UB Paderborn / Katalog / Suche / in CDI

1 – 23 von 23

Sortiert nach

Innate Immune Cell Recovery Is Positively Regulated by NLRP12 during Emergency Hematopoiesis

The Journal of immunology (1950), 2017-03, Vol.198 (6), p.2426-2433

2017

Link zum Volltext

Verfügbarkeit prüfen

Lymphopenia-Induced Homeostatic Proliferation of CD8+ T Cells Is a Mechanism for Effective Allogeneic Skin Graft Rejection following Burn Injury

The Journal of immunology (1950), 2006-06, Vol.176 (11), p.6717-6726

2006

Link zum Volltext

Verfügbarkeit prüfen

The Mammalian Target of Rapamycin (mTOR) / Peroxisome Proliferator-Activated Receptor γ (PPARγ) axis drives immune dysfunction and outcome after burn injury

The Journal of immunology (1950), 2018-05, Vol.200 (1_Supplement), p.114-114.21

2018

Link zum Volltext

Verfügbarkeit prüfen

Blocking CXCL1-dependent neutrophil recruitment prevents immune damage and reduces pulmonary bacterial infection after inhalation injury

The Journal of immunology (1950), 2018-05, Vol.200 (1_Supplement), p.114-114.23

2018

Link zum Volltext

Verfügbarkeit prüfen

Immunoparalysis is mediated by a dysfunctional IL-10/IL-12 axis in severe burn injury

The Journal of immunology (1950), 2017-05, Vol.198 (1_Supplement), p.63-63.20

2017

Link zum Volltext

Verfügbarkeit prüfen

A blood immune suppression index (BISI) based on IL10/IL12 and ARG1/NOS2 ratios is predictive of immune dysfunction and respiratory failure after burn injury in patients

The Journal of immunology (1950), 2018-05, Vol.200 (1_Supplement), p.166-166.30

2018

Link zum Volltext

Verfügbarkeit prüfen

NLRP12 limits TNFα-induced apoptosis of monocyte progenitor cells during emergency hematopoiesis

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.62-62.13

2016

Link zum Volltext

Verfügbarkeit prüfen

LPS potentiates insulin-driven hypoglycemic shock

The Journal of immunology (1950), 2017-10, Vol.199 (10), p.3634-3643

2017

Link zum Volltext

Verfügbarkeit prüfen

Inhalation injury causes recruitment of IL-12lo neutrophils late after injury

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.60-60.27

2016

Link zum Volltext

Verfügbarkeit prüfen

Burn injury induces a myeloid suppressor phenotype and susceptibility to Pseudomonas infection (129.24)

The Journal of immunology (1950), 2009-04, Vol.182 (1_Supplement), p.129-129.24

2009

Link zum Volltext

Verfügbarkeit prüfen

Microvesicle-associated immunomodulators are critical regulators of immune dysfunction following burn injury

The Journal of immunology (1950), 2021-05, Vol.206 (1_Supplement), p.109-109.13

2021

Link zum Volltext

Verfügbarkeit prüfen

Nrf2-mediated pentose phosphate pathway activity promotes macrophage inflammasome activation and bacterial killing in microbial sepsis (INM6P.330)

The Journal of immunology (1950), 2015-05, Vol.194 (1_Supplement), p.193-193.4

2015

Link zum Volltext

Verfügbarkeit prüfen

Mitochondrial damage-associated molecular patterns exacerbate acute lung injury following burn (IRM11P.640)

The Journal of immunology (1950), 2015-05, Vol.194 (1_Supplement), p.132-132.19

2015

Link zum Volltext

Verfügbarkeit prüfen

NLRP12-deficiency results in TNFα-mediated bone marrow apoptosis during emergency hematopoiesis (INM2P.355)

The Journal of immunology (1950), 2015-05, Vol.194 (1_Supplement), p.126-126.8

2015

Link zum Volltext

Verfügbarkeit prüfen

NLRP12 suppresses host innate immune signaling to limit inflammation during emergency hematopoiesis (INM6P.404)

The Journal of immunology (1950), 2014-05, Vol.192 (1_Supplement), p.122-122.1

2014

Link zum Volltext

Verfügbarkeit prüfen

Suppressed pulmonary innate immune responses after burn injury are regulated by mTOR (INM7P.429)

The Journal of immunology (1950), 2014-05, Vol.192 (1_Supplement), p.123-123.7

2014

Link zum Volltext

Verfügbarkeit prüfen

Pseudomonas aeruginosa infection after burn injury polarizes innate immune cells towards an alternatively activated phenotype (55.19)

The Journal of immunology (1950), 2012-05, Vol.188 (1_Supplement), p.55-55.19

2012

Link zum Volltext

Verfügbarkeit prüfen

Combined radiation and thermal injury induces a non-suppressive myeloid CD11b+ Gr1+ cell population (P4025)

The Journal of immunology (1950), 2013-05, Vol.190 (1_Supplement), p.131-131.13

2013

Link zum Volltext

Verfügbarkeit prüfen

Increased neutrophil migration rescues impaired function and susceptibility to Pseudomonas pneumonia after burn injury (56.25)

