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Diabetes-associated cardiac fibrosis: Cellular effectors, molecular mechanisms and therapeutic opportunities

Journal of molecular and cellular cardiology, 2016-01, Vol.90, p.84-93

2016

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Advanced glycation end products and their receptor in age-related, non-communicable chronic inflammatory diseases; Overview of clinical evidence and potential contributions to disease

The international journal of biochemistry & cell biology, 2016-12, Vol.81 (Pt B), p.403-418

2016

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Advanced glycation end products (AGEs) and other adducts in aging-related diseases and alcohol-mediated tissue injury

Experimental and Molecular Medicine, 2021, 53(0), , pp.1-21

2021

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RAGE-Aptamer Blocks the Development and Progression of Experimental Diabetic Nephropathy

Diabetes (New York, N.Y.), 2017-06, Vol.66 (6), p.1683-1695

2017

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Persistent Increase in Microglial RAGE Contributes to Chronic Stress–Induced Priming of Depressive-like Behavior

Biological psychiatry (1969), 2018-01, Vol.83 (1), p.50-60

2018

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High Mobility Group Box Protein 1 (HMGB1): The Prototypical Endogenous Danger Molecule

Molecular medicine (Cambridge, Mass.), 2015-01, Vol.21 Suppl 1 (S1), p.S6-S12

2015

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Hyperglycemia-associated alterations in cellular signaling and dysregulated mitochondrial bioenergetics in human metabolic disorders

European Journal of Nutrition, 2016-12, Vol.55 (8), p.2339-2345

2016

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Interplay between RAGE and TLR4 Regulates HMGB1-Induced Inflammation by Promoting Cell Surface Expression of RAGE and TLR4

The Journal of immunology (1950), 2020-08, Vol.205 (3), p.767-775

2020

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Matrine inhibits advanced glycation end products-induced macrophage M1 polarization by reducing DNMT3a/b-mediated DNA methylation of GPX1 promoter

European journal of pharmacology, 2022-07, Vol.926, p.175039-175039, Article 175039

2022

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AGE/RAGE signaling-mediated endoplasmic reticulum stress and future prospects in non-coding RNA therapeutics for diabetic nephropathy

Biomedicine & pharmacotherapy, 2020-11, Vol.131, p.110655-110655, Article 110655

2020

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Advanced glycation end products (AGEs) and cardiovascular dysfunction: focus on high molecular weight AGEs

Amino acids, 2017-09, Vol.49 (9), p.1535-1541

2017

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Advanced Glycation End Products Enhance Macrophages Polarization into M1 Phenotype through Activating RAGE/NF-κB Pathway

BioMed research international, 2015-01, Vol.2015 (2015), p.1-12

2015

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Hypoxia Driven Glycation: Mechanisms and Therapeutic Opportunities

Seminars in cancer biology, 2018-04, Vol.49, p.75-82

2018

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Structural Basis for Pattern Recognition by the Receptor for Advanced Glycation End Products (RAGE)

The Journal of biological chemistry, 2008-10, Vol.283 (40), p.27255-27269

2008

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HMGB1 promotes ductular reaction and tumorigenesis in autophagy-deficient livers

The Journal of clinical investigation, 2018-06, Vol.128 (6), p.2419-2435

2018

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Hyperglycemia-Induced Reactive Oxygen Species Increase Expression of the Receptor for Advanced Glycation End Products (RAGE) and RAGE Ligands

Diabetes (New York, N.Y.), 2010-01, Vol.59 (1), p.249-255

2010

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Generation, biochemical characterizations and validation of potent nanobodies derived from alpaca specific for human receptor of advanced glycation end product

Biochemical and biophysical research communications, 2021-12, Vol.581, p.38-45

2021

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Advanced Glycation End Products: A Molecular Target for Vascular Complications in Diabetes

Molecular medicine (Cambridge, Mass.), 2015-10, Vol.21 Suppl 1 (S1), p.S32-S40

2015

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RAGE Enhances TLR Responses through Binding and Internalization of RNA

