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VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like disease

Science (American Association for the Advancement of Science), 2019-12, Vol.366 (6472), p.1531-1536

2019

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Emerging molecular mechanisms in chemotherapy: Ca2+ signaling at the mitochondria-associated endoplasmic reticulum membranes

Cell death & disease, 2018-02, Vol.9 (3), p.1-15, Article 334

2018

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Role of Mitochondria-Associated ER Membranes in Calcium Regulation in Cancer-Specific Settings

Neoplasia (New York, N.Y.), 2018-05, Vol.20 (5), p.510-523

2018

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Recent advances in uncovering the mechanisms contributing to BIRD-2-induced cell death in B-cell cancer cells

Cell death & disease, 2019-01, Vol.10 (2), p.42-42, Article 42

2019

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Endoplasmic Reticulum–Mitochondrial Ca2+ Fluxes Underlying Cancer Cell Survival

Frontiers in oncology, 2017-05, Vol.7, p.70-70

2017

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The AMPK-related kinase NUAK1 controls cortical axons branching by locally modulating mitochondrial metabolic functions

Nature communications, 2024-03, Vol.15 (1), p.2487-2487

2024

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Endoplasmic Reticulum-Mitochondria Communication Through Ca2+ Signaling: The Importance of Mitochondria-Associated Membranes (MAMs)

Organelle Contact Sites, 2017, p.49-67

2017

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Intracellular BAPTA directly inhibits PFKFB3, thereby impeding mTORC1-driven Mcl-1 translation and killing MCL-1-addicted cancer cells

Cell death & disease, 2023-09, Vol.14 (9), p.600-600, Article 600

2023

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DLBCL Cells with Acquired Resistance to Venetoclax Are Not Sensitized to BIRD-2 But Can Be Resensitized to Venetoclax through Bcl-XL Inhibition

Biomolecules (Basel, Switzerland), 2020-07, Vol.10 (7), p.1081

2020

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Endoplasmic Reticulum-Mitochondrial Ca 2+ Fluxes Underlying Cancer Cell Survival

Frontiers in oncology, 2017, Vol.7, p.70

2017

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Recent advances in understanding IP3R function with focus on ER-mitochondrial Ca2+ transfers

Current opinion in physiology, 2020-10, Vol.17, p.80-88

2020

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ABT-199 (Venetoclax), a BH3-mimetic Bcl-2 inhibitor, does not cause Ca 2+ -signalling dysregulation or toxicity in pancreatic acinar cells

British journal of pharmacology, 2019-11, Vol.176 (22), p.4402-4415

2019

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ABT‐199 (Venetoclax), a BH3‐mimetic Bcl‐2 inhibitor, does not cause Ca2+‐signalling dysregulation or toxicity in pancreatic acinar cells

British journal of pharmacology, 2019-11, Vol.176 (22), p.4402-4415

2019

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VDAC oligomers form mitochondrial pores that release small mtDNA fragments and promote lupus-like disease

Science (American Association for the Advancement of Science), 2019-12, Vol.366 (6472), p.1531-1536

2019

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Role of Mitochondria-Associated ER Membranes in Calcium Regulation in Cancer-Specific Settings1

Neoplasia (New York, N.Y.), 2018-04, Vol.20 (5), p.510-523

2018

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Alterations in Ca 2+ Signalling via ER-Mitochondria Contact Site Remodelling in Cancer

Advances in experimental medicine and biology, 2017, Vol.997, p.225

2017

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Alterations in Ca2+ Signalling via ER-Mitochondria Contact Site Remodelling in Cancer

Organelle Contact Sites, 2017, Vol.997, p.225-254

2017

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Endoplasmic Reticulum-Mitochondria Communication Through Ca 2+ Signaling: The Importance of Mitochondria-Associated Membranes (MAMs)

Advances in experimental medicine and biology, 2017, Vol.997, p.49

2017

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Emerging molecular mechanisms in chemotherapy: Ca 2+ signaling at the mitochondria-associated endoplasmic reticulum membranes

Cell death & disease, 2018-02, Vol.9 (3), p.334

2018

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GlycA, a Biomarker of Low-Grade Inflammation, Is Increased in Male Night Shift Workers

