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Reducing protein oxidation reverses lung fibrosis

Nature medicine, 2018-08, Vol.24 (8), p.1128-1135

2018

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Muscle Wasting and Impaired Muscle Regeneration in a Murine Model of Chronic Pulmonary Inflammation

American journal of respiratory cell and molecular biology, 2006-12, Vol.35 (6), p.689-696

2006

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Futures for invasive alien species management: using bottom-up innovations to envision positive systemic change

Sustainability science, 2023-11, Vol.18 (6), p.2567-2587

2023

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JNK inhibition reduces lung remodeling and pulmonary fibrotic systemic markers

Clinical and translational medicine, 2016-09, Vol.5 (1), p.1-18

2016

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IL-1/inhibitory κB kinase ε–induced glycolysis augment epithelial effector function and promote allergic airways disease

Journal of allergy and clinical immunology, 2018-08, Vol.142 (2), p.435-450.e10

2018

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Non-adherence to antimicrobial treatment guidelines results in more broad-spectrum but not more appropriate therapy

European journal of clinical microbiology & infectious diseases, 2012-07, Vol.31 (7), p.1561-1568

2012

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c-Jun N-terminal kinase 1 promotes transforming growth factor-β1-induced epithelial-to-mesenchymal transition via control of linker phosphorylation and transcriptional activity of Smad3

American journal of respiratory cell and molecular biology, 2011-04, Vol.44 (4), p.571-581

2011

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Segregation of myoblast fusion and muscle-specific gene expression by distinct ligand-dependent inactivation of GSK-3β

Cellular and molecular life sciences : CMLS, 2011-02, Vol.68 (3), p.523-535

2011

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Genetic ablation of glutaredoxin-1 causes enhanced resolution of airways hyperresponsiveness and mucus metaplasia in mice with allergic airways disease

American journal of physiology. Lung cellular and molecular physiology, 2012-09, Vol.303 (6), p.L528-L538

2012

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Age-dependent dysregulation of redox genes may contribute to fibrotic pulmonary disease susceptibility

Free radical biology & medicine, 2019-09, Vol.141, p.438-446

2019

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Tumor necrosis factor‐alpha inhibits myogenic differentiation through MyoD protein destabilization

The FASEB journal, 2004-02, Vol.18 (2), p.227-237

2004

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The Duffy antigen receptor for chemokines regulates asthma pathophysiology

Clinical and experimental allergy, 2017-09, Vol.47 (9), p.1214-1222

2017

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Glutathione S-transferase pi modulates NF-κB activation and pro-inflammatory responses in lung epithelial cells

Redox biology, 2016-08, Vol.8 (C), p.375-382

2016

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Glycogen synthase kinase-3β is required for the induction of skeletal muscle atrophy

American Journal of Physiology: Cell Physiology, 2011-11, Vol.301 (5), p.C995-C1007

2011

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Interruption of Wnt Signaling Attenuates the Onset of Pressure Overload-Induced Cardiac Hypertrophy

Hypertension (Dallas, Tex. 1979), 2007-03, Vol.49 (3), p.473-480

2007

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Induction of a mesenchymal expression program in lung epithelial cells by wingless protein (Wnt)/β-catenin requires the presence of c-Jun N-terminal kinase-1 (JNK1)

American journal of respiratory cell and molecular biology, 2012-09, Vol.47 (3), p.306-314

2012

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Absence of c-Jun NH2-terminal kinase 1 protects against house dust mite-induced pulmonary remodeling but not airway hyperresponsiveness and inflammation

American journal of physiology. Lung cellular and molecular physiology, 2014-05, Vol.306 (9), p.L866-L875

2014

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Glycogen Synthase Kinase 3β Suppresses Myogenic Differentiation through Negative Regulation of NFATc3

The Journal of biological chemistry, 2008-01, Vol.283 (1), p.358-366

2008

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C31 BASIC AND TRANSLATIONAL STUDIES ON THE ROLE OF THE EPITHELIUM: Absence Of Epithelial C-Jun N-Terminal Kinase 1 Protects Against House Dust Mite- Induced Pulmonary Remodeling And Airway Hyper Responsiveness

American journal of respiratory and critical care medicine, 2017-01, Vol.195

2017

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C38 UNDERSTANDING THERAPEUTICS IN IPF: Cc-90001, A Second Generation Jun N-Terminal Kinase (JNK) Inhibitor For The Treatment Of Idiopathic Pulmonary Fibrosis

