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Potential therapeutic molecular targets for blood-brain barrier disruption after subarachnoid hemorrhage

Neural regeneration research, 2019-07, Vol.14 (7), p.1138-1143

2019

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Toll-like receptor 4 as a possible therapeutic target for delayed brain injuries after aneurysmal subarachnoid hemorrhage

Neural regeneration research, 2017-02, Vol.12 (2), p.193-196

2017

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Obstructive sleep apnea aggravates neuroinflammation and pyroptosis in early brain injury following subarachnoid hemorrhage via ASC/HIF-1α pathway

Neural regeneration research, 2022-11, Vol.17 (11), p.2537-2543

2022

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Neuroprotection mediated by the Wnt/Frizzled signaling pathway in early brain injury induced by subarachnoid hemorrhage

Neural regeneration research, 2019-06, Vol.14 (6), p.1013-1024

2019

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Atorvastatin ameliorates early brain injury after subarachnoid hemorrhage via inhibition of pyroptosis and neuroinflammation

Journal of cellular physiology, 2021-10, Vol.236 (10), p.6920-6931

2021

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Early Brain Injury After Poor-Grade Subarachnoid Hemorrhage

Current neurology and neuroscience reports, 2019-10, Vol.19 (10), p.78-9, Article 78

2019

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Controversies and evolving new mechanisms in subarachnoid hemorrhage

Progress in neurobiology, 2014-04, Vol.115, p.64-91

2014

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Neutrophil membrane-based biomimetic metal-polyphenol self-assembled nanozyme for the targeting treatment of early brain injury following subarachnoid hemorrhage

Chemical engineering journal (Lausanne, Switzerland : 1996), 2024-10, Vol.498, p.155643, Article 155643

2024

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Dl-3-n-Butylphthalide attenuates early brain injury and delayed neurological dysfunction by regulating NLRP3 inflammasome after subarachnoid hemorrhage

Brain research bulletin, 2024-10, Vol.217, p.111084, Article 111084

2024

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Inhibition of Ferroptosis Alleviates Early Brain Injury After Subarachnoid Hemorrhage In Vitro and In Vivo via Reduction of Lipid Peroxidation

Cellular and molecular neurobiology, 2021-03, Vol.41 (2), p.263-278

2021

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Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway

Journal of neuroinflammation, 2018-12, Vol.15 (1), p.347-347, Article 347

2018

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Apelin-13/APJ system attenuates early brain injury via suppression of endoplasmic reticulum stress-associated TXNIP/NLRP3 inflammasome activation and oxidative stress in a AMPK-dependent manner after subarachnoid hemorrhage in rats

Journal of neuroinflammation, 2019-12, Vol.16 (1), p.247-247, Article 247

2019

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The rabbit blood shunt subarachnoid haemorrhage model

Acta neurochirurgica. Supplement, 2015, Vol.120, p.337

2015

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Innate immunity activation in the early brain injury period following subarachnoid hemorrhage

Journal of neuroinflammation, 2019-12, Vol.16 (1), p.253-253, Article 253

2019

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Isoliquiritigenin alleviates early brain injury after experimental intracerebral hemorrhage via suppressing ROS- and/or NF-κB-mediated NLRP3 inflammasome activation by promoting Nrf2 antioxidant pathway

Journal of neuroinflammation, 2017-06, Vol.14 (1), p.119-119, Article 119

2017

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Tenascin‐C in brain injuries and edema after subarachnoid hemorrhage: Findings from basic and clinical studies

Journal of neuroscience research, 2020-01, Vol.98 (1), p.42-56

2020

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The importance of early brain injury after subarachnoid hemorrhage

Progress in neurobiology, 2012-04, Vol.97 (1), p.14-37

2012

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The Subarachnoid Hemorrhage Early Brain Edema Score Predicts Delayed Cerebral Ischemia and Clinical Outcomes

Neurosurgery, 2018-07, Vol.83 (1), p.137-145

2018

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The role of immune inflammation in aneurysmal subarachnoid hemorrhage

Experimental neurology, 2021-02, Vol.336, p.113535-113535, Article 113535

2021

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Mesenchymal stem cells alleviate the early brain injury of subarachnoid hemorrhage partly by suppression of Notch1-dependent neuroinflammation: involvement of Botch

