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Renal tubule injury: a driving force toward chronic kidney disease

Kidney international, 2018-03, Vol.93 (3), p.568-579

2018

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Cell profiling of mouse acute kidney injury reveals conserved cellular responses to injury

Proceedings of the National Academy of Sciences - PNAS, 2020-07, Vol.117 (27), p.15874-15883

2020

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Microvesicles derived from endothelial progenitor cells protect the kidney from ischemia–reperfusion injury by microRNA-dependent reprogramming of resident renal cells

Kidney international, 2012-08, Vol.82 (4), p.412-427

2012

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Cellular and molecular pathways of renal repair after acute kidney injury

Kidney international, 2018-01, Vol.93 (1), p.27-40

Kumar, Sanjeev

2018

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Mitochondrial Damage Causes Inflammation via cGAS-STING Signaling in Acute Kidney Injury

Cell reports (Cambridge), 2019-10, Vol.29 (5), p.1261-1273.e6

2019

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Epigenetic regulator BRD4 is involved in cadmium-induced acute kidney injury via contributing to lysosomal dysfunction, autophagy blockade and oxidative stress

Journal of hazardous materials, 2022-02, Vol.423 (Pt A), p.127110-127110, Article 127110

2022

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Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

Kidney international, 2012-07, Vol.82 (2), p.172-183

2012

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PINK1-parkin pathway of mitophagy protects against contrast-induced acute kidney injury via decreasing mitochondrial ROS and NLRP3 inflammasome activation

Redox biology, 2019-09, Vol.26, p.101254-101254, Article 101254

2019

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Fluoro‐Photoacoustic Polymeric Renal Reporter for Real‐Time Dual Imaging of Acute Kidney Injury

Advanced materials (Weinheim), 2020-04, Vol.32 (17), p.e1908530-n/a

2020

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Mitochondria ROS and mitophagy in acute kidney injury

Autophagy, 2023-02, Vol.19 (2), p.401-414

2023

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Cisplatin nephrotoxicity: a review of the literature

Journal of nephrology, 2018-02, Vol.31 (1), p.15-25

2018

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Mitochondrial dysfunction and the AKI-to-CKD transition

American journal of physiology. Renal physiology, 2020-12, Vol.319 (6), p.F1105-F1116

2020

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Inhibiting NLRP3 inflammasome attenuates apoptosis in contrast-induced acute kidney injury through the upregulation of HIF1A and BNIP3-mediated mitophagy

Autophagy, 2021-10, Vol.17 (10), p.2975-2990

2021

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Fasting reduces oxidative stress, mitochondrial dysfunction and fibrosis induced by renal ischemia-reperfusion injury

Free radical biology & medicine, 2019-05, Vol.135, p.60-67

2019

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NADPH oxidase 4 promotes cisplatin-induced acute kidney injury via ROS-mediated programmed cell death and inflammation

Laboratory investigation, 2018-01, Vol.98 (1), p.63-78

2018

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Recombinant α-Klotho may be prophylactic and therapeutic for acute to chronic kidney disease progression and uremic cardiomyopathy

Kidney international, 2017-05, Vol.91 (5), p.1104-1114

2017

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Cell Death in the Kidney

International journal of molecular sciences, 2019-07, Vol.20 (14), p.3598

2019

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The myeloid mineralocorticoid receptor controls inflammatory and fibrotic responses after renal injury via macrophage interleukin-4 receptor signaling

Kidney international, 2018-06, Vol.93 (6), p.1344-1355

2018

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Evaluation of cisplatin-induced injury in human kidney organoids

American journal of physiology. Renal physiology, 2020-04, Vol.318 (4), p.F971-F978

2020

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Sirtuins in Renal Health and Disease

Journal of the American Society of Nephrology, 2018-07, Vol.29 (7), p.1799-1809

2018

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Necroptosis and ferroptosis are alternative cell death pathways that operate in acute kidney failure

Cellular and molecular life sciences : CMLS, 2017-10, Vol.74 (19), p.3631-3645

2017

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The pattern recognition receptor, Mincle, is essential for maintaining the M1 macrophage phenotype in acute renal inflammation

Kidney international, 2017-03, Vol.91 (3), p.587-602

2017

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IL-4/IL-13–mediated polarization of renal macrophages/dendritic cells to an M2a phenotype is essential for recovery from acute kidney injury

Kidney international, 2017-02, Vol.91 (2), p.375-386

2017

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Exosomal miRNA-19b-3p of tubular epithelial cells promotes M1 macrophage activation in kidney injury

Cell death and differentiation, 2020-01, Vol.27 (1), p.210-226

2020

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Renal tubule injury: a driving force toward chronic kidney disease

Cell profiling of mouse acute kidney injury reveals conserved cellular responses to injury

Microvesicles derived from endothelial progenitor cells protect the kidney from ischemia–reperfusion injury by microRNA-dependent reprogramming of resident renal cells

Cellular and molecular pathways of renal repair after acute kidney injury

Mitochondrial Damage Causes Inflammation via cGAS-STING Signaling in Acute Kidney Injury

Epigenetic regulator BRD4 is involved in cadmium-induced acute kidney injury via contributing to lysosomal dysfunction, autophagy blockade and oxidative stress

Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

PINK1-parkin pathway of mitophagy protects against contrast-induced acute kidney injury via decreasing mitochondrial ROS and NLRP3 inflammasome activation

Fluoro‐Photoacoustic Polymeric Renal Reporter for Real‐Time Dual Imaging of Acute Kidney Injury

Mitochondria ROS and mitophagy in acute kidney injury

PEBP1 Wardens Ferroptosis by Enabling Lipoxygenase Generation of Lipid Death Signals

Cisplatin nephrotoxicity: a review of the literature

Mitochondrial dysfunction and the AKI-to-CKD transition

Inhibiting NLRP3 inflammasome attenuates apoptosis in contrast-induced acute kidney injury through the upregulation of HIF1A and BNIP3-mediated mitophagy

Fasting reduces oxidative stress, mitochondrial dysfunction and fibrosis induced by renal ischemia-reperfusion injury

NADPH oxidase 4 promotes cisplatin-induced acute kidney injury via ROS-mediated programmed cell death and inflammation

Recombinant α-Klotho may be prophylactic and therapeutic for acute to chronic kidney disease progression and uremic cardiomyopathy

Cell Death in the Kidney

The myeloid mineralocorticoid receptor controls inflammatory and fibrotic responses after renal injury via macrophage interleukin-4 receptor signaling

Evaluation of cisplatin-induced injury in human kidney organoids

Sirtuins in Renal Health and Disease

Necroptosis and ferroptosis are alternative cell death pathways that operate in acute kidney failure

The pattern recognition receptor, Mincle, is essential for maintaining the M1 macrophage phenotype in acute renal inflammation

IL-4/IL-13–mediated polarization of renal macrophages/dendritic cells to an M2a phenotype is essential for recovery from acute kidney injury

Exosomal miRNA-19b-3p of tubular epithelial cells promotes M1 macrophage activation in kidney injury

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