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HIV-1 accessory proteins adapt cellular adaptors to facilitate immune evasion

PLoS pathogens, 2014-01, Vol.10 (1), p.e1003851-e1003851

2014

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Vpr Is a VIP: HIV Vpr and Infected Macrophages Promote Viral Pathogenesis

Viruses, 2020-07, Vol.12 (8), p.809

2020

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Data we can trust

The Journal of clinical investigation, 2022-08, Vol.132 (15), p.1-2

2022

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Vpr Overcomes Macrophage-Specific Restriction of HIV-1 Env Expression and Virion Production

Cell host & microbe, 2014-12, Vol.16 (6), p.722-735

2014

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HIV-1 infects multipotent progenitor cells causing cell death and establishing latent cellular reservoirs

Nature medicine, 2010-04, Vol.16 (4), p.446-451

2010

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Class 1-Selective Histone Deacetylase (HDAC) Inhibitors Enhance HIV Latency Reversal while Preserving the Activity of HDAC Isoforms Necessary for Maximal HIV Gene Expression

Journal of virology, 2018-03, Vol.92 (6)

2018

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Vpr Promotes Macrophage-Dependent HIV-1 Infection of CD4+ T Lymphocytes

PLoS pathogens, 2015-07, Vol.11 (7), p.e1005054-e1005054

2015

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CD4 is expressed on a heterogeneous subset of hematopoietic progenitors, which persistently harbor CXCR4 and CCR5-tropic HIV proviral genomes in vivo

PLoS pathogens, 2017-07, Vol.13 (7), p.e1006509-e1006509

2017

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Human Immunodeficiency Virus Type 1 Nef: Adapting to Intracellular Trafficking Pathways

Microbiology and Molecular Biology Reviews, 2006-06, Vol.70 (2), p.548-563

2006

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Resource and Technical Advance papers: expanding our resources

JCI insight, 2021-06, Vol.6 (11)

Collins, Kathleen L

2021

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Latent HIV-1 Infection Occurs in Multiple Subsets of Hematopoietic Progenitor Cells and Is Reversed by NF-κB Activation

Journal of Virology, 2012-09, Vol.86 (17), p.9337-9350

2012

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The Impact of Cellular Proliferation on the HIV-1 Reservoir

Viruses, 2020-01, Vol.12 (2), p.127

2020

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A year at the helm

JCI insight, 2020-09, Vol.5 (17)

Collins, Kathleen L

2020

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The emerging role of HLA-C in HIV-1 infection: HLA-C in HIV-1 infection

Immunology, 2011-10, Vol.134 (2), p.116-122

2011

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Mannose receptor is an HIV restriction factor counteracted by Vpr in macrophages

eLife, 2020-03, Vol.9

2020

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Epigenetic silencing of engineered L1 retrotransposition events in human embryonic carcinoma cells

Nature (London), 2010-08, Vol.466 (7307), p.769-773

2010

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Hematopoietic Stem and Progenitor Cells Are a Distinct HIV Reservoir that Contributes to Persistent Viremia in Suppressed Patients

Cell reports (Cambridge), 2018-12, Vol.25 (13), p.3759-3773.e9

2018

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HIV-1 Nef Disrupts Intracellular Trafficking of Major Histocompatibility Complex Class I, CD4, CD8, and CD28 by Distinct Pathways That Share Common Elements

Journal of Virology, 2011-07, Vol.85 (14), p.6867-6881

2011

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HIV-1 utilizes the CXCR4 chemokine receptor to infect multipotent hematopoietic stem and progenitor cells

Cell host & microbe, 2011-03, Vol.9 (3), p.223-234

2011

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HIV-1 Nef targets MHC-I and CD4 for degradation via a final common beta-COP-dependent pathway in T cells

PLoS pathogens, 2008-08, Vol.4 (8), p.e1000131-e1000131

2008

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HIV-1 Nef Disrupts MHC-I Trafficking by Recruiting AP-1 to the MHC-I Cytoplasmic Tail

