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BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

Cell death and differentiation, 2019-06, Vol.26 (6), p.1037-1047

2019

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The in vitro cleavage of the hAtg proteins by cell death proteases

Autophagy, 2010-11, Vol.6 (8), p.1042-1056

2010

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Concurrent up-regulation of BCL-XL and BCL2A1 induces approximately 1000-fold resistance to ABT-737 in chronic lymphocytic leukemia

Blood, 2009-04, Vol.113 (18), p.4403-4413

2009

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BIM-Mediated Membrane Insertion of the BAK Pore Domain Is an Essential Requirement for Apoptosis

Cell reports (Cambridge), 2013-10, Vol.5 (2), p.409-420

2013

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BCL2/BCL-XL inhibition induces apoptosis, disrupts cellular calcium homeostasis, and prevents platelet activation

Blood, 2011-06, Vol.117 (26), p.7145-7154

2011

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The E3 Ubiquitin Ligase cIAP1 Binds and Ubiquitinates Caspase-3 and -7 via Unique Mechanisms at Distinct Steps in Their Processing

The Journal of biological chemistry, 2009-05, Vol.284 (19), p.12772-12782

2009

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Novel roles of RTN4 and CLIMP-63 in regulating mitochondrial structure, bioenergetics and apoptosis

Cell death & disease, 2022-05, Vol.13 (5), p.436-436, Article 436

2022

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Evidence for the Involvement of Carbon-centered Radicals in the Induction of Apoptotic Cell Death by Artemisinin Compounds

The Journal of biological chemistry, 2007-03, Vol.282 (13), p.9372-9382

2007

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Barriers to effective TRAIL-targeted therapy of malignancy

Journal of clinical oncology, 2007-10, Vol.25 (28), p.4505-4506

2007

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Exploring the potential of BH3 mimetic therapy in squamous cell carcinoma of the head and neck

Cell death & disease, 2019-12, Vol.10 (12), p.912-10, Article 912

2019

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Sabutoclax (BI97C1) and BI112D1, Putative Inhibitors of MCL-1, Induce Mitochondrial Fragmentation Either Upstream of or Independent of Apoptosis

Neoplasia (New York, N.Y.), 2013-05, Vol.15 (5), p.568-IN22

2013

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Proteasome-mediated Degradation of Smac during Apoptosis: XIAP Promotes Smac Ubiquitination in Vitro

The Journal of biological chemistry, 2002-09, Vol.277 (39), p.36611-36616

2002

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Heat-shock protein 70 inhibits apoptosis by preventing recruitment of procaspase-9 to the Apaf-1 apoptosome

Nature cell biology, 2000-08, Vol.2 (8), p.469-475

2000

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Receptor-mediated Endocytosis Is Not Required for Tumor Necrosis Factor-related Apoptosis-inducing Ligand (TRAIL)-induced Apoptosis

The Journal of biological chemistry, 2007-04, Vol.282 (17), p.12831-12841

2007

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Increased hepatotoxicity of tumor necrosis factor–related apoptosis‐inducing ligand in diseased human liver

Hepatology (Baltimore, Md.), 2007-11, Vol.46 (5), p.1498-1508

2007

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The Apaf-1 apoptosome: a large caspase-activating complex

Biochimie, 2002-02, Vol.84 (2), p.203-214

2002

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Caspase Activation Inhibits Proteasome Function during Apoptosis

Molecular cell, 2004-04, Vol.14 (1), p.81-93

2004

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Role of NOXA and its ubiquitination in proteasome inhibitor-induced apoptosis in chronic lymphocytic leukemia cells

Haematologica (Roma), 2010-09, Vol.95 (9), p.1510-1518

2010

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Fas-associated Death Domain Protein and Caspase-8 Are Not Recruited to the Tumor Necrosis Factor Receptor 1 Signaling Complex during Tumor Necrosis Factor-induced Apoptosis

The Journal of biological chemistry, 2003-07, Vol.278 (28), p.25534-25541

2003

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TRAIL receptor-selective mutants signal to apoptosis via TRAIL-R1 in primary lymphoid malignancies

