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Systemic LPS causes chronic neuroinflammation and progressive neurodegeneration

Glia, 2007-04, Vol.55 (5), p.453-462

2007

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Intrastriatal injection of preformed alpha-synuclein fibrils alters central and peripheral immune cell profiles in non-transgenic mice

Journal of neuroinflammation, 2019-12, Vol.16 (1), p.250-250, Article 250

2019

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RANTES-induced invasion of Th17 cells into substantia nigra potentiates dopaminergic cell loss in MPTP mouse model of Parkinson's disease

Neurobiology of disease, 2019-12, Vol.132, p.104575-104575, Article 104575

2019

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Evidence for Immune Response, Axonal Dysfunction and Reduced Endocytosis in the Substantia Nigra in Early Stage Parkinson's Disease

PloS one, 2015-06, Vol.10 (6), p.e0128651-e0128651

2015

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Glial reactions in Parkinson's disease

Movement disorders, 2008-03, Vol.23 (4), p.474-483

2008

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Targeted Overexpression of Human α-Synuclein Triggers Microglial Activation and an Adaptive Immune Response in a Mouse Model of Parkinson Disease

Journal of neuropathology and experimental neurology, 2008-12, Vol.67 (12), p.1149-1158

2008

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Neutralization of RANTES and Eotaxin Prevents the Loss of Dopaminergic Neurons in a Mouse Model of Parkinson Disease

The Journal of biological chemistry, 2016-07, Vol.291 (29), p.15267-15281

2016

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Neuroinflammation and α-Synuclein Dysfunction Potentiate Each Other, Driving Chronic Progression of Neurodegeneration in a Mouse Model of Parkinson's Disease

Environmental health perspectives, 2011-06, Vol.119 (6), p.807-814

2011

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Increase in blood-brain barrier permeability, oxidative stress, and activated microglia in a rat model of blast-induced traumatic brain injury

Journal of neuroscience research, 2010-12, Vol.88 (16), p.3530-3539

2010

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Dopamine receptor D3 expressed on CD4+ T cells favors neurodegeneration of dopaminergic neurons during Parkinson's disease

The Journal of immunology (1950), 2013-05, Vol.190 (10), p.5048-5056

2013

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Flavin-containing monooxygenase 1 deficiency promotes neuroinflammation in dopaminergic neurons in mice

Neuroscience letters, 2021-11, Vol.764, p.136222-136222, Article 136222

2021

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Exendin-4 protects dopaminergic neurons by inhibition of microglial activation and matrix metalloproteinase-3 expression in an animal model of Parkinson's disease

Journal of endocrinology, 2009-09, Vol.202 (3), p.431-439

2009

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The acute inflammatory response to intranigral α-synuclein differs significantly from intranigral lipopolysaccharide and is exacerbated by peripheral inflammation

Journal of neuroinflammation, 2011-11, Vol.8 (1), p.166-166, Article 166

2011

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Induction of Adaptive Immunity Leads to Nigrostriatal Disease Progression in MPTP Mouse Model of Parkinson's Disease

The Journal of immunology (1950), 2017-06, Vol.198 (11), p.4312-4326

2017

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Cannabinoid receptor type 1 protects nigrostriatal dopaminergic neurons against MPTP neurotoxicity by inhibiting microglial activation

The Journal of immunology (1950), 2011-12, Vol.187 (12), p.6508-6517

2011

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HMGB1 A box protects neurons by potently inhibiting both microglia and T cell-mediated inflammation in a mouse Parkinson's disease model

Clinical science (1979), 2020-08, Vol.134 (15), p.2075-2090

2020

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Intranasal PRGF-Endoret enhances neuronal survival and attenuates NF-κB-dependent inflammation process in a mouse model of Parkinson's disease

Journal of controlled release, 2015-04, Vol.203, p.170-180

2015

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α-Synuclein Vaccination Prevents the Accumulation of Parkinson Disease–Like Pathologic Inclusions in Striatum in Association With Regulatory T Cell Recruitment in a Rat Model

Journal of neuropathology and experimental neurology, 2013-07, Vol.72 (7), p.624-645

2013

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Distinct alterations in motor & reward seeking behavior are dependent on the gestational age of exposure to LPS-induced maternal immune activation

Brain, behavior, and immunity, 2017-07, Vol.63, p.21-34

2017

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Schisandrin A ameliorates MPTP-induced Parkinson’s disease in a mouse model via regulation of brain autophagy

