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PERK is a critical metabolic hub for immunosuppressive function in macrophages

Nature immunology, 2022-03, Vol.23 (3), p.431-445

2022

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eIF2α kinases: their structures and functions

Cellular and molecular life sciences : CMLS, 2013-10, Vol.70 (19), p.3493-3511

2013

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Activation of the ISR mediates the behavioral and neurophysiological abnormalities in Down syndrome

Science (American Association for the Advancement of Science), 2019-11, Vol.366 (6467), p.843-849

2019

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PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation

Nature communications, 2016-10, Vol.7 (1), p.13280-13280, Article 13280

2016

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Human ADAR1 Prevents Endogenous RNA from Triggering Translational Shutdown

Cell, 2018-02, Vol.172 (4), p.811-824.e14

2018

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Carbon Monoxide Activates PERK-Regulated Autophagy to Induce Immunometabolic Reprogramming and Boost Antitumor T-cell Function

Cancer research (Chicago, Ill.), 2022-05, Vol.82 (10), p.1969-1990

2022

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The Unfolded Protein Response Mediator PERK Governs Myeloid Cell-Driven Immunosuppression in Tumors through Inhibition of STING Signaling

Immunity (Cambridge, Mass.), 2020-04, Vol.52 (4), p.668-682.e7

2020

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A cold-stress-inducible PERK/OGT axis controls TOM70-assisted mitochondrial protein import and cristae formation

Cell metabolism, 2021-03, Vol.33 (3), p.598-614.e7

2021

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The Role of the PERK/eIF2α/ATF4/CHOP Signaling Pathway in Tumor Progression During Endoplasmic Reticulum Stress

Current molecular medicine, 2016-07, Vol.16 (6), p.533-544

2016

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PERK is required at the ER-mitochondrial contact sites to convey apoptosis after ROS-based ER stress

Cell death and differentiation, 2012-11, Vol.19 (11), p.1880-1891

2012

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Novel role of PKR in inflammasome activation and HMGB1 release

Nature (London), 2012-08, Vol.488 (7413), p.670-674

2012

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Double-Stranded RNA-Dependent Protein Kinase Links Pathogen Sensing with Stress and Metabolic Homeostasis

Cell, 2010-02, Vol.140 (3), p.338-348

2010

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Suppression of eIF2α kinases alleviates Alzheimer's disease-related plasticity and memory deficits

Nature neuroscience, 2013-09, Vol.16 (9), p.1299-1305

2013

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Membrane lipid saturation activates endoplasmic reticulum unfolded protein response transducers through their transmembrane domains

Proceedings of the National Academy of Sciences - PNAS, 2013-03, Vol.110 (12), p.4628-4633

2013

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Inflammation‐Induced Long Intergenic Noncoding RNA (LINC00665) Increases Malignancy Through Activating the Double‐Stranded RNA–Activated Protein Kinase/Nuclear Factor Kappa B Pathway in Hepatocellular Carcinoma

Hepatology (Baltimore, Md.), 2020-11, Vol.72 (5), p.1666-1681

2020

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ER and Nutrient Stress Promote Assembly of Respiratory Chain Supercomplexes through the PERK-eIF2α Axis

Molecular cell, 2019-06, Vol.74 (5), p.877-890.e6

2019

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Thbs1 induces lethal cardiac atrophy through PERK-ATF4 regulated autophagy

Nature communications, 2021-06, Vol.12 (1), p.3928-3928, Article 3928

2021

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RNase L Reprograms Translation by Widespread mRNA Turnover Escaped by Antiviral mRNAs

Molecular cell, 2019-09, Vol.75 (6), p.1203-1217.e5

2019

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Domain-focused CRISPR screen identifies HRI as a fetal hemoglobin regulator in human erythroid cells

Science (American Association for the Advancement of Science), 2018-07, Vol.361 (6399), p.285-290

2018

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Exosomes from HIV-1-infected Cells Stimulate Production of Pro-inflammatory Cytokines through Trans-activating Response (TAR) RNA

The Journal of biological chemistry, 2016-01, Vol.291 (3), p.1251-1266

2016

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Signaling by the integrated stress response kinase PKR is fine-tuned by dynamic clustering

The Journal of cell biology, 2022-07, Vol.221 (7), p.1

2022

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The kinase PERK and the transcription factor ATF4 play distinct and essential roles in autophagy resulting from tunicamycin-induced ER stress

The Journal of biological chemistry, 2019-05, Vol.294 (20), p.8197-8217

2019

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PERK is a critical metabolic hub for immunosuppressive function in macrophages

eIF2α kinases: their structures and functions

Activation of the ISR mediates the behavioral and neurophysiological abnormalities in Down syndrome

PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation

Human ADAR1 Prevents Endogenous RNA from Triggering Translational Shutdown

Carbon Monoxide Activates PERK-Regulated Autophagy to Induce Immunometabolic Reprogramming and Boost Antitumor T-cell Function

The Unfolded Protein Response Mediator PERK Governs Myeloid Cell-Driven Immunosuppression in Tumors through Inhibition of STING Signaling

A cold-stress-inducible PERK/OGT axis controls TOM70-assisted mitochondrial protein import and cristae formation

The Role of the PERK/eIF2α/ATF4/CHOP Signaling Pathway in Tumor Progression During Endoplasmic Reticulum Stress

PERK is required at the ER-mitochondrial contact sites to convey apoptosis after ROS-based ER stress

Novel role of PKR in inflammasome activation and HMGB1 release

EIF2AK2 Missense Variants Associated with Early Onset Generalized Dystonia

Double-Stranded RNA-Dependent Protein Kinase Links Pathogen Sensing with Stress and Metabolic Homeostasis

Suppression of eIF2α kinases alleviates Alzheimer's disease-related plasticity and memory deficits

Membrane lipid saturation activates endoplasmic reticulum unfolded protein response transducers through their transmembrane domains

Inflammation‐Induced Long Intergenic Noncoding RNA (LINC00665) Increases Malignancy Through Activating the Double‐Stranded RNA–Activated Protein Kinase/Nuclear Factor Kappa B Pathway in Hepatocellular Carcinoma

ER and Nutrient Stress Promote Assembly of Respiratory Chain Supercomplexes through the PERK-eIF2α Axis

TNF-α Mediates PKR-Dependent Memory Impairment and Brain IRS-1 Inhibition Induced by Alzheimer’s β-Amyloid Oligomers in Mice and Monkeys

Thbs1 induces lethal cardiac atrophy through PERK-ATF4 regulated autophagy

RNase L Reprograms Translation by Widespread mRNA Turnover Escaped by Antiviral mRNAs

Domain-focused CRISPR screen identifies HRI as a fetal hemoglobin regulator in human erythroid cells

Exosomes from HIV-1-infected Cells Stimulate Production of Pro-inflammatory Cytokines through Trans-activating Response (TAR) RNA

Signaling by the integrated stress response kinase PKR is fine-tuned by dynamic clustering

The kinase PERK and the transcription factor ATF4 play distinct and essential roles in autophagy resulting from tunicamycin-induced ER stress

Ablation of the endoplasmic reticulum stress kinase PERK induces paraptosis and type I interferon to promote anti-tumor T cell responses

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