American journal of respiratory and critical care medicine, 2017-01, Vol.195

2017

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C107 MITOCHONDRIA AND METABOLIC ALTERATIONS IN LUNG DISEASE: Gstp1-Catalyzed Pkm2 S-Glutathionylation Regulates Glycolysis In Non-Small Cell Lung Cancer And Is Attenuated With A Clinically Relevant Inhibitor Of Glutathione-S-Transferase P

American journal of respiratory and critical care medicine, 2017-01, Vol.195

2017

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Acute myocardial infarction complicating subarachnoid haemorrhage

Netherlands heart journal, 2009-08, Vol.17 (7), p.284-287

2009

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C22 AIRWAY EPITHELIUM: MOLECULAR REGULATION OF INFLAMMATION: Increased Nuclear Factor-kgr;b Activation In Airway Epithelial Cells In A Mouse Model Of Allergic Airways Disease Promotes Pathophysiology Consistent With Asthma And COPD

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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A rare combination of coronary anomalies

Netherlands heart journal, 2008-11, Vol.16 (11), p.387-389

2008

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A110 THORACIC CANCERS: PATHOGENESIS AND NOVEL TARGETS: Gstp1-Catalyzed S-Glutathionylation In Krasg12d-Induced Lung Tumorigenesis

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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Reducing protein oxidation reverses lung fibrosis

Muscle Wasting and Impaired Muscle Regeneration in a Murine Model of Chronic Pulmonary Inflammation

Futures for invasive alien species management: using bottom-up innovations to envision positive systemic change

JNK inhibition reduces lung remodeling and pulmonary fibrotic systemic markers

IL-1/inhibitory κB kinase ε–induced glycolysis augment epithelial effector function and promote allergic airways disease

Non-adherence to antimicrobial treatment guidelines results in more broad-spectrum but not more appropriate therapy

c-Jun N-terminal kinase 1 promotes transforming growth factor-β1-induced epithelial-to-mesenchymal transition via control of linker phosphorylation and transcriptional activity of Smad3

Segregation of myoblast fusion and muscle-specific gene expression by distinct ligand-dependent inactivation of GSK-3β

Genetic ablation of glutaredoxin-1 causes enhanced resolution of airways hyperresponsiveness and mucus metaplasia in mice with allergic airways disease

Age-dependent dysregulation of redox genes may contribute to fibrotic pulmonary disease susceptibility

Tumor necrosis factor‐alpha inhibits myogenic differentiation through MyoD protein destabilization

The Duffy antigen receptor for chemokines regulates asthma pathophysiology

Glutathione S-transferase pi modulates NF-κB activation and pro-inflammatory responses in lung epithelial cells

Glycogen synthase kinase-3β is required for the induction of skeletal muscle atrophy

Interruption of Wnt Signaling Attenuates the Onset of Pressure Overload-Induced Cardiac Hypertrophy

Induction of a mesenchymal expression program in lung epithelial cells by wingless protein (Wnt)/β-catenin requires the presence of c-Jun N-terminal kinase-1 (JNK1)

Absence of c-Jun NH2-terminal kinase 1 protects against house dust mite-induced pulmonary remodeling but not airway hyperresponsiveness and inflammation

Glycogen Synthase Kinase 3β Suppresses Myogenic Differentiation through Negative Regulation of NFATc3

C31 BASIC AND TRANSLATIONAL STUDIES ON THE ROLE OF THE EPITHELIUM: Absence Of Epithelial C-Jun N-Terminal Kinase 1 Protects Against House Dust Mite- Induced Pulmonary Remodeling And Airway Hyper Responsiveness

C38 UNDERSTANDING THERAPEUTICS IN IPF: Cc-90001, A Second Generation Jun N-Terminal Kinase (JNK) Inhibitor For The Treatment Of Idiopathic Pulmonary Fibrosis

C107 MITOCHONDRIA AND METABOLIC ALTERATIONS IN LUNG DISEASE: Gstp1-Catalyzed Pkm2 S-Glutathionylation Regulates Glycolysis In Non-Small Cell Lung Cancer And Is Attenuated With A Clinically Relevant Inhibitor Of Glutathione-S-Transferase P

Acute myocardial infarction complicating subarachnoid haemorrhage

C22 AIRWAY EPITHELIUM: MOLECULAR REGULATION OF INFLAMMATION: Increased Nuclear Factor-kgr;b Activation In Airway Epithelial Cells In A Mouse Model Of Allergic Airways Disease Promotes Pathophysiology Consistent With Asthma And COPD

A rare combination of coronary anomalies

A110 THORACIC CANCERS: PATHOGENESIS AND NOVEL TARGETS: Gstp1-Catalyzed S-Glutathionylation In Krasg12d-Induced Lung Tumorigenesis

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