UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 957

Sortiert nach

PI3Kγ inhibition suppresses microglia/TAM accumulation in glioblastoma microenvironment to promote exceptional temozolomide response

Proceedings of the National Academy of Sciences - PNAS, 2021-04, Vol.118 (16), p.1

2021

Link zum Volltext

Verfügbarkeit prüfen

Phosphoinositide 3-kinase inhibitors induce DNA damage through nucleoside depletion

Proceedings of the National Academy of Sciences - PNAS, 2016-07, Vol.113 (30), p.E4338-E4347

2016

Link zum Volltext

Verfügbarkeit prüfen

Oncogenic activity of the regulatory subunit p85β of phosphatidylinositol 3-kinase (PI3K)

Proceedings of the National Academy of Sciences - PNAS, 2014-11, Vol.111 (47), p.16826-16829

2014

Link zum Volltext

Verfügbarkeit prüfen

Molecular determinants of PI3Kγ-mediated activation downstream of G-protein–coupled receptors (GPCRs)

Proceedings of the National Academy of Sciences - PNAS, 2013-11, Vol.110 (47), p.18862-18867

2013

Link zum Volltext

Verfügbarkeit prüfen

Cancer-associated mutations in the p85α N-terminal SH2 domain activate a spectrum of receptor tyrosine kinases

Proceedings of the National Academy of Sciences - PNAS, 2021-09, Vol.118 (37)

2021

Link zum Volltext

Verfügbarkeit prüfen

Insulin growth factor signaling is regulated by microRNA-486, an underexpressed microRNA in lung cancer

Proceedings of the National Academy of Sciences - PNAS, 2013-09, Vol.110 (37), p.15043-15048

2013

Link zum Volltext

Verfügbarkeit prüfen

PIK3CA C-terminal frameshift mutations are novel oncogenic events that sensitize tumors to PI3K-α inhibition

Proceedings of the National Academy of Sciences - PNAS, 2020-09, Vol.117 (39), p.24427-24433

2020

Link zum Volltext

Verfügbarkeit prüfen

Quantitation of class IA PI3Ks in mice reveals p110-free-p85s and isoform-selective subunit associations and recruitment to receptors

Proceedings of the National Academy of Sciences - PNAS, 2018-11, Vol.115 (48), p.12176-12181

2018

Link zum Volltext

Verfügbarkeit prüfen

PI3K-p110α mediates the oncogenic activity induced by loss of the novel tumor suppressor PI3K-p85α

Proceedings of the National Academy of Sciences - PNAS, 2017-07, Vol.114 (27), p.7095-7100

2017

Link zum Volltext

Verfügbarkeit prüfen

Activation of PI3K/Akt pathway by CD133-p85 interaction promotes tumorigenic capacity of glioma stem cells

Proceedings of the National Academy of Sciences - PNAS, 2013-04, Vol.110 (17), p.6829-6834

2013

Link zum Volltext

Verfügbarkeit prüfen

Long noncoding RNA-mediated activation of PROTOR1/PRR5-AKT signaling shunt downstream of PI3K in triple-negative breast cancer

Proceedings of the National Academy of Sciences - PNAS, 2022-10, Vol.119 (43), p.e2203180119-e2203180119

2022

Link zum Volltext

Verfügbarkeit prüfen

p110γ deficiency protects against pancreatic carcinogenesis yet predisposes to diet-induced hepatotoxicity

Proceedings of the National Academy of Sciences - PNAS, 2019-07, Vol.116 (29), p.14724-14733

2019

Link zum Volltext

Verfügbarkeit prüfen

PARP-1–dependent recruitment of cold-inducible RNA-binding protein promotes double-strand break repair and genome stability

Proceedings of the National Academy of Sciences - PNAS, 2018-02, Vol.115 (8), p.E1759-E1768

2018

Link zum Volltext

Verfügbarkeit prüfen

Rubicon controls endosome maturation as a Rab7 effector

Proceedings of the National Academy of Sciences - PNAS, 2010-11, Vol.107 (45), p.19338-19343

2010

Link zum Volltext

Verfügbarkeit prüfen

Insulin resistance and diabetes caused by genetic or diet-induced KBTBD2 deficiency in mice

Proceedings of the National Academy of Sciences - PNAS, 2016-10, Vol.113 (42), p.E6418-E6426

2016

Link zum Volltext

Verfügbarkeit prüfen

MYO6 is targeted by Salmonella virulence effectors to trigger PI3-kinase signaling and pathogen invasion into host cells

Proceedings of the National Academy of Sciences - PNAS, 2017-04, Vol.114 (15), p.3915-3920

2017

Link zum Volltext

Verfügbarkeit prüfen

Essential role of Stat3 in PI3K-induced oncogenic transformation

Proceedings of the National Academy of Sciences - PNAS, 2011-08, Vol.108 (32), p.13247-13252

2011

Link zum Volltext

Verfügbarkeit prüfen

Tissue-specific disruption of Kbtbd2 uncovers adipocyte-intrinsic and -extrinsic features of the teeny lipodystrophy syndrome

Proceedings of the National Academy of Sciences - PNAS, 2020-05, Vol.117 (21), p.11829-11835

2020

Link zum Volltext

Verfügbarkeit prüfen

G q/11 -dependent regulation of endosomal cAMP generation by parathyroid hormone class B GPCR

Proceedings of the National Academy of Sciences - PNAS, 2020-03, Vol.117 (13), p.7455-7460

2020

Link zum Volltext

Verfügbarkeit prüfen

PI3Kγ within a nonhematopoietic cell type negatively regulates diet-induced thermogenesis and promotes obesity and insulin resistance

Proceedings of the National Academy of Sciences - PNAS, 2011-10, Vol.108 (42), p.E854-E863

