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Structure of APP-C991–99 and implications for role of extra-membrane domains in function and oligomerization

Biochimica et biophysica acta. Biomembranes, 2018-09, Vol.1860 (9), p.1698-1708

2018

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Genetic characterization and molecular epidemiological analysis of enterovirus C99 in China

Journal of medical virology, 2024-02, Vol.96 (2), p.e29449-n/a

2024

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Is γ-secretase a beneficial inactivating enzyme of the toxic APP C-terminal fragment C99?

The Journal of biological chemistry, 2021-01, Vol.296, p.100489, Article 100489

2021

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Gathering Data for Archival, Field, Survey, and Experimental Accounting Research

Journal of accounting research, 2016-05, Vol.54 (2), p.341-395

2016

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Intraneuronal accumulation of C99 contributes to synaptic alterations, apathy-like behavior, and spatial learning deficits in 3×TgAD and 2×TgAD mice

Neurobiology of aging, 2018-11, Vol.71, p.21-31

2018

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Increased localization of APP‐C99 in mitochondria‐associated ER membranes causes mitochondrial dysfunction in Alzheimer disease

The EMBO journal, 2017-11, Vol.36 (22), p.3356-3371

2017

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Amyloid precursor protein products concentrate in a subset of exosomes specifically endocytosed by neurons

Cellular and molecular life sciences : CMLS, 2018-02, Vol.75 (4), p.757-773

2018

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C99 selectively accumulates in vulnerable neurons in Alzheimer's disease

Alzheimer's & dementia, 2020-02, Vol.16 (2), p.273-282

2020

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Intraneuronal aggregation of the β-CTF fragment of APP (C99) induces Aβ-independent lysosomal-autophagic pathology

Acta neuropathologica, 2016-08, Vol.132 (2), p.257-276

2016

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The silence of the fats: A MAM’s story about Alzheimer

Neurobiology of disease, 2020-11, Vol.145, p.105062-105062, Article 105062

2020

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ErbB2 regulates autophagic flux to modulate the proteostasis of APP-CTFs in Alzheimer’s disease

Proceedings of the National Academy of Sciences - PNAS, 2017-04, Vol.114 (15), p.E3129-E3138

2017

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Work Motivation under Communist Rule: Heritage from the Past in Modern Public Sector Organisations

Central European economic journal., 2024-07, Vol.11 (58), p.269-285

Prysmakova, Palina

2024

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Does Intraneuronal Accumulation of Carboxyl-terminal Fragments of the Amyloid Precursor Protein Trigger Early Neurotoxicity in Alzheimer’s Disease?

Current Alzheimer research, 2019-01, Vol.16 (5), p.453-457

2019

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Increasing the Survival of a Neuronal Model of Alzheimer’s Disease Using Docosahexaenoic Acid, Restoring Endolysosomal Functioning by Modifying the Interactions between the Membrane Proteins C99 and Rab5

International journal of molecular sciences, 2024-07, Vol.25 (13), p.6816

2024

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Cholesterol as a co‐solvent and a ligand for membrane proteins

Protein science, 2014-01, Vol.23 (1), p.1-22

2014

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Economic Costs of Civil Conflicts: The Case of Burundi

Defence and peace economics, 2023-07, Vol.ahead-of-print (ahead-of-print), p.1-24

2023

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The transmembrane amyloid precursor C99 protein exhibits non-specific interaction with tau

Biochemical and biophysical research communications, 2021-10, Vol.576, p.48-52

2021

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Taking risks by flying paper airplanes

Frontiers in education (Lausanne), 2024-01, Vol.9

Alfonso, Antonio

2024

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The vexing complexity of the amyloidogenic pathway

Protein science, 2019-07, Vol.28 (7), p.1177-1193

2019

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Towards a Unitary Hypothesis of Alzheimer's Disease Pathogenesis

Journal of Alzheimer's disease, 2024-01, Vol.98 (4), p.1243-1275

2024

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The Transcription Factor EB Reduces the Intraneuronal Accumulation of the Beta-Secretase-Derived APP Fragment C99 in Cellular and Mouse AD Models

Cells (Basel, Switzerland), 2020-05, Vol.9 (5)

2020

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Do preferences for urban amenities differ by skill?

Journal of economic geography, 2023-05, Vol.23 (3), p.541-576

2023

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Perceptions of inevitability and demand for redistribution: Evidence from a survey experiment

Journal of economic behavior & organization, 2019-03, Vol.159, p.274-288

2019

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MT5-MMP promotes neuroinflammation, neuronal excitability and Aβ production in primary neuron/astrocyte cultures from the 5xFAD mouse model of Alzheimer's disease

Journal of neuroinflammation, 2022-03, Vol.19 (1), p.65-65, Article 65

2022

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Structure of APP-C991–99 and implications for role of extra-membrane domains in function and oligomerization

Genetic characterization and molecular epidemiological analysis of enterovirus C99 in China

Is γ-secretase a beneficial inactivating enzyme of the toxic APP C-terminal fragment C99?

Gathering Data for Archival, Field, Survey, and Experimental Accounting Research

Intraneuronal accumulation of C99 contributes to synaptic alterations, apathy-like behavior, and spatial learning deficits in 3×TgAD and 2×TgAD mice

Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer’s disease models and human brains

Increased localization of APP‐C99 in mitochondria‐associated ER membranes causes mitochondrial dysfunction in Alzheimer disease

Amyloid precursor protein products concentrate in a subset of exosomes specifically endocytosed by neurons

C99 selectively accumulates in vulnerable neurons in Alzheimer's disease

Intraneuronal aggregation of the β-CTF fragment of APP (C99) induces Aβ-independent lysosomal-autophagic pathology

The silence of the fats: A MAM’s story about Alzheimer

ErbB2 regulates autophagic flux to modulate the proteostasis of APP-CTFs in Alzheimer’s disease

Work Motivation under Communist Rule: Heritage from the Past in Modern Public Sector Organisations

Does Intraneuronal Accumulation of Carboxyl-terminal Fragments of the Amyloid Precursor Protein Trigger Early Neurotoxicity in Alzheimer’s Disease?

Increasing the Survival of a Neuronal Model of Alzheimer’s Disease Using Docosahexaenoic Acid, Restoring Endolysosomal Functioning by Modifying the Interactions between the Membrane Proteins C99 and Rab5

Cholesterol as a co‐solvent and a ligand for membrane proteins

Economic Costs of Civil Conflicts: The Case of Burundi

The transmembrane amyloid precursor C99 protein exhibits non-specific interaction with tau

Taking risks by flying paper airplanes

The vexing complexity of the amyloidogenic pathway

Towards a Unitary Hypothesis of Alzheimer's Disease Pathogenesis

The Transcription Factor EB Reduces the Intraneuronal Accumulation of the Beta-Secretase-Derived APP Fragment C99 in Cellular and Mouse AD Models

Do preferences for urban amenities differ by skill?

Perceptions of inevitability and demand for redistribution: Evidence from a survey experiment

MT5-MMP promotes neuroinflammation, neuronal excitability and Aβ production in primary neuron/astrocyte cultures from the 5xFAD mouse model of Alzheimer's disease

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