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Deletion of the mitochondrial superoxide dismutase sod-2 extends lifespan in Caenorhabditis elegans

PLoS genetics, 2009-02, Vol.5 (2), p.e1000361

2009

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Beneficial and Detrimental Effects of Reactive Oxygen Species on Lifespan: A Comprehensive Review of Comparative and Experimental Studies

Frontiers in cell and developmental biology, 2021-02, Vol.9, p.628157-628157

2021

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Mitochondrial and cytoplasmic ROS have opposing effects on lifespan

PLoS genetics, 2015-02, Vol.11 (2), p.e1004972-e1004972

2015

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Overexpression of mitochondrial fission or mitochondrial fusion genes enhances