2011

Link zum Volltext

Verfügbarkeit prüfen

Detection of a rare BCR–ABL tyrosine kinase fusion protein in H929 multiple myeloma cells using immunoprecipitation (IP)-tandem mass spectrometry (MS/MS)

Proceedings of the National Academy of Sciences - PNAS, 2012-10, Vol.109 (40), p.16190-16195

2012

Link zum Volltext

Verfügbarkeit prüfen

Inhibition of PI3K binding to activators by serine phosphorylation of PI3K regulatory subunit p85alpha Src homology-2 domains

Proceedings of the National Academy of Sciences - PNAS, 2011-08, Vol.108 (34), p.14157

2011

Link zum Volltext

Verfügbarkeit prüfen

Oncogenic activation of PI3K-AKT-mTOR signaling suppresses ferroptosis via SREBP-mediated lipogenesis

Proceedings of the National Academy of Sciences - PNAS, 2020-12, Vol.117 (49), p.31189-31197

2020

Link zum Volltext

Verfügbarkeit prüfen

Lipid kinase PIK3C3 maintains healthy brown and white adipose tissues to prevent metabolic diseases

Proceedings of the National Academy of Sciences - PNAS, 2023-01, Vol.120 (1), p.e2214874120-e2214874120

2023

Link zum Volltext

Verfügbarkeit prüfen

Oncogenic mutations mimic and enhance dynamic events in the natural activation of phosphoinositide 3-kinase p110α (PIK3CA)

Proceedings of the National Academy of Sciences - PNAS, 2012-09, Vol.109 (38), p.15259-15264

2012

Link zum Volltext

Verfügbarkeit prüfen

1 – 25 von 957

Suche als RSS-Feed

Aktive Filter

Zeitschrift / SerieDiabetes

Zeitschrift / SerieProceedings Of The National Academy Of Sciences - Pnas

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(957)
Mit Volltextzugriff
(956)

Publikationsform

Artikel
(957)

Sprache

Englisch
(957)
Japanisch
(443)

Erscheinungsjahr

n.n

Thema

Multidisciplinary Sciences
(854)
Science & Technology
(854)
Science & Technology - Other Topics
(854)
Biological Sciences
(775)
Animals
(663)
Humans
(525)
Mice
(445)
Multidisciplinary
(431)
Proteins
(415)
Signal Transduction
(364)
Phosphorylation
(356)
Antibodies
(283)
Cell Lines
(272)
Receptors
(246)
Cells
(235)
Phosphatidylinositol 3-Kinases - Metabolism
(233)
Cell Line
(189)
Cellular Biology
(184)
Tumors
(167)
Cell Growth
(162)

15 weitere anzeigen...

Weniger anzeigen...

Kollektion

Zeitschrift / Serie

2 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

PI3Kγ inhibition suppresses microglia/TAM accumulation in glioblastoma microenvironment to promote exceptional temozolomide response

Phosphoinositide 3-kinase inhibitors induce DNA damage through nucleoside depletion

Oncogenic activity of the regulatory subunit p85β of phosphatidylinositol 3-kinase (PI3K)

Molecular determinants of PI3Kγ-mediated activation downstream of G-protein–coupled receptors (GPCRs)

Cancer-associated mutations in the p85α N-terminal SH2 domain activate a spectrum of receptor tyrosine kinases

Insulin growth factor signaling is regulated by microRNA-486, an underexpressed microRNA in lung cancer

PIK3CA C-terminal frameshift mutations are novel oncogenic events that sensitize tumors to PI3K-α inhibition

Quantitation of class IA PI3Ks in mice reveals p110-free-p85s and isoform-selective subunit associations and recruitment to receptors

PI3K-p110α mediates the oncogenic activity induced by loss of the novel tumor suppressor PI3K-p85α

Activation of PI3K/Akt pathway by CD133-p85 interaction promotes tumorigenic capacity of glioma stem cells

Long noncoding RNA-mediated activation of PROTOR1/PRR5-AKT signaling shunt downstream of PI3K in triple-negative breast cancer

p110γ deficiency protects against pancreatic carcinogenesis yet predisposes to diet-induced hepatotoxicity

PARP-1–dependent recruitment of cold-inducible RNA-binding protein promotes double-strand break repair and genome stability

Rubicon controls endosome maturation as a Rab7 effector

Insulin resistance and diabetes caused by genetic or diet-induced KBTBD2 deficiency in mice

MYO6 is targeted by Salmonella virulence effectors to trigger PI3-kinase signaling and pathogen invasion into host cells

Essential role of Stat3 in PI3K-induced oncogenic transformation

Tissue-specific disruption of Kbtbd2 uncovers adipocyte-intrinsic and -extrinsic features of the teeny lipodystrophy syndrome

G q/11 -dependent regulation of endosomal cAMP generation by parathyroid hormone class B GPCR

PI3Kγ within a nonhematopoietic cell type negatively regulates diet-induced thermogenesis and promotes obesity and insulin resistance

Detection of a rare BCR–ABL tyrosine kinase fusion protein in H929 multiple myeloma cells using immunoprecipitation (IP)-tandem mass spectrometry (MS/MS)

Inhibition of PI3K binding to activators by serine phosphorylation of PI3K regulatory subunit p85alpha Src homology-2 domains

Oncogenic activation of PI3K-AKT-mTOR signaling suppresses ferroptosis via SREBP-mediated lipogenesis

Lipid kinase PIK3C3 maintains healthy brown and white adipose tissues to prevent metabolic diseases

Oncogenic mutations mimic and enhance dynamic events in the natural activation of phosphoinositide 3-kinase p110α (PIK3CA)

Aktive Filter

Suchergebnisse filtern