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Breast Cancer Patients Have Greatly Benefited from the Progress in Molecular Oncology

PLoS biology, 2016-09, Vol.14 (9), p.e2000314-e2000314

2016

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Current strategies for the development of peptide-based anti-cancer therapeutics

Journal of peptide science, 2005-11, Vol.11 (11), p.713-726

2005

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The Coactivator of Transcription CREB-binding Protein Interacts Preferentially with the Glycosylated Form of Stat5

The Journal of biological chemistry, 2004-01, Vol.279 (5), p.3563-3572

2004

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Peptide aptamer libraries

Combinatorial chemistry & high throughput screening, 2008-02, Vol.11 (2), p.135

2008

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Sequence-specific Peptide Aptamers, Interacting with the Intracellular Domain of the Epidermal Growth Factor Receptor, Interfere with Stat3 Activation and Inhibit the Growth of Tumor Cells

The Journal of biological chemistry, 2003-09, Vol.278 (39), p.37610-37621

2003

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Restoration of the growth suppression function of mutant p53 by a synthetic peptide derived from the p53 C-terminal domain

Nature medicine, 1997-06, Vol.3 (6), p.632-638

1997

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Characterization of Stat5a and Stat5b Homodimers and Heterodimers and Their Association with the Glucocortiocoid Receptor in Mammary Cells

Molecular and Cellular Biology, 1998-04, Vol.18 (4), p.1783-1792

1998

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Deletion of the carboxyl-terminal transactivation domain of MGF-Stat5 results in sustained DNA binding and a dominant negative phenotype

Molecular and Cellular Biology, 1996-10, Vol.16 (10), p.5691-5700

1996

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Endotoxin depletion of recombinant protein preparations through their preferential binding to histidine tags

Analytical biochemistry, 2014-12, Vol.466, p.83-88

2014

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Comparative Proteomic Analysis of Proliferating and Functionally Differentiated Mammary Epithelial Cells

Molecular & cellular proteomics, 2003-10, Vol.2 (10), p.1039-1054

2003

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Distinct Roles for STAT1, STAT3, and STAT5 in Differentiation Gene Induction and Apoptosis Inhibition by Interleukin-9

The Journal of biological chemistry, 1999-09, Vol.274 (36), p.25855-25861

1999

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STAT3 induction of miR-146b forms a feedback loop to inhibit the NF-κB to IL-6 signaling axis and STAT3-driven cancer phenotypes

Science signaling, 2014-01, Vol.7 (310), p.ra11

2014

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Comparison of the transactivation domains of Stat5 and Stat6 in lymphoid cells and mammary epithelial cells

Molecular and Cellular Biology, 1997-07, Vol.17 (7), p.3663-3678

1997

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Mediation of Growth Hormone-dependent Transcriptional Activation by Mammary Gland Factor/Stat 5

The Journal of biological chemistry, 1995-04, Vol.270 (16), p.9448-9453

1995

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Suppression of metastasis formation by a recombinant single chain antibody-toxin targeted to full-length and oncogenic variant EGF receptors

Oncogene, 1999-03, Vol.18 (9), p.1711-1721

1999

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Jak2-Stat5 Interactions Analyzed in Yeast

The Journal of biological chemistry, 1998-05, Vol.273 (20), p.12567-12575

1998

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Participation of JAK and STAT Proteins in Growth Hormone-induced Signaling (∗)

The Journal of biological chemistry, 1996-03, Vol.271 (10), p.5947-5952

1996

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The histone deacetylase inhibitor valproic acid selectively induces proteasomal degradation of HDAC2

The EMBO journal, 2003-07, Vol.22 (13), p.3411-3420

2003

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The versatile regulation of cellular events by Jak-Stat signaling: from transcriptional control to microtubule dynamics and energy metabolism

Hormone Molecular Biology and Clinical Investigation, 2012-06, Vol.10 (1), p.193-200

2012

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5-Lipoxygenase is a direct p53 target gene in humans

Biochimica et biophysica acta, 2015-08, Vol.1849 (8), p.1003-1016

2015

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Protein interactions at the carboxyl terminus of p53 result in the induction of its in vitro transactivation potential

Oncogene, 1997-07, Vol.15 (2), p.237-244

1997

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Stat5 assumes distinct functions in mammary gland development and mammary tumor formation

Frontiers in bioscience (Landmark. Print), 2012-01, Vol.17 (4), p.1232-1250

2012

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Cytokine receptors and their Stat-mediated mechanisms of action

Hormone molecular biology and clinical investigation, 2012-06, Vol.10 (1), p.191-191

2012

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Mammary gland factor (MGF) is a novel member of the cytokine regulated transcription factor gene family and confers the prolactin response

The EMBO journal, 1994-05, Vol.13 (9), p.2182-2191

1994

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Cytokine Receptor-independent, Constitutively Active Variants of STAT5

The Journal of biological chemistry, 1997-11, Vol.272 (48), p.30237-30243

1997

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Breast Cancer Patients Have Greatly Benefited from the Progress in Molecular Oncology

Current strategies for the development of peptide-based anti-cancer therapeutics

The Coactivator of Transcription CREB-binding Protein Interacts Preferentially with the Glycosylated Form of Stat5

Peptide aptamer libraries

Sequence-specific Peptide Aptamers, Interacting with the Intracellular Domain of the Epidermal Growth Factor Receptor, Interfere with Stat3 Activation and Inhibit the Growth of Tumor Cells

Restoration of the growth suppression function of mutant p53 by a synthetic peptide derived from the p53 C-terminal domain

Characterization of Stat5a and Stat5b Homodimers and Heterodimers and Their Association with the Glucocortiocoid Receptor in Mammary Cells

Deletion of the carboxyl-terminal transactivation domain of MGF-Stat5 results in sustained DNA binding and a dominant negative phenotype

Endotoxin depletion of recombinant protein preparations through their preferential binding to histidine tags

Comparative Proteomic Analysis of Proliferating and Functionally Differentiated Mammary Epithelial Cells

Distinct Roles for STAT1, STAT3, and STAT5 in Differentiation Gene Induction and Apoptosis Inhibition by Interleukin-9

STAT3 induction of miR-146b forms a feedback loop to inhibit the NF-κB to IL-6 signaling axis and STAT3-driven cancer phenotypes

Comparison of the transactivation domains of Stat5 and Stat6 in lymphoid cells and mammary epithelial cells

Mediation of Growth Hormone-dependent Transcriptional Activation by Mammary Gland Factor/Stat 5

Suppression of metastasis formation by a recombinant single chain antibody-toxin targeted to full-length and oncogenic variant EGF receptors

Jak2-Stat5 Interactions Analyzed in Yeast

Participation of JAK and STAT Proteins in Growth Hormone-induced Signaling (∗)

The histone deacetylase inhibitor valproic acid selectively induces proteasomal degradation of HDAC2

The versatile regulation of cellular events by Jak-Stat signaling: from transcriptional control to microtubule dynamics and energy metabolism

5-Lipoxygenase is a direct p53 target gene in humans

Protein interactions at the carboxyl terminus of p53 result in the induction of its in vitro transactivation potential

Stat5 assumes distinct functions in mammary gland development and mammary tumor formation

Cytokine receptors and their Stat-mediated mechanisms of action

Mammary gland factor (MGF) is a novel member of the cytokine regulated transcription factor gene family and confers the prolactin response

Cytokine Receptor-independent, Constitutively Active Variants of STAT5

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