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TGFBIp regulates differentiation of EPC (CD133+c‐kit+lin− cells) to EC through activation of the notch signaling pathway

Stem cells (Dayton, Ohio), 2015-06, Vol.33 (6), p.2052-2062

2015

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Ophthalmic drug effects on the amyloidogenesis of a transforming growth factor β-induced protein (TGFBIp) peptide fragment

Experimental eye research, 2024-07, Vol.244, p.109932, Article 109932

2024

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Mutation-induced dimerization of transforming growth factor-β–induced protein may drive protein aggregation in granular corneal dystrophy

The Journal of biological chemistry, 2021-07, Vol.297 (1), p.100858-100858, Article 100858

2021

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Biochemical mechanisms of aggregation in TGFBI-linked corneal dystrophies

Progress in retinal and eye research, 2020-07, Vol.77, p.100843-100843, Article 100843

2020

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Corneal Dystrophy Mutations Drive Pathogenesis by Targeting TGFBIp Stability and Solubility in a Latent Amyloid-forming Domain

Journal of molecular biology, 2018-04, Vol.430 (8), p.1116-1140

2018

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Pharmaceutical modulation of the proteolytic profile of Transforming Growth Factor Beta induced protein (TGFBIp) offers a new avenue for treatment of TGFBI-corneal dystrophy

Journal of advanced research, 2020-07, Vol.24, p.529-543

2020

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Proteomic profiling of TGFBI‐null mouse corneas reveals only minor changes in matrix composition supportive of TGFBI knockdown as therapy against TGFBI‐linked corneal dystrophies

The FEBS journal, 2018-01, Vol.285 (1), p.101-114

2018

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Lysosomal dysfunction of corneal fibroblasts underlies the pathogenesis of Granular Corneal Dystrophy Type 2 and can be rescued by TFEB

Journal of cellular and molecular medicine, 2020-09, Vol.24 (18), p.10343-10355

2020

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Melatonin induces autophagy via an mTOR-dependent pathway and enhances clearance of mutant-TGFBIp

Journal of pineal research, 2013-05, Vol.54 (4), p.361-372

2013

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Suppressive effects of zingerone on TGFBIp-mediated septic responses

Archives of Pharmacal Research, 2018, 41(3), , pp.276-287

2018

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The serine protease HtrA1 cleaves misfolded transforming growth factor β–induced protein (TGFBIp) and induces amyloid formation

The Journal of biological chemistry, 2019-08, Vol.294 (31), p.11817-11828

2019

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The Changing Face of Aging: Highly Sulfated Glycosaminoglycans Induce Amyloid Formation in a Lattice Corneal Dystrophy Model Protein

Journal of molecular biology, 2017-09, Vol.429 (18), p.2755-2764

2017

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Autophagy is induced by raptor degradation via the ubiquitin/proteasome system in granular corneal dystrophy type 2

Biochemical and biophysical research communications, 2014-08, Vol.450 (4), p.1505-1511

2014

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Transforming growth factor-β-induced gene product, as a novel ligand of integrin αMβ2, promotes monocytes adhesion, migration and chemotaxis

The international journal of biochemistry & cell biology, 2008-01, Vol.40 (5), p.991-1004

2008

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TGF-β regulates TGFBIp expression in corneal fibroblasts via miR-21, miR-181a, and Smad signaling

Biochemical and biophysical research communications, 2016-03, Vol.472 (1), p.150-155

2016

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Inhibitory functions of cornuside on TGFBIp-mediated septic responses

Journal of natural medicines, 2022-03, Vol.76 (2), p.451-461

2022

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Proteomic Analysis of Amyloid Corneal Aggregates from TGFBI-H626R Lattice Corneal Dystrophy Patient Implicates Serine-Protease HTRA1 in Mutation-Specific Pathogenesis of TGFBIp

Journal of proteome research, 2017-08, Vol.16 (8), p.2899-2913

2017

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Growth Hormone Induces Transforming Growth Factor-Beta-Induced Protein in Podocytes: Implications for Podocyte Depletion and Proteinuria

Journal of cellular biochemistry, 2015-09, Vol.116 (9), p.1947-1956

2015

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Mutation in transforming growth factor beta induced protein associated with granular corneal dystrophy type 1 reduces the proteolytic susceptibility through local structural stabilization

