UB Paderborn / Katalog / Suche / in CDI

Common PIEZO1 Allele in African Populations Causes RBC Dehydration and Attenuates Plasmodium Infection

Cell, 2018-04, Vol.173 (2), p.443-455.e12

2018

Link zum Volltext

Verfügbarkeit prüfen

Calcium-activated potassium channels BK and IK1 are functionally expressed in human gliomas but do not regulate cell proliferation

PloS one, 2010-08, Vol.5 (8), p.e12304

2010

Link zum Volltext

Verfügbarkeit prüfen

Positive feedback regulation of agonist-stimulated endothelial Ca2+ dynamics by KCa3.1 channels in mouse mesenteric arteries

Arteriosclerosis, thrombosis, and vascular biology, 2014-01, Vol.34 (1), p.127

2014

Link zum Volltext

Verfügbarkeit prüfen

Inhibition of the K⁺ channel KCa3.1 ameliorates T cell-mediated colitis

Proceedings of the National Academy of Sciences - PNAS, 2010-01, Vol.107 (4), p.1541-1546

2010

Link zum Volltext

Verfügbarkeit prüfen

Targeted inhibition of KCa3.1 attenuates TGF‐β‐induced reactive astrogliosis through the Smad2/3 signaling pathway

Journal of neurochemistry, 2014-07, Vol.130 (1), p.41-49

2014

Link zum Volltext

Verfügbarkeit prüfen

Combined genetic disruption of K-Cl cotransporters and Gardos channel KCNN4 rescues erythrocyte dehydration in the SAD mouse model of sickle cell disease

Blood cells, molecules, & diseases, 2019-11, Vol.79, p.102346-102346, Article 102346

2019

Link zum Volltext

Verfügbarkeit prüfen

Blockade of KCa3.1 potassium channels protects against cisplatin-induced acute kidney injury

Archives of toxicology, 2016-09, Vol.90 (9), p.2249-2260

2016

Link zum Volltext

Verfügbarkeit prüfen

Genetic KCa3.1-deficiency produces locomotor hyperactivity and alterations in cerebral monoamine levels

PloS one, 2012-10, Vol.7 (10), p.e47744-e47744

2012

Link zum Volltext

Verfügbarkeit prüfen

Pulmonary hypertension in wild type mice and animals with genetic deficit in KCa2.3 and KCa3.1 channels

PloS one, 2014-05, Vol.9 (5), p.e97687

2014

Link zum Volltext

Verfügbarkeit prüfen

Kcnn4 is a modifier gene of intestinal cystic fibrosis preventing lethality in the Cftr-F508del mouse

Scientific reports, 2018-06, Vol.8 (1), p.9320-12, Article 9320

2018

Link zum Volltext

Verfügbarkeit prüfen

Impaired Endothelium-Derived Hyperpolarizing Factor–Mediated Dilations and Increased Blood Pressure in Mice Deficient of the Intermediate-Conductance Ca2+-Activated K+ Channel

Circulation research, 2006-09, Vol.99 (5), p.537-544

2006

Link zum Volltext

Verfügbarkeit prüfen

Regulation of membrane potential and fluid secretion by Ca2+-activated K+ channels in mouse submandibular glands

The Journal of physiology, 2007-06, Vol.581 (2), p.801-817

2007

Link zum Volltext

Verfügbarkeit prüfen

Nucleoside diphosphate kinase B-activated intermediate conductance potassium channels are critical for neointima formation in mouse carotid arteries

Arteriosclerosis, thrombosis, and vascular biology, 2015-08, Vol.35 (8), p.1852

2015

Link zum Volltext

Verfügbarkeit prüfen

P2Y2 receptor activation decreases blood pressure via intermediate conductance potassium channels and connexin 37

Acta Physiologica, 2015-03, Vol.213 (3), p.628-641

2015

Link zum Volltext

Verfügbarkeit prüfen

Control of hypothalamic–pituitary–adrenal stress axis activity by the intermediate conductance calcium‐activated potassium channel, SK4

The Journal of physiology, 2011-12, Vol.589 (24), p.5965-5986

2011

Link zum Volltext

Verfügbarkeit prüfen

The potassium channel KCa3.1 as new therapeutic target for the prevention of obliterative airway disease

Transplantation, 2013-01, Vol.95 (2), p.285

2013

Link zum Volltext

Verfügbarkeit prüfen

High glucose induces CCL20 in proximal tubular cells via activation of the KCa3.1 channel

PloS one, 2014-04, Vol.9 (4), p.e95173

2014

Link zum Volltext

Verfügbarkeit prüfen

Abolition of Ca2+-mediated intestinal anion secretion and increased stool dehydration in mice lacking the intermediate conductance Ca2+-dependent K+ channel Kcnn4

