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A unified hypothesis for the genesis of cerebral malaria: sequestration, inflammation and hemostasis leading to microcirculatory dysfunction

Trends in parasitology, 2006-11, Vol.22 (11), p.503-508

2006

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Citrulline protects mice from experimental cerebral malaria by ameliorating hypoargininemia, urea cycle changes and vascular leak

PloS one, 2019-03, Vol.14 (3), p.e0213428-e0213428

2019

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Low nitric oxide bioavailability contributes to the genesis of experimental cerebral malaria

Nature medicine, 2006-12, Vol.12 (12), p.1417-1422

2006

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γδ T Cells but Not NK Cells Are Essential for Cell-Mediated Immunity against Plasmodium chabaudi Malaria

Infection and Immunity, 2010-10, Vol.78 (10), p.4331-4340

2010

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Platelets activate a pathogenic response to blood-stage Plasmodium infection but not a protective immune response

Blood, 2017-03, Vol.129 (12), p.1669-1679

2017

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Inhibition of Platelet Adherence to Brain Microvasculature Protects against Severe Plasmodium berghei Malaria

Infection and Immunity, 2003-11, Vol.71 (11), p.6553-6561

2003

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CD8+-T-Cell Depletion Ameliorates Circulatory Shock in Plasmodium berghei-Infected Mice

Infection and Immunity, 2001-12, Vol.69 (12), p.7341-7348

2001

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Splenic γδ T Cells Regulated by CD4+ T Cells Are Required To Control Chronic Plasmodium chabaudi Malaria in the B-Cell-Deficient Mouse

Infection and Immunity, 2006-05, Vol.74 (5), p.2717-2725

2006

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Regulation of Endothelial Cell Adhesion Molecule Expression in an Experimental Model of Cerebral Malaria

Microcirculation (New York, N.Y. 1994), 2002-12, Vol.9 (6), p.463-470

2002

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Assessing Vascular Permeability during Experimental Cerebral Malaria by a Radiolabeled Monoclonal Antibody Technique

Infection and Immunity, 2001-05, Vol.69 (5), p.3460-3465

2001

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γδ T cell-induced nitric oxide production enhances resistance to mucosal candidiasis

Nature medicine, 1995-06, Vol.1 (6), p.552-557

1995

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Platelet depletion by anti-CD41 (αIIb) mAb injection early but not late in the course of disease protects against Plasmodium berghei pathogenesis by altering the levels of pathogenic cytokines

Blood, 2005-03, Vol.105 (5), p.1956-1963

2005

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Flow cytometric analysis of microparticles

Methods in molecular biology (Clifton, N.J.), 2011-01, Vol.699, p.337

2011

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P-Selectin Contributes to Severe Experimental Malaria but Is Not Required for Leukocyte Adhesion to Brain Microvasculature

Infection and Immunity, 2003-04, Vol.71 (4), p.1911-1918

2003

Volltextzugriff (PDF)

Nitric oxide bioavailability in malaria

Trends in parasitology, 2005-09, Vol.21 (9), p.415-422

2005

Volltextzugriff (PDF)

Impairment of Functional Capillary Density but Not Oxygen Delivery in the Hamster Window Chamber during Severe Experimental Malaria

The American journal of pathology, 2007-02, Vol.170 (2), p.505-517

2007

Volltextzugriff (PDF)

Cell- Rather Than Antibody-Mediated Immunity Leads to the Development of Profound Thrombocytopenia during Experimental Plasmodium berghei Malaria

Journal of Immunology, 2005-12, Vol.175 (11), p.7699-7707

2005

Volltextzugriff (PDF)

Continuous Inhaled Nitric Oxide Therapy in a Case of Sickle Cell Disease With Multiorgan Involvement

Journal of investigative medicine, 2008-12, Vol.56 (8), p.1023-1027

2008

Volltextzugriff (PDF)

Bead-based multiplexed analysis of analytes by flow cytometry

Methods in molecular biology (Clifton, N.J.), 2011-01, Vol.699, p.85

2011

Volltextzugriff (PDF)