Archives of Pharmacal Research, 2019, 42(11), , pp.1012-1020

2019

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Chronic expression of low levels of tumor necrosis factor-α in the substantia nigra elicits progressive neurodegeneration, delayed motor symptoms and microglia/macrophage activation

Neurobiology of disease, 2010-03, Vol.37 (3), p.630-640

2010

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Peli1 controls the survival of dopaminergic neurons through modulating microglia-mediated neuroinflammation

Scientific reports, 2019-05, Vol.9 (1), p.8034, Article 8034

2019

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CB2 receptor activation prevents glial-derived neurotoxic mediator production, BBB leakage and peripheral immune cell infiltration and rescues dopamine neurons in the MPTP model of Parkinson’s disease

Experimental & molecular medicine, 2016-01, Vol.48 (1), p.e205-e205

2016

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Inflammatory response in Parkinson's disease (Review)

Molecular medicine reports, 2014-11, Vol.10 (5), p.2223-2233

2014

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Interaction between NADPH-oxidase and Rho-kinase in angiotensin II-induced microglial activation

Glia, 2015-03, Vol.63 (3), p.466-482

2015

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Systemic LPS causes chronic neuroinflammation and progressive neurodegeneration

Intrastriatal injection of preformed alpha-synuclein fibrils alters central and peripheral immune cell profiles in non-transgenic mice

RANTES-induced invasion of Th17 cells into substantia nigra potentiates dopaminergic cell loss in MPTP mouse model of Parkinson's disease

Evidence for Immune Response, Axonal Dysfunction and Reduced Endocytosis in the Substantia Nigra in Early Stage Parkinson's Disease

Glial reactions in Parkinson's disease

Targeted Overexpression of Human α-Synuclein Triggers Microglial Activation and an Adaptive Immune Response in a Mouse Model of Parkinson Disease

Neutralization of RANTES and Eotaxin Prevents the Loss of Dopaminergic Neurons in a Mouse Model of Parkinson Disease

Neuroinflammation and α-Synuclein Dysfunction Potentiate Each Other, Driving Chronic Progression of Neurodegeneration in a Mouse Model of Parkinson's Disease

Increase in blood-brain barrier permeability, oxidative stress, and activated microglia in a rat model of blast-induced traumatic brain injury

Dopamine receptor D3 expressed on CD4+ T cells favors neurodegeneration of dopaminergic neurons during Parkinson's disease

Flavin-containing monooxygenase 1 deficiency promotes neuroinflammation in dopaminergic neurons in mice

Exendin-4 protects dopaminergic neurons by inhibition of microglial activation and matrix metalloproteinase-3 expression in an animal model of Parkinson's disease

The acute inflammatory response to intranigral α-synuclein differs significantly from intranigral lipopolysaccharide and is exacerbated by peripheral inflammation

Induction of Adaptive Immunity Leads to Nigrostriatal Disease Progression in MPTP Mouse Model of Parkinson's Disease

Cannabinoid receptor type 1 protects nigrostriatal dopaminergic neurons against MPTP neurotoxicity by inhibiting microglial activation

HMGB1 A box protects neurons by potently inhibiting both microglia and T cell-mediated inflammation in a mouse Parkinson's disease model

Intranasal PRGF-Endoret enhances neuronal survival and attenuates NF-κB-dependent inflammation process in a mouse model of Parkinson's disease

α-Synuclein Vaccination Prevents the Accumulation of Parkinson Disease–Like Pathologic Inclusions in Striatum in Association With Regulatory T Cell Recruitment in a Rat Model

Distinct alterations in motor & reward seeking behavior are dependent on the gestational age of exposure to LPS-induced maternal immune activation

Schisandrin A ameliorates MPTP-induced Parkinson’s disease in a mouse model via regulation of brain autophagy

Chronic expression of low levels of tumor necrosis factor-α in the substantia nigra elicits progressive neurodegeneration, delayed motor symptoms and microglia/macrophage activation

Peli1 controls the survival of dopaminergic neurons through modulating microglia-mediated neuroinflammation

CB2 receptor activation prevents glial-derived neurotoxic mediator production, BBB leakage and peripheral immune cell infiltration and rescues dopamine neurons in the MPTP model of Parkinson’s disease

Inflammatory response in Parkinson's disease (Review)

Interaction between NADPH-oxidase and Rho-kinase in angiotensin II-induced microglial activation

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