The Journal of physiology, 2007-09, Vol.583 (2), p.705-717

2007

Link zum Volltext

Verfügbarkeit prüfen

Enhanced Glucose Tolerance by SK4 Channel Inhibition in Pancreatic β-Cells

Diabetes (New York, N.Y.), 2009-08, Vol.58 (8), p.1835-1843

2009

Link zum Volltext

Verfügbarkeit prüfen

Lipopolysaccharide induces cell volume increase and migration of dendritic cells

Microbiology and immunology, 2014-01, Vol.58 (1), p.61-67

2014

Link zum Volltext

Verfügbarkeit prüfen

Disruption of the Gardos channel (KCa3.1) in mice causes subtle erythrocyte macrocytosis and progressive splenomegaly

Pflügers Archiv, 2009-06, Vol.458 (2), p.291-302

2009

Link zum Volltext

Verfügbarkeit prüfen

The role of cell cholesterol and the cytoskeleton in the interaction between IK1 and maxi-K channels

American Journal of Physiology: Cell Physiology, 2009-04, Vol.296 (4), p.C878-C888

2009

Link zum Volltext

Verfügbarkeit prüfen

K Ca 3.1 channels modulate the processing of noxious chemical stimuli in mice

Neuropharmacology, 2017-10, Vol.125, p.386

2017

Link zum Volltext

Verfügbarkeit prüfen

Functional significance of the intermediate conductance Ca2+-activated K+ channel for the short-term survival of injured erythrocytes

Pflügers Archiv, 2010-11, Vol.460 (6), p.1029-1044

2010

Link zum Volltext

Verfügbarkeit prüfen

Blunted IgE-Mediated Activation of Mast Cells in Mice Lacking the Ca2+-Activated K+ Channel KCa3.1

The Journal of immunology (1950), 2008-06, Vol.180 (12), p.8040-8047

2008

Link zum Volltext

Verfügbarkeit prüfen

Menü

Weitere Dienste

Einstellungen

Common PIEZO1 Allele in African Populations Causes RBC Dehydration and Attenuates Plasmodium Infection

Calcium-activated potassium channels BK and IK1 are functionally expressed in human gliomas but do not regulate cell proliferation

Positive feedback regulation of agonist-stimulated endothelial Ca2+ dynamics by KCa3.1 channels in mouse mesenteric arteries

Inhibition of the K⁺ channel KCa3.1 ameliorates T cell-mediated colitis

Targeted inhibition of KCa3.1 attenuates TGF‐β‐induced reactive astrogliosis through the Smad2/3 signaling pathway

Combined genetic disruption of K-Cl cotransporters and Gardos channel KCNN4 rescues erythrocyte dehydration in the SAD mouse model of sickle cell disease

Blockade of KCa3.1 potassium channels protects against cisplatin-induced acute kidney injury

Genetic KCa3.1-deficiency produces locomotor hyperactivity and alterations in cerebral monoamine levels

Pulmonary hypertension in wild type mice and animals with genetic deficit in KCa2.3 and KCa3.1 channels

Kcnn4 is a modifier gene of intestinal cystic fibrosis preventing lethality in the Cftr-F508del mouse

Impaired Endothelium-Derived Hyperpolarizing Factor–Mediated Dilations and Increased Blood Pressure in Mice Deficient of the Intermediate-Conductance Ca2+-Activated K+ Channel

Regulation of membrane potential and fluid secretion by Ca2+-activated K+ channels in mouse submandibular glands

Nucleoside diphosphate kinase B-activated intermediate conductance potassium channels are critical for neointima formation in mouse carotid arteries

P2Y2 receptor activation decreases blood pressure via intermediate conductance potassium channels and connexin 37

Control of hypothalamic–pituitary–adrenal stress axis activity by the intermediate conductance calcium‐activated potassium channel, SK4

The potassium channel KCa3.1 as new therapeutic target for the prevention of obliterative airway disease

High glucose induces CCL20 in proximal tubular cells via activation of the KCa3.1 channel

Abolition of Ca2+-mediated intestinal anion secretion and increased stool dehydration in mice lacking the intermediate conductance Ca2+-dependent K+ channel Kcnn4

Enhanced Glucose Tolerance by SK4 Channel Inhibition in Pancreatic β-Cells

Lipopolysaccharide induces cell volume increase and migration of dendritic cells

Disruption of the Gardos channel (KCa3.1) in mice causes subtle erythrocyte macrocytosis and progressive splenomegaly

The role of cell cholesterol and the cytoskeleton in the interaction between IK1 and maxi-K channels

K Ca 3.1 channels modulate the processing of noxious chemical stimuli in mice

Functional significance of the intermediate conductance Ca2+-activated K+ channel for the short-term survival of injured erythrocytes

Blunted IgE-Mediated Activation of Mast Cells in Mice Lacking the Ca2+-Activated K+ Channel KCa3.1

Suchergebnisse filtern