Analysis of antigen‐specific antibodies and their isotypes in experimental malaria

Cytometry. Part A, 2007-04, Vol.71A (4), p.242-250

2007

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γδ T cells function in cell-mediated immunity to malaria

Nature reviews. Immunology, 2004-08, Vol.4 (8), p.656-656

2004

Volltextzugriff (PDF)

Nitric Oxide Is Neither Necessary Nor Sufficient for Resolution of Plasmodium chabaudi Malaria in Mice

The Journal of immunology (1950), 2000-09, Vol.165 (6), p.3317-3323

2000

Volltextzugriff (PDF)

The Time Course of Selected Malarial Infections in Cytokine-Deficient Mice

Experimental parasitology, 1997-02, Vol.85 (2), p.206-213

1997

Volltextzugriff (PDF)

Hemoglobin Serves to Protect Plasmodium Parasites from Nitric Oxide and Reactive Oxygen Species

Journal of Investigative Medicine, 2005-07, Vol.53 (5), p.246-252

2005

Volltextzugriff (PDF)

Intercellular Adhesion Molecule 1 Is Important for the Development of Severe Experimental Malaria but Is Not Required for Leukocyte Adhesion in the Brain

Journal of investigative medicine, 2003-05, Vol.51 (3), p.128-140

2003

Volltextzugriff (PDF)

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A unified hypothesis for the genesis of cerebral malaria: sequestration, inflammation and hemostasis leading to microcirculatory dysfunction

Citrulline protects mice from experimental cerebral malaria by ameliorating hypoargininemia, urea cycle changes and vascular leak

Low nitric oxide bioavailability contributes to the genesis of experimental cerebral malaria

γδ T Cells but Not NK Cells Are Essential for Cell-Mediated Immunity against Plasmodium chabaudi Malaria

Platelets activate a pathogenic response to blood-stage Plasmodium infection but not a protective immune response

Inhibition of Platelet Adherence to Brain Microvasculature Protects against Severe Plasmodium berghei Malaria

CD8+-T-Cell Depletion Ameliorates Circulatory Shock in Plasmodium berghei-Infected Mice

Splenic γδ T Cells Regulated by CD4+ T Cells Are Required To Control Chronic Plasmodium chabaudi Malaria in the B-Cell-Deficient Mouse

Regulation of Endothelial Cell Adhesion Molecule Expression in an Experimental Model of Cerebral Malaria

Assessing Vascular Permeability during Experimental Cerebral Malaria by a Radiolabeled Monoclonal Antibody Technique

γδ T cell-induced nitric oxide production enhances resistance to mucosal candidiasis

Platelet depletion by anti-CD41 (αIIb) mAb injection early but not late in the course of disease protects against Plasmodium berghei pathogenesis by altering the levels of pathogenic cytokines

Flow cytometric analysis of microparticles

P-Selectin Contributes to Severe Experimental Malaria but Is Not Required for Leukocyte Adhesion to Brain Microvasculature

Nitric oxide bioavailability in malaria

Impairment of Functional Capillary Density but Not Oxygen Delivery in the Hamster Window Chamber during Severe Experimental Malaria

Cell- Rather Than Antibody-Mediated Immunity Leads to the Development of Profound Thrombocytopenia during Experimental Plasmodium berghei Malaria

Continuous Inhaled Nitric Oxide Therapy in a Case of Sickle Cell Disease With Multiorgan Involvement

Bead-based multiplexed analysis of analytes by flow cytometry

Analysis of antigen‐specific antibodies and their isotypes in experimental malaria

γδ T cells function in cell-mediated immunity to malaria

Nitric Oxide Is Neither Necessary Nor Sufficient for Resolution of Plasmodium chabaudi Malaria in Mice

The Time Course of Selected Malarial Infections in Cytokine-Deficient Mice

Hemoglobin Serves to Protect Plasmodium Parasites from Nitric Oxide and Reactive Oxygen Species

Intercellular Adhesion Molecule 1 Is Important for the Development of Severe Experimental Malaria but Is Not Required for Leukocyte Adhesion in the Brain

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