The Journal of cell biology, 2004-12, Vol.167 (5), p.903-913

2004

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The Tyrosine Binding Pocket in the Adaptor Protein 1 (AP-1) μ1 Subunit Is Necessary for Nef to Recruit AP-1 to the Major Histocompatibility Complex Class I Cytoplasmic Tail

The Journal of biological chemistry, 2008-02, Vol.283 (6), p.3011-3022

2008

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Adaptor Protein 1 Promotes Cross-Presentation through the Same Tyrosine Signal in Major Histocompatibility Complex Class I as That Targeted by HIV-1

Journal of Virology, 2013-07, Vol.87 (14), p.8085-8098

2013

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Vpr Overcomes Macrophage-Specific Restriction of HIV-1 Env Expression and Virion Production

Cell host & microbe, 2015-03, Vol.17 (3), p.414-414

2015

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Multi-omic single-cell velocity models epigenome-transcriptome interactions and improves cell fate prediction

Nature biotechnology, 2023-03, Vol.41 (3), p.387-398

2023

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HIV-1 accessory proteins adapt cellular adaptors to facilitate immune evasion

Vpr Is a VIP: HIV Vpr and Infected Macrophages Promote Viral Pathogenesis

Data we can trust

Vpr Overcomes Macrophage-Specific Restriction of HIV-1 Env Expression and Virion Production

HIV-1 infects multipotent progenitor cells causing cell death and establishing latent cellular reservoirs

Class 1-Selective Histone Deacetylase (HDAC) Inhibitors Enhance HIV Latency Reversal while Preserving the Activity of HDAC Isoforms Necessary for Maximal HIV Gene Expression

Vpr Promotes Macrophage-Dependent HIV-1 Infection of CD4+ T Lymphocytes

CD4 is expressed on a heterogeneous subset of hematopoietic progenitors, which persistently harbor CXCR4 and CCR5-tropic HIV proviral genomes in vivo

Human Immunodeficiency Virus Type 1 Nef: Adapting to Intracellular Trafficking Pathways

Resource and Technical Advance papers: expanding our resources

Latent HIV-1 Infection Occurs in Multiple Subsets of Hematopoietic Progenitor Cells and Is Reversed by NF-κB Activation

The Impact of Cellular Proliferation on the HIV-1 Reservoir

A year at the helm

The emerging role of HLA-C in HIV-1 infection: HLA-C in HIV-1 infection

Mannose receptor is an HIV restriction factor counteracted by Vpr in macrophages

Epigenetic silencing of engineered L1 retrotransposition events in human embryonic carcinoma cells

Hematopoietic Stem and Progenitor Cells Are a Distinct HIV Reservoir that Contributes to Persistent Viremia in Suppressed Patients

HIV-1 Nef Disrupts Intracellular Trafficking of Major Histocompatibility Complex Class I, CD4, CD8, and CD28 by Distinct Pathways That Share Common Elements

HIV-1 utilizes the CXCR4 chemokine receptor to infect multipotent hematopoietic stem and progenitor cells

HIV-1 Nef targets MHC-I and CD4 for degradation via a final common beta-COP-dependent pathway in T cells

HIV-1 Nef Disrupts MHC-I Trafficking by Recruiting AP-1 to the MHC-I Cytoplasmic Tail

The Tyrosine Binding Pocket in the Adaptor Protein 1 (AP-1) μ1 Subunit Is Necessary for Nef to Recruit AP-1 to the Major Histocompatibility Complex Class I Cytoplasmic Tail

Adaptor Protein 1 Promotes Cross-Presentation through the Same Tyrosine Signal in Major Histocompatibility Complex Class I as That Targeted by HIV-1

Vpr Overcomes Macrophage-Specific Restriction of HIV-1 Env Expression and Virion Production

Multi-omic single-cell velocity models epigenome-transcriptome interactions and improves cell fate prediction

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