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Regional astrocyte IFN signaling restricts pathogenesis during neurotropic viral infection

The Journal of clinical investigation, 2017-03, Vol.127 (3), p.843-856

2017

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IRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling

PLoS pathogens, 2013-01, Vol.9 (1), p.e1003118-e1003118

2013

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Activation of RNase L is dependent on OAS3 expression during infection with diverse human viruses

Proceedings of the National Academy of Sciences - PNAS, 2016-02, Vol.113 (8), p.2241-2246

2016

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Infection-induced type I interferons critically modulate the homeostasis and function of CD8+ naïve T cells

Nature communications, 2021-09, Vol.12 (1), p.5303-5303, Article 5303

2021

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West Nile virus-induced disruption of the blood― brain barrier in mice is characterized by the degradation of the junctional complex proteins and increase in multiple matrix metalloproteinases

Journal of general virology, 2012-06, Vol.93 (Pt 6), p.1193-1203

2012

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IL-34 is a tissue-restricted ligand of CSF1R required for the development of Langerhans cells and microglia

Nature immunology, 2012-08, Vol.13 (8), p.753-760

2012

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RIG-I-like receptors direct inflammatory macrophage polarization against West Nile virus infection

Nature communications, 2019-08, Vol.10 (1), p.3649-16, Article 3649

2019

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Memory B cells, but not long-lived plasma cells, possess antigen specificities for viral escape mutants

The Journal of experimental medicine, 2011-12, Vol.208 (13), p.2599-2606

2011

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A single mutation in the envelope protein modulates flavivirus antigenicity, stability, and pathogenesis

PLoS pathogens, 2017-02, Vol.13 (2), p.e1006178

2017

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Flavivirus Infection Impairs Peroxisome Biogenesis and Early Antiviral Signaling

Journal of virology, 2015-12, Vol.89 (24), p.12349-12361

2015

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IL-1β signaling promotes CNS-intrinsic immune control of West Nile virus infection

PLoS pathogens, 2012-11, Vol.8 (11), p.e1003039-e1003039

2012

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VAMP8 Contributes to the TRIM6-Mediated Type I Interferon Antiviral Response during West Nile Virus Infection

Journal of virology, 2020-01, Vol.94 (2)

2020

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Persistent impacts of West Nile virus on North American bird populations

Proceedings of the National Academy of Sciences - PNAS, 2015-11, Vol.112 (46), p.14290-14294

2015

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Differential Roles of Chemokines CCL2 and CCL7 in Monocytosis and Leukocyte Migration during West Nile Virus Infection

The Journal of immunology (1950), 2015-11, Vol.195 (9), p.4306-4318

2015

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Increased genetic diversity and immigration after West Nile virus emergence in American crows: No evidence for a genetic bottleneck

Molecular ecology, 2023-08, Vol.32 (15), p.4199-4208

2023

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West Nile virus

Lancet neurology, 2007-02, Vol.6 (2), p.171-181

2007

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RNA interference screen for human genes associated with West Nile virus infection

Nature, 2008-09, Vol.455 (7210), p.242-245

2008

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The natural killer cell response to West Nile virus in young and old individuals with or without a prior history of infection

PloS one, 2017-02, Vol.12 (2), p.e0172625

2017

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Peli1 facilitates virus replication and promotes neuroinflammation during West Nile virus infection

The Journal of clinical investigation, 2018-11, Vol.128 (11), p.4980-4991

2018

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Species' life-history traits explain interspecific variation in reservoir competence: a possible mechanism underlying the dilution effect

PloS one, 2013-01, Vol.8 (1), p.e54341-e54341

2013

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Host genetic risk factors for West Nile virus infection and disease progression

PloS one, 2011-09, Vol.6 (9), p.e24745-e24745

2011

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Determination of system level alterations in host transcriptome due to Zika virus (ZIKV) Infection in retinal pigment epithelium

Scientific reports, 2018-07, Vol.8 (1), p.11209-16, Article 11209

2018

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Death Receptor-Mediated Apoptotic Signaling Is Activated in the Brain following Infection with West Nile Virus in the Absence of a Peripheral Immune Response

Journal of Virology, 2014-01, Vol.88 (2), p.1080-1089

2014

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Genetic Deficiency of Chemokine Receptor CCR5 Is a Strong Risk Factor for Symptomatic West Nile Virus Infection: A Meta-Analysis of 4 Cohorts in the US Epidemic

The Journal of infectious diseases, 2008-01, Vol.197 (2), p.262-265

2008

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Vector derived artificial miRNA mediated inhibition of West Nile virus replication and protein expression

Gene, 2020-03, Vol.729, p.144300-144300, Article 144300

2020

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Regional astrocyte IFN signaling restricts pathogenesis during neurotropic viral infection

IRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling

Activation of RNase L is dependent on OAS3 expression during infection with diverse human viruses

Infection-induced type I interferons critically modulate the homeostasis and function of CD8+ naïve T cells

West Nile virus-induced disruption of the blood― brain barrier in mice is characterized by the degradation of the junctional complex proteins and increase in multiple matrix metalloproteinases

IL-34 is a tissue-restricted ligand of CSF1R required for the development of Langerhans cells and microglia

RIG-I-like receptors direct inflammatory macrophage polarization against West Nile virus infection

Memory B cells, but not long-lived plasma cells, possess antigen specificities for viral escape mutants

A single mutation in the envelope protein modulates flavivirus antigenicity, stability, and pathogenesis

Flavivirus Infection Impairs Peroxisome Biogenesis and Early Antiviral Signaling

IL-1β signaling promotes CNS-intrinsic immune control of West Nile virus infection

VAMP8 Contributes to the TRIM6-Mediated Type I Interferon Antiviral Response during West Nile Virus Infection

Persistent impacts of West Nile virus on North American bird populations

Differential Roles of Chemokines CCL2 and CCL7 in Monocytosis and Leukocyte Migration during West Nile Virus Infection

Increased genetic diversity and immigration after West Nile virus emergence in American crows: No evidence for a genetic bottleneck

West Nile virus

RNA interference screen for human genes associated with West Nile virus infection

The natural killer cell response to West Nile virus in young and old individuals with or without a prior history of infection

Peli1 facilitates virus replication and promotes neuroinflammation during West Nile virus infection

Species' life-history traits explain interspecific variation in reservoir competence: a possible mechanism underlying the dilution effect

Host genetic risk factors for West Nile virus infection and disease progression

Determination of system level alterations in host transcriptome due to Zika virus (ZIKV) Infection in retinal pigment epithelium

Death Receptor-Mediated Apoptotic Signaling Is Activated in the Brain following Infection with West Nile Virus in the Absence of a Peripheral Immune Response

Genetic Deficiency of Chemokine Receptor CCR5 Is a Strong Risk Factor for Symptomatic West Nile Virus Infection: A Meta-Analysis of 4 Cohorts in the US Epidemic

Vector derived artificial miRNA mediated inhibition of West Nile virus replication and protein expression

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