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Refining the molecular organization of the cardiac intercalated disc

Cardiovascular research, 2017-03, Vol.113 (3), p.259-275

2017

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The immature electrophysiological phenotype of iPSC-CMs still hampers in vitro drug screening: Special focus on I K1

Pharmacology & therapeutics (Oxford), 2018-03, Vol.183, p.127-136

2018

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Sex-specific influence on cardiac structural remodeling and therapy in cardiovascular disease

Biology of sex differences, 2019-02, Vol.10 (1), p.7-11, Article 7

2019

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Uremic toxins in chronic kidney disease highlight a fundamental gap in understanding their detrimental effects on cardiac electrophysiology and arrhythmogenesis

Acta Physiologica, 2022-11, Vol.236 (3), p.e13888-n/a

2022

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Assembly of the cardiac intercalated disk during pre- and postnatal development of the human heart

PloS one, 2014-04, Vol.9 (4), p.e94722-e94722

2014

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Plakophilin-2 is required for transcription of genes that control calcium cycling and cardiac rhythm

Nature communications, 2017-07, Vol.8 (1), p.106-16, Article 106

2017

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The intercalated disc: a unique organelle for electromechanical synchrony in cardiomyocytes

Physiological reviews, 2023-07, Vol.103 (3), p.2271-2319

2023

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Lack of Evidence for the Role of the p.(Ser96Ala) Polymorphism in Histidine-Rich Calcium Binding Protein as a Secondary Hit in Cardiomyopathies

International journal of molecular sciences, 2023-11, Vol.24 (21), p.15931

2023

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Triplex protein quantification based on stable isotope labeling by peptide dimethylation applied to cell and tissue lysates

Proteomics (Weinheim), 2008-11, Vol.8 (22), p.4624-4632

2008

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Pro-Arrhythmic Potential of Accumulated Uremic Toxins Is Mediated via Vulnerability of Action Potential Repolarization

International journal of molecular sciences, 2023-03, Vol.24 (6), p.5373

2023

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Application of human stem cell-derived cardiomyocytes in safety pharmacology requires caution beyond hERG

Journal of molecular and cellular cardiology, 2012-05, Vol.52 (5), p.998-1008

2012

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Mitochondrial Dysfunction as Substrate for Arrhythmogenic Cardiomyopathy: A Search for New Disease Mechanisms

Frontiers in physiology, 2019-12, Vol.10, p.1496-1496

2019

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Desmosomal protein degradation as an underlying cause of arrhythmogenic cardiomyopathy

Science translational medicine, 2023-03, Vol.15 (688), p.eadd4248

2023

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Gene expression profiling of hypertrophic cardiomyocytes identifies new players in pathological remodelling

Cardiovascular research, 2021-05, Vol.117 (6), p.1532-1545

2021

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A Hybrid Model for Safety Pharmacology on an Automated Patch Clamp Platform: Using Dynamic Clamp to Join iPSC-Derived Cardiomyocytes and Simulations of Ik1 Ion Channels in Real-Time

Frontiers in physiology, 2018-01, Vol.8, p.1094-1094

2018

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Arrhythmogenic cardiomyopathy: pathogenesis, pro-arrhythmic remodelling, and novel approaches for risk stratification and therapy

Cardiovascular research, 2020-07, Vol.116 (9), p.1571-1584

2020

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Defective Cx40 Maintains Cx37 Expression but Intact Cx40 Is Crucial for Conducted Dilations Irrespective of Hypertension

Hypertension (Dallas, Tex. 1979), 2012-12, Vol.60 (6), p.1422-1429

2012

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Plakophilin-2 Haploinsufficiency Causes Calcium Handling Deficits and Modulates the Cardiac Response Towards Stress

International journal of molecular sciences, 2019-08, Vol.20 (17), p.4076

2019

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Fibrosis and Cardiac Arrhythmias

Journal of cardiovascular pharmacology, 2011-06, Vol.57 (6), p.630-638

2011

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Cardiac connexins and impulse propagation

Journal of molecular and cellular cardiology, 2010-01, Vol.48 (1), p.76-82

2010

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Buccal Mucosa Cells as a Potential Diagnostic Tool to Study Onset and Progression of Arrhythmogenic Cardiomyopathy

International journal of molecular sciences, 2021-12, Vol.23 (1), p.57

2021

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Lessons from the heart: Mirroring electrophysiological characteristics during cardiac development to in vitro differentiation of stem cell derived cardiomyocytes

Journal of molecular and cellular cardiology, 2014-02, Vol.67, p.12-25

2014

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Reduced Cx43 Expression Triggers Increased Fibrosis Due to Enhanced Fibroblast Activity

Circulation. Arrhythmia and electrophysiology, 2012-04, Vol.5 (2), p.380-390

2012

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Refining the molecular organization of the cardiac intercalated disc

The immature electrophysiological phenotype of iPSC-CMs still hampers in vitro drug screening: Special focus on I K1

Sex-specific influence on cardiac structural remodeling and therapy in cardiovascular disease

Uremic toxins in chronic kidney disease highlight a fundamental gap in understanding their detrimental effects on cardiac electrophysiology and arrhythmogenesis

Assembly of the cardiac intercalated disk during pre- and postnatal development of the human heart

Plakophilin-2 is required for transcription of genes that control calcium cycling and cardiac rhythm

The intercalated disc: a unique organelle for electromechanical synchrony in cardiomyocytes

Lack of Evidence for the Role of the p.(Ser96Ala) Polymorphism in Histidine-Rich Calcium Binding Protein as a Secondary Hit in Cardiomyopathies

Triplex protein quantification based on stable isotope labeling by peptide dimethylation applied to cell and tissue lysates

Pro-Arrhythmic Potential of Accumulated Uremic Toxins Is Mediated via Vulnerability of Action Potential Repolarization

Application of human stem cell-derived cardiomyocytes in safety pharmacology requires caution beyond hERG

Mitochondrial Dysfunction as Substrate for Arrhythmogenic Cardiomyopathy: A Search for New Disease Mechanisms

Desmosomal protein degradation as an underlying cause of arrhythmogenic cardiomyopathy

Gene expression profiling of hypertrophic cardiomyocytes identifies new players in pathological remodelling

A Hybrid Model for Safety Pharmacology on an Automated Patch Clamp Platform: Using Dynamic Clamp to Join iPSC-Derived Cardiomyocytes and Simulations of Ik1 Ion Channels in Real-Time

Arrhythmogenic cardiomyopathy: pathogenesis, pro-arrhythmic remodelling, and novel approaches for risk stratification and therapy

Defective Cx40 Maintains Cx37 Expression but Intact Cx40 Is Crucial for Conducted Dilations Irrespective of Hypertension

Plakophilin-2 Haploinsufficiency Causes Calcium Handling Deficits and Modulates the Cardiac Response Towards Stress

Multilevel analyses of SCN5A mutations in arrhythmogenic right ventricular dysplasia/cardiomyopathy suggest non-canonical mechanisms for disease pathogenesis

Fibrosis and Cardiac Arrhythmias

Cardiac connexins and impulse propagation

Buccal Mucosa Cells as a Potential Diagnostic Tool to Study Onset and Progression of Arrhythmogenic Cardiomyopathy

Impact of genotype on clinical course in arrhythmogenic right ventricular dysplasia/cardiomyopathy-associated mutation carriers

Lessons from the heart: Mirroring electrophysiological characteristics during cardiac development to in vitro differentiation of stem cell derived cardiomyocytes

Reduced Cx43 Expression Triggers Increased Fibrosis Due to Enhanced Fibroblast Activity

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