal of Physiology: Cell Physiology, 2010-11, Vol.299 (5), p.C1195-C1202

2010

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Microtubule structures underlying the sarcoplasmic reticulum support peripheral coupling sites to regulate smooth muscle contractility

Science signaling, 2017-09, Vol.10 (497)

2017

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Dopaminergic neurotoxicants cause biphasic inhibition of purinergic calcium signaling in astrocytes

PloS one, 2014-11, Vol.9 (11), p.e110996

2014

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Regulation of Cerebral Artery Smooth Muscle Membrane Potential by Ca2+-Activated Cation Channels

Microcirculation (New York, N.Y. 1994), 2013-05, Vol.20 (4), p.337-347

2013

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Ex vivo ocular perfusion model to study vascular physiology in the mouse eye

Experimental eye research, 2023-08, Vol.233, p.109543-109543, Article 109543

2023

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Intraluminal pressure elevates intracellular calcium and contracts CNS pericytes: Role of voltage-dependent calcium channels

Proceedings of the National Academy of Sciences - PNAS, 2023-02, Vol.120 (9), p.e2216421120-e2216421120

2023

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Endogenous cytosolic Ca(2+) buffering is necessary for TRPM4 activity in cerebral artery smooth muscle cells

Cell calcium (Edinburgh), 2012-01, Vol.51 (1), p.82-93

2012

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Vascular TRP channels: performing under pressure and going with the flow

Physiology (Bethesda, Md.), 2014-09, Vol.29 (5), p.343-360

2014

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Ca2+ release from the sarcoplasmic reticulum is required for sustained TRPM4 activity in cerebral artery smooth muscle cells

American Journal of Physiology: Cell Physiology, 2010-08, Vol.299 (2), p.C279-C288

2010

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A PLCγ1-dependent, force-sensitive signaling network in the myogenic constriction of cerebral arteries

Science signaling, 2014-05, Vol.7 (327), p.ra49

2014

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CaV3.2 Channels and the Induction of Negative Feedback in Cerebral Arteries

Circulation research, 2014-09, Vol.115 (7), p.650-661

2014

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Calcineurin/nuclear factor of activated T cells-coupled vanilliod transient receptor potential channel 4 ca2+ sparklets stimulate airway smooth muscle cell proliferation

American journal of respiratory cell and molecular biology, 2014-06, Vol.50 (6), p.1064-1075

2014

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PIP2 Improves Cerebral Blood Flow in a Mouse Model of Alzheimer’s Disease

Function (Oxford, England), 2021-01, Vol.2 (2), p.zqab010-zqab010

2021

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Basal protein kinase Cδ activity is required for membrane localization and activity of TRPM4 channels in cerebral artery smooth muscle cells

Channels (Austin, Tex.), 2011-05, Vol.5 (3), p.210-214

2011

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Nitric Oxide Signals Through IRAG to Inhibit TRPM4 Channels and Dilate Cerebral Arteries

Function (Oxford, England), 2021-01, Vol.2 (6), p.zqab051-zqab051

2021

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Ca(V)3.2 channels and the induction of negative feedback in cerebral arteries

Circulation research, 2014-09, Vol.115 (7), p.650

2014

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Vasoconstriction resulting from dynamic membrane trafficking of TRPM4 in vascular smooth muscle cells

American Journal of Physiology: Cell Physiology, 2010-09, Vol.299 (3), p.C682-C694

2010

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(Sub)family feud: crosstalk between TRPC channels in vascular smooth muscle cells during vasoconstrictor agonist stimulation

The Journal of physiology, 2010-10, Vol.588 (19), p.3637-3638

2010

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Capillary K + -sensing initiates retrograde hyperpolarization to increase local cerebral blood flow

Contractile pericytes determine the direction of blood flow at capillary junctions

Endothelium-Dependent Cerebral Artery Dilation Mediated by TRPA1 and Ca2+-Activated K+ Channels

Localized TRPA1 channel Ca2+ signals stimulated by reactive oxygen species promote cerebral artery dilation

A dietary agonist of transient receptor potential cation channel V3 elicits endothelium-dependent vasodilation

Endogenous cytosolic Ca2+ buffering is necessary for TRPM4 activity in cerebral artery smooth muscle cells

STIM1-dependent peripheral coupling governs the contractility of vascular smooth muscle cells

Pharmacological inhibition of TRPM4 hyperpolarizes vascular smooth muscle

Microtubule structures underlying the sarcoplasmic reticulum support peripheral coupling sites to regulate smooth muscle contractility

Dopaminergic neurotoxicants cause biphasic inhibition of purinergic calcium signaling in astrocytes

Regulation of Cerebral Artery Smooth Muscle Membrane Potential by Ca2+-Activated Cation Channels

Ex vivo ocular perfusion model to study vascular physiology in the mouse eye

Intraluminal pressure elevates intracellular calcium and contracts CNS pericytes: Role of voltage-dependent calcium channels

Endogenous cytosolic Ca(2+) buffering is necessary for TRPM4 activity in cerebral artery smooth muscle cells

Vascular TRP channels: performing under pressure and going with the flow

Ca2+ release from the sarcoplasmic reticulum is required for sustained TRPM4 activity in cerebral artery smooth muscle cells

A PLCγ1-dependent, force-sensitive signaling network in the myogenic constriction of cerebral arteries

CaV3.2 Channels and the Induction of Negative Feedback in Cerebral Arteries

Calcineurin/nuclear factor of activated T cells-coupled vanilliod transient receptor potential channel 4 ca2+ sparklets stimulate airway smooth muscle cell proliferation

PIP2 Improves Cerebral Blood Flow in a Mouse Model of Alzheimer’s Disease

Basal protein kinase Cδ activity is required for membrane localization and activity of TRPM4 channels in cerebral artery smooth muscle cells

Nitric Oxide Signals Through IRAG to Inhibit TRPM4 Channels and Dilate Cerebral Arteries

Ca(V)3.2 channels and the induction of negative feedback in cerebral arteries

Vasoconstriction resulting from dynamic membrane trafficking of TRPM4 in vascular smooth muscle cells

(Sub)family feud: crosstalk between TRPC channels in vascular smooth muscle cells during vasoconstrictor agonist stimulation

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