Metabolites, 2022-11, Vol.12 (12), p.1172

2022

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Exploration of genome-wide DNA methylation profiles in night shift workers

Epigenetics, 2023-12, Vol.18 (1), p.2152637-2152637

2023

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A randomized crossover trial assessing time of day snack consumption and resulting postprandial glycemic response in a real-life setting among healthy adults

Chronobiology international, 2022-10, Vol.39 (10), p.1329-1339

2022

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Measuring CMAPs in addition to MEPs can distinguish peripheral ischemia from spinal cord ischemia during endovascular aortic repair

Clinical neurophysiology practice, 2021-01, Vol.6, p.16-21

2021

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Diet, Physical Activity, and Daylight Exposure Patterns in Night-Shift Workers and Day Workers

Annals of work exposures and health, 2019-01, Vol.63 (1), p.9-21

2019

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Effects of Nightshift Work on Blood Metabolites in Female Nurses and Paramedic Staff: A Cross-sectional Study

Annals of work exposures and health, 2023-07, Vol.67 (6), p.694-705

2023

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VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like disease

Emerging molecular mechanisms in chemotherapy: Ca2+ signaling at the mitochondria-associated endoplasmic reticulum membranes

Role of Mitochondria-Associated ER Membranes in Calcium Regulation in Cancer-Specific Settings

Recent advances in uncovering the mechanisms contributing to BIRD-2-induced cell death in B-cell cancer cells

Endoplasmic Reticulum–Mitochondrial Ca2+ Fluxes Underlying Cancer Cell Survival

The AMPK-related kinase NUAK1 controls cortical axons branching by locally modulating mitochondrial metabolic functions

Endoplasmic Reticulum-Mitochondria Communication Through Ca2+ Signaling: The Importance of Mitochondria-Associated Membranes (MAMs)

Intracellular BAPTA directly inhibits PFKFB3, thereby impeding mTORC1-driven Mcl-1 translation and killing MCL-1-addicted cancer cells

DLBCL Cells with Acquired Resistance to Venetoclax Are Not Sensitized to BIRD-2 But Can Be Resensitized to Venetoclax through Bcl-XL Inhibition

Endoplasmic Reticulum-Mitochondrial Ca 2+ Fluxes Underlying Cancer Cell Survival

Recent advances in understanding IP3R function with focus on ER-mitochondrial Ca2+ transfers

ABT-199 (Venetoclax), a BH3-mimetic Bcl-2 inhibitor, does not cause Ca 2+ -signalling dysregulation or toxicity in pancreatic acinar cells

ABT‐199 (Venetoclax), a BH3‐mimetic Bcl‐2 inhibitor, does not cause Ca2+‐signalling dysregulation or toxicity in pancreatic acinar cells

VDAC oligomers form mitochondrial pores that release small mtDNA fragments and promote lupus-like disease

Role of Mitochondria-Associated ER Membranes in Calcium Regulation in Cancer-Specific Settings1

Alterations in Ca 2+ Signalling via ER-Mitochondria Contact Site Remodelling in Cancer

Alterations in Ca2+ Signalling via ER-Mitochondria Contact Site Remodelling in Cancer

Endoplasmic Reticulum-Mitochondria Communication Through Ca 2+ Signaling: The Importance of Mitochondria-Associated Membranes (MAMs)

Emerging molecular mechanisms in chemotherapy: Ca 2+ signaling at the mitochondria-associated endoplasmic reticulum membranes

GlycA, a Biomarker of Low-Grade Inflammation, Is Increased in Male Night Shift Workers

Exploration of genome-wide DNA methylation profiles in night shift workers

A randomized crossover trial assessing time of day snack consumption and resulting postprandial glycemic response in a real-life setting among healthy adults

Measuring CMAPs in addition to MEPs can distinguish peripheral ischemia from spinal cord ischemia during endovascular aortic repair

Diet, Physical Activity, and Daylight Exposure Patterns in Night-Shift Workers and Day Workers

Effects of Nightshift Work on Blood Metabolites in Female Nurses and Paramedic Staff: A Cross-sectional Study

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