Journal of neuroinflammation, 2019-01, Vol.16 (1), p.8-20, Article 8

2019

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HucMSCs-Derived miR-206-Knockdown Exosomes Contribute to Neuroprotection in Subarachnoid Hemorrhage Induced Early Brain Injury by Targeting BDNF

Neuroscience, 2019-10, Vol.417, p.11-23

2019

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TSG-6 attenuates inflammation-induced brain injury via modulation of microglial polarization in SAH rats through the SOCS3/STAT3 pathway

Journal of neuroinflammation, 2018-08, Vol.15 (1), p.231-231, Article 231

2018

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Treatment of early brain injury after subarachnoid hemorrhage in the rat model by inhibiting p53-induced ferroptosis

Neuroscience letters, 2021-09, Vol.762, p.136134-136134, Article 136134

2021

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Astaxanthin mitigates subarachnoid hemorrhage injury primarily by increasing sirtuin 1 and inhibiting the Toll‐like receptor 4 signaling pathway

The FASEB journal, 2019-01, Vol.33 (1), p.722-737

2019

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Amelioration of oxidative stress and protection against early brain injury by astaxanthin after experimental subarachnoid hemorrhage: Laboratory investigation

Journal of neurosurgery, 2014-07, Vol.121 (1), p.42-54

2014

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Potential therapeutic molecular targets for blood-brain barrier disruption after subarachnoid hemorrhage

Toll-like receptor 4 as a possible therapeutic target for delayed brain injuries after aneurysmal subarachnoid hemorrhage

Obstructive sleep apnea aggravates neuroinflammation and pyroptosis in early brain injury following subarachnoid hemorrhage via ASC/HIF-1α pathway

Neuroprotection mediated by the Wnt/Frizzled signaling pathway in early brain injury induced by subarachnoid hemorrhage

Atorvastatin ameliorates early brain injury after subarachnoid hemorrhage via inhibition of pyroptosis and neuroinflammation

Early Brain Injury After Poor-Grade Subarachnoid Hemorrhage

Controversies and evolving new mechanisms in subarachnoid hemorrhage

Neutrophil membrane-based biomimetic metal-polyphenol self-assembled nanozyme for the targeting treatment of early brain injury following subarachnoid hemorrhage

Dl-3-n-Butylphthalide attenuates early brain injury and delayed neurological dysfunction by regulating NLRP3 inflammasome after subarachnoid hemorrhage

Inhibition of Ferroptosis Alleviates Early Brain Injury After Subarachnoid Hemorrhage In Vitro and In Vivo via Reduction of Lipid Peroxidation

Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway

Apelin-13/APJ system attenuates early brain injury via suppression of endoplasmic reticulum stress-associated TXNIP/NLRP3 inflammasome activation and oxidative stress in a AMPK-dependent manner after subarachnoid hemorrhage in rats

The rabbit blood shunt subarachnoid haemorrhage model

Innate immunity activation in the early brain injury period following subarachnoid hemorrhage

Isoliquiritigenin alleviates early brain injury after experimental intracerebral hemorrhage via suppressing ROS- and/or NF-κB-mediated NLRP3 inflammasome activation by promoting Nrf2 antioxidant pathway

Tenascin‐C in brain injuries and edema after subarachnoid hemorrhage: Findings from basic and clinical studies

The importance of early brain injury after subarachnoid hemorrhage

The Subarachnoid Hemorrhage Early Brain Edema Score Predicts Delayed Cerebral Ischemia and Clinical Outcomes

The role of immune inflammation in aneurysmal subarachnoid hemorrhage

Mesenchymal stem cells alleviate the early brain injury of subarachnoid hemorrhage partly by suppression of Notch1-dependent neuroinflammation: involvement of Botch

HucMSCs-Derived miR-206-Knockdown Exosomes Contribute to Neuroprotection in Subarachnoid Hemorrhage Induced Early Brain Injury by Targeting BDNF

TSG-6 attenuates inflammation-induced brain injury via modulation of microglial polarization in SAH rats through the SOCS3/STAT3 pathway

Treatment of early brain injury after subarachnoid hemorrhage in the rat model by inhibiting p53-induced ferroptosis

Astaxanthin mitigates subarachnoid hemorrhage injury primarily by increasing sirtuin 1 and inhibiting the Toll‐like receptor 4 signaling pathway

Amelioration of oxidative stress and protection against early brain injury by astaxanthin after experimental subarachnoid hemorrhage: Laboratory investigation

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