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Pathogenic potential of interferon αβ in acute influenza infection

Nature communications, 2014-05, Vol.5 (1), p.3864-3864, Article 3864

2014

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Influenza-induced monocyte-derived alveolar macrophages confer prolonged antibacterial protection

Nature immunology, 2020-02, Vol.21 (2), p.145

2020

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Type I and III interferons disrupt lung epithelial repair during recovery from viral infection

Science (American Association for the Advancement of Science), 2020-08, Vol.369 (6504), p.712-717

2020

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The Environmental Sensor AHR Protects from Inflammatory Damage by Maintaining Intestinal Stem Cell Homeostasis and Barrier Integrity

Immunity (Cambridge, Mass.), 2018-08, Vol.49 (2), p.353-362.e5

2018

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Microbiota-Driven Tonic Interferon Signals in Lung Stromal Cells Protect from Influenza Virus Infection

Cell reports (Cambridge), 2019-07, Vol.28 (1), p.245-256.e4

2019

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IFNλ is a potent anti‐influenza therapeutic without the inflammatory side effects of IFNα treatment

EMBO molecular medicine, 2016-09, Vol.8 (9), p.1099-1112

2016

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Type I and type III interferons drive redundant amplification loops to induce a transcriptional signature in influenza-infected airway epithelia

PLoS pathogens, 2013-11, Vol.9 (11), p.e1003773-e1003773

2013

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Repair of airway epithelia requires metabolic rewiring towards fatty acid oxidation

Nature communications, 2023-02, Vol.14 (1), p.721-721, Article 721

2023

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Impaired immune response drives age-dependent severity of COVID-19

The Journal of experimental medicine, 2022-12, Vol.219 (12)

2022

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CD81 Is a Central Regulator of Cellular Events Required for Hepatitis C Virus Infection of Human Hepatocytes

Journal of Virology, 2008-09, Vol.82 (17), p.8316-8329

2008

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A Serpin Shapes the Extracellular Environment to Prevent Influenza A Virus Maturation

Cell, 2015-02, Vol.160 (4), p.631-643

2015

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The Environmental Sensor AHR Protects from Inflammatory Damage by Maintaining Intestinal Stem Cell Homeostasis and Barrier Integrity

Immunity (Cambridge, Mass.), 2019-06, Vol.50 (6), p.1542-1542

2019

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Hypoxia inducible factors regulate infectious SARS-CoV-2, epithelial damage and respiratory symptoms in a hamster COVID-19 model

PLoS pathogens, 2022-09, Vol.18 (9), p.e1010807-e1010807

2022

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Rotavirus susceptibility of antibiotic-treated mice ascribed to diminished expression of interleukin-22

PloS one, 2021-08, Vol.16 (8), p.e0247738

2021

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Hepatitis C virions subvert natural killer cell activation to generate a cytokine environment permissive for infection

Journal of hepatology, 2010-02, Vol.52 (2), p.183-190

2010

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The aryl hydrocarbon receptor controls cyclin O to promote epithelial multiciliogenesis

Nature communications, 2016-08, Vol.7 (1), p.12652-11, Article 12652

2016

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Inhibition of natural killer cells through engagement of CD81 by the major hepatitis C virus envelope protein

The Journal of experimental medicine, 2002-01, Vol.195 (1), p.35-41

2002

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Influenza A induces lactate formation to inhibit type I IFN in primary human airway epithelium

iScience, 2021-11, Vol.24 (11), p.103300-103300, Article 103300

2021

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Human RNA Polymerase I-Driven Reverse Genetics for Influenza A Virus in Canine Cells

Journal of Virology, 2010-04, Vol.84 (7), p.3721-3725

2010

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TRAIL+ monocytes and monocyte-related cells cause lung damage and thereby increase susceptibility to influenza-Streptococcus pneumoniae coinfection

EMBO reports, 2015-09, Vol.16 (9), p.1203-1218

2015

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Mutations at positions 186 and 194 in the HA gene of the 2009 H1N1 pandemic influenza virus improve replication in cell culture and eggs

Virology journal, 2010-07, Vol.7 (1), p.157-157, Article 157

2010

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Endothelial AHR activity prevents lung barrier disruption in viral infection

Nature (London), 2023-09, Vol.621 (7980), p.813

2023

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A TLR7 antagonist restricts interferon-dependent and -independent immunopathology in a mouse model of severe influenza

The Journal of experimental medicine, 2021-11, Vol.218 (11)

2021

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Type I interferons drive MAIT cell functions against bacterial pneumonia

The Journal of experimental medicine, 2023-10, Vol.220 (10)

2023

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Pathogenic potential of interferon αβ in acute influenza infection

Influenza-induced monocyte-derived alveolar macrophages confer prolonged antibacterial protection

Type I and III interferons disrupt lung epithelial repair during recovery from viral infection

The Environmental Sensor AHR Protects from Inflammatory Damage by Maintaining Intestinal Stem Cell Homeostasis and Barrier Integrity

Microbiota-Driven Tonic Interferon Signals in Lung Stromal Cells Protect from Influenza Virus Infection

The interferon landscape along the respiratory tract impacts the severity of COVID-19

IFNλ is a potent anti‐influenza therapeutic without the inflammatory side effects of IFNα treatment

Type I and type III interferons drive redundant amplification loops to induce a transcriptional signature in influenza-infected airway epithelia

Repair of airway epithelia requires metabolic rewiring towards fatty acid oxidation

Impaired immune response drives age-dependent severity of COVID-19

CD81 Is a Central Regulator of Cellular Events Required for Hepatitis C Virus Infection of Human Hepatocytes

A Serpin Shapes the Extracellular Environment to Prevent Influenza A Virus Maturation

The Environmental Sensor AHR Protects from Inflammatory Damage by Maintaining Intestinal Stem Cell Homeostasis and Barrier Integrity

Hypoxia inducible factors regulate infectious SARS-CoV-2, epithelial damage and respiratory symptoms in a hamster COVID-19 model

Rotavirus susceptibility of antibiotic-treated mice ascribed to diminished expression of interleukin-22

Hepatitis C virions subvert natural killer cell activation to generate a cytokine environment permissive for infection

The aryl hydrocarbon receptor controls cyclin O to promote epithelial multiciliogenesis

Inhibition of natural killer cells through engagement of CD81 by the major hepatitis C virus envelope protein

Influenza A induces lactate formation to inhibit type I IFN in primary human airway epithelium

Human RNA Polymerase I-Driven Reverse Genetics for Influenza A Virus in Canine Cells

TRAIL+ monocytes and monocyte-related cells cause lung damage and thereby increase susceptibility to influenza-Streptococcus pneumoniae coinfection

Mutations at positions 186 and 194 in the HA gene of the 2009 H1N1 pandemic influenza virus improve replication in cell culture and eggs

Endothelial AHR activity prevents lung barrier disruption in viral infection

A TLR7 antagonist restricts interferon-dependent and -independent immunopathology in a mouse model of severe influenza

Type I interferons drive MAIT cell functions against bacterial pneumonia

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