Cancer research (Chicago, Ill.), 2005-12, Vol.65 (24), p.11265-11270

2005

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Bcl-2 and Bcl-xL Inhibit CD95-mediated Apoptosis by Preventing Mitochondrial Release of Smac/DIABLO and Subsequent Inactivation of X-linked Inhibitor-of-Apoptosis Protein

The Journal of biological chemistry, 2002-03, Vol.277 (13), p.11345-11351

2002

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Endoplasmic reticulum membrane reorganization is regulated by ionic homeostasis

PloS one, 2013-02, Vol.8 (2), p.e56603

2013

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p53 Is Cleaved by Caspases Generating Fragments Localizing to Mitochondria

The Journal of biological chemistry, 2006-05, Vol.281 (19), p.13566-13573

2006

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BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-X L

Cell death and differentiation, 2019-06, Vol.26 (6), p.1037

2019

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Enhanced Fas-associated death domain recruitment by histone deacetylase inhibitors is critical for the sensitization of chronic lymphocytic leukemia cells to TRAIL-induced apoptosis

Molecular cancer therapeutics, 2009-11, Vol.8 (11), p.3088-3097

2009

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BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

The in vitro cleavage of the hAtg proteins by cell death proteases

Concurrent up-regulation of BCL-XL and BCL2A1 induces approximately 1000-fold resistance to ABT-737 in chronic lymphocytic leukemia

BIM-Mediated Membrane Insertion of the BAK Pore Domain Is an Essential Requirement for Apoptosis

BCL2/BCL-XL inhibition induces apoptosis, disrupts cellular calcium homeostasis, and prevents platelet activation

The E3 Ubiquitin Ligase cIAP1 Binds and Ubiquitinates Caspase-3 and -7 via Unique Mechanisms at Distinct Steps in Their Processing

Novel roles of RTN4 and CLIMP-63 in regulating mitochondrial structure, bioenergetics and apoptosis

Evidence for the Involvement of Carbon-centered Radicals in the Induction of Apoptotic Cell Death by Artemisinin Compounds

Barriers to effective TRAIL-targeted therapy of malignancy

Exploring the potential of BH3 mimetic therapy in squamous cell carcinoma of the head and neck

Sabutoclax (BI97C1) and BI112D1, Putative Inhibitors of MCL-1, Induce Mitochondrial Fragmentation Either Upstream of or Independent of Apoptosis

Proteasome-mediated Degradation of Smac during Apoptosis: XIAP Promotes Smac Ubiquitination in Vitro

Heat-shock protein 70 inhibits apoptosis by preventing recruitment of procaspase-9 to the Apaf-1 apoptosome

Receptor-mediated Endocytosis Is Not Required for Tumor Necrosis Factor-related Apoptosis-inducing Ligand (TRAIL)-induced Apoptosis

Increased hepatotoxicity of tumor necrosis factor–related apoptosis‐inducing ligand in diseased human liver

The Apaf-1 apoptosome: a large caspase-activating complex

Caspase Activation Inhibits Proteasome Function during Apoptosis

Role of NOXA and its ubiquitination in proteasome inhibitor-induced apoptosis in chronic lymphocytic leukemia cells

Fas-associated Death Domain Protein and Caspase-8 Are Not Recruited to the Tumor Necrosis Factor Receptor 1 Signaling Complex during Tumor Necrosis Factor-induced Apoptosis

TRAIL receptor-selective mutants signal to apoptosis via TRAIL-R1 in primary lymphoid malignancies

Bcl-2 and Bcl-xL Inhibit CD95-mediated Apoptosis by Preventing Mitochondrial Release of Smac/DIABLO and Subsequent Inactivation of X-linked Inhibitor-of-Apoptosis Protein

Endoplasmic reticulum membrane reorganization is regulated by ionic homeostasis

p53 Is Cleaved by Caspases Generating Fragments Localizing to Mitochondria

BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-X L

Enhanced Fas-associated death domain recruitment by histone deacetylase inhibitors is critical for the sensitization of chronic lymphocytic leukemia cells to TRAIL-induced apoptosis

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