The Journal of immunology (1950), 2016-11, Vol.197 (10), p.4118-4126

2016

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The HMGB1/RAGE inflammatory pathway promotes pancreatic tumor growth by regulating mitochondrial bioenergetics

Oncogene, 2014-01, Vol.33 (5), p.567-577

2014

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Early- and life-long intake of dietary advanced glycation end-products (dAGEs) leads to transient tissue accumulation, increased gut sensitivity to inflammation, and slight changes in gut microbial diversity, without causing overt disease

Food research international, 2024-11, Vol.195, p.114967, Article 114967

2024

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Long-term intake of the reactive metabolite methylglyoxal is not toxic in mice

Food and chemical toxicology, 2020-07, Vol.141, p.111333, Article 111333

2020

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Advanced glycation end products receptor RAGE controls myocardial dysfunction and oxidative stress in high-fat fed mice by sustaining mitochondrial dynamics and autophagy-lysosome pathway

Free radical biology & medicine, 2017-11, Vol.112, p.397-410

2017

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Diabetes-associated cardiac fibrosis: Cellular effectors, molecular mechanisms and therapeutic opportunities

Advanced glycation end products and their receptor in age-related, non-communicable chronic inflammatory diseases; Overview of clinical evidence and potential contributions to disease

Advanced glycation end products (AGEs) and other adducts in aging-related diseases and alcohol-mediated tissue injury

RAGE-Aptamer Blocks the Development and Progression of Experimental Diabetic Nephropathy

Persistent Increase in Microglial RAGE Contributes to Chronic Stress–Induced Priming of Depressive-like Behavior

High Mobility Group Box Protein 1 (HMGB1): The Prototypical Endogenous Danger Molecule

Disulfide HMGB1 derived from platelets coordinates venous thrombosis in mice

Hyperglycemia-associated alterations in cellular signaling and dysregulated mitochondrial bioenergetics in human metabolic disorders

Interplay between RAGE and TLR4 Regulates HMGB1-Induced Inflammation by Promoting Cell Surface Expression of RAGE and TLR4

Glucagon-like peptide-1 receptor signaling modifies the extent of diabetic kidney disease through dampening the receptor for advanced glycation end products–induced inflammation

Matrine inhibits advanced glycation end products-induced macrophage M1 polarization by reducing DNMT3a/b-mediated DNA methylation of GPX1 promoter

AGE/RAGE signaling-mediated endoplasmic reticulum stress and future prospects in non-coding RNA therapeutics for diabetic nephropathy

Advanced glycation end products (AGEs) and cardiovascular dysfunction: focus on high molecular weight AGEs

Advanced Glycation End Products Enhance Macrophages Polarization into M1 Phenotype through Activating RAGE/NF-κB Pathway

Hypoxia Driven Glycation: Mechanisms and Therapeutic Opportunities

Structural Basis for Pattern Recognition by the Receptor for Advanced Glycation End Products (RAGE)

HMGB1 promotes ductular reaction and tumorigenesis in autophagy-deficient livers

Hyperglycemia-Induced Reactive Oxygen Species Increase Expression of the Receptor for Advanced Glycation End Products (RAGE) and RAGE Ligands

Generation, biochemical characterizations and validation of potent nanobodies derived from alpaca specific for human receptor of advanced glycation end product

Advanced Glycation End Products: A Molecular Target for Vascular Complications in Diabetes

RAGE Enhances TLR Responses through Binding and Internalization of RNA

The HMGB1/RAGE inflammatory pathway promotes pancreatic tumor growth by regulating mitochondrial bioenergetics

Early- and life-long intake of dietary advanced glycation end-products (dAGEs) leads to transient tissue accumulation, increased gut sensitivity to inflammation, and slight changes in gut microbial diversity, without causing overt disease

Long-term intake of the reactive metabolite methylglyoxal is not toxic in mice

Advanced glycation end products receptor RAGE controls myocardial dysfunction and oxidative stress in high-fat fed mice by sustaining mitochondrial dynamics and autophagy-lysosome pathway

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