Biochimica et biophysica acta, 2013-12, Vol.1834 (12), p.2812-2822

2013

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Transforming growth factor‐beta‐induced protein secreted by peritoneal cells increases the metastatic potential of ovarian cancer cells

International journal of cancer, 2011-04, Vol.128 (7), p.1570-1584

2011

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Distinct ocular surface soluble factor profile in human corneal dystrophies

The ocular surface, 2020-04, Vol.18 (2), p.237-248

2020

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Autophagy in granular corneal dystrophy type 2

Experimental eye research, 2016-03, Vol.144, p.14-21

2016

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Inhibitory effects of aloin on TGFBIp-mediated septic responses

Journal of asian natural products research, 2021-02, Vol.23 (2), p.189-203

2021

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4-Phenylbutyric acid reduces mutant-TGFBIp levels and ER stress through activation of ERAD pathway in corneal fibroblasts of granular corneal dystrophy type 2

Biochemical and biophysical research communications, 2016-09, Vol.477 (4), p.841-846

2016

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Role of TGFBIp in Wound Healing and Mucin Expression in Corneal Epithelial Cells

Yonsei Medical Journal, 2017, 58(2), , pp.423-431

2017

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TGFBIp regulates differentiation of EPC (CD133+c‐kit+lin− cells) to EC through activation of the notch signaling pathway

Ophthalmic drug effects on the amyloidogenesis of a transforming growth factor β-induced protein (TGFBIp) peptide fragment

Mutation-induced dimerization of transforming growth factor-β–induced protein may drive protein aggregation in granular corneal dystrophy

Biochemical mechanisms of aggregation in TGFBI-linked corneal dystrophies

Corneal Dystrophy Mutations Drive Pathogenesis by Targeting TGFBIp Stability and Solubility in a Latent Amyloid-forming Domain

Pharmaceutical modulation of the proteolytic profile of Transforming Growth Factor Beta induced protein (TGFBIp) offers a new avenue for treatment of TGFBI-corneal dystrophy

Proteomic profiling of TGFBI‐null mouse corneas reveals only minor changes in matrix composition supportive of TGFBI knockdown as therapy against TGFBI‐linked corneal dystrophies

Lysosomal dysfunction of corneal fibroblasts underlies the pathogenesis of Granular Corneal Dystrophy Type 2 and can be rescued by TFEB

Melatonin induces autophagy via an mTOR-dependent pathway and enhances clearance of mutant-TGFBIp

Suppressive effects of zingerone on TGFBIp-mediated septic responses

The serine protease HtrA1 cleaves misfolded transforming growth factor β–induced protein (TGFBIp) and induces amyloid formation

The Changing Face of Aging: Highly Sulfated Glycosaminoglycans Induce Amyloid Formation in a Lattice Corneal Dystrophy Model Protein

Autophagy is induced by raptor degradation via the ubiquitin/proteasome system in granular corneal dystrophy type 2

Transforming growth factor-β-induced gene product, as a novel ligand of integrin αMβ2, promotes monocytes adhesion, migration and chemotaxis

TGF-β regulates TGFBIp expression in corneal fibroblasts via miR-21, miR-181a, and Smad signaling

Inhibitory functions of cornuside on TGFBIp-mediated septic responses

Proteomic Analysis of Amyloid Corneal Aggregates from TGFBI-H626R Lattice Corneal Dystrophy Patient Implicates Serine-Protease HTRA1 in Mutation-Specific Pathogenesis of TGFBIp

Growth Hormone Induces Transforming Growth Factor-Beta-Induced Protein in Podocytes: Implications for Podocyte Depletion and Proteinuria

Mutation in transforming growth factor beta induced protein associated with granular corneal dystrophy type 1 reduces the proteolytic susceptibility through local structural stabilization

Transforming growth factor‐beta‐induced protein secreted by peritoneal cells increases the metastatic potential of ovarian cancer cells

Distinct ocular surface soluble factor profile in human corneal dystrophies

Autophagy in granular corneal dystrophy type 2

Inhibitory effects of aloin on TGFBIp-mediated septic responses

4-Phenylbutyric acid reduces mutant-TGFBIp levels and ER stress through activation of ERAD pathway in corneal fibroblasts of granular corneal dystrophy type 2

Role of TGFBIp in Wound Healing and Mucin Expression in Corneal Epithelial Cells

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