The Journal of immunology (1950), 2011-04, Vol.186 (1_Supplement), p.56-56.25

2011

Link zum Volltext

Verfügbarkeit prüfen

Radiation combined with thermal injury: induction of a radio-resistant immature myeloid cell. (118.10)

The Journal of immunology (1950), 2012-05, Vol.188 (1_Supplement), p.118-118.10

2012

Link zum Volltext

Verfügbarkeit prüfen

Acute inflammasome activation and downregulation after severe burn injury in humans. (169.23)

The Journal of immunology (1950), 2012-05, Vol.188 (1_Supplement), p.169-169.23

2012

Link zum Volltext

Verfügbarkeit prüfen

Cytomegalovirus reactivation occurs in burn patients and correlates with increased central memory and regulatory T cell populations. (39.32)

The Journal of immunology (1950), 2010-04, Vol.184 (1_Supplement), p.39-39.32

2010

Link zum Volltext

Verfügbarkeit prüfen

Severe burn injury leads to spontaneous generation of Th17 CD4+ T cells in wound draining lymph nodes (42.18)

The Journal of immunology (1950), 2010-04, Vol.184 (1_Supplement), p.42-42.18

2010

Link zum Volltext

Verfügbarkeit prüfen

1 – 23 von 23

Suche als RSS-Feed

Aktive Filter

Zeitschrift / SerieThe Journal Of Immunology

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(23)
Mit Volltextzugriff
(23)

Publikationsform

Artikel
(23)

Sprache

Englisch
(23)

Erscheinungsjahr

n.n

Thema

Animals
(2)
Burns - Immunology
(2)
Mice
(2)
Mice, Inbred C57bl
(2)
Adoptive Transfer
(1)
Animal Tissues
(1)
Apoptosis
(1)
Apoptosis - Drug Effects
(1)
Apoptosis - Immunology
(1)
Bacteria
(1)
Bone Marrow
(1)
Burns - Pathology
(1)
Cd8-Positive T-Lymphocytes - Immunology
(1)
Cd8-Positive T-Lymphocytes - Pathology
(1)
Cd8-Positive T-Lymphocytes - Transplantation
(1)
Cell Cycle - Immunology
(1)
Cell Proliferation
(1)
Cell Self Renewal
(1)
Cytokines
(1)
Cytokines - Biosynthesis
(1)

15 weitere anzeigen...

Weniger anzeigen...

Kollektion

Menü

Weitere Dienste

Einstellungen

Innate Immune Cell Recovery Is Positively Regulated by NLRP12 during Emergency Hematopoiesis

Lymphopenia-Induced Homeostatic Proliferation of CD8+ T Cells Is a Mechanism for Effective Allogeneic Skin Graft Rejection following Burn Injury

The Mammalian Target of Rapamycin (mTOR) / Peroxisome Proliferator-Activated Receptor γ (PPARγ) axis drives immune dysfunction and outcome after burn injury

Blocking CXCL1-dependent neutrophil recruitment prevents immune damage and reduces pulmonary bacterial infection after inhalation injury

Immunoparalysis is mediated by a dysfunctional IL-10/IL-12 axis in severe burn injury

A blood immune suppression index (BISI) based on IL10/IL12 and ARG1/NOS2 ratios is predictive of immune dysfunction and respiratory failure after burn injury in patients

NLRP12 limits TNFα-induced apoptosis of monocyte progenitor cells during emergency hematopoiesis

LPS potentiates insulin-driven hypoglycemic shock

Inhalation injury causes recruitment of IL-12lo neutrophils late after injury

Burn injury induces a myeloid suppressor phenotype and susceptibility to Pseudomonas infection (129.24)

Microvesicle-associated immunomodulators are critical regulators of immune dysfunction following burn injury

Nrf2-mediated pentose phosphate pathway activity promotes macrophage inflammasome activation and bacterial killing in microbial sepsis (INM6P.330)

Mitochondrial damage-associated molecular patterns exacerbate acute lung injury following burn (IRM11P.640)

NLRP12-deficiency results in TNFα-mediated bone marrow apoptosis during emergency hematopoiesis (INM2P.355)

NLRP12 suppresses host innate immune signaling to limit inflammation during emergency hematopoiesis (INM6P.404)

Suppressed pulmonary innate immune responses after burn injury are regulated by mTOR (INM7P.429)

Pseudomonas aeruginosa infection after burn injury polarizes innate immune cells towards an alternatively activated phenotype (55.19)

Combined radiation and thermal injury induces a non-suppressive myeloid CD11b+ Gr1+ cell population (P4025)

Increased neutrophil migration rescues impaired function and susceptibility to Pseudomonas pneumonia after burn injury (56.25)

Radiation combined with thermal injury: induction of a radio-resistant immature myeloid cell. (118.10)

Acute inflammasome activation and downregulation after severe burn injury in humans. (169.23)

Cytomegalovirus reactivation occurs in burn patients and correlates with increased central memory and regulatory T cell populations. (39.32)

Severe burn injury leads to spontaneous generation of Th17 CD4+ T cells in wound draining lymph nodes (42.18)

Aktive Filter

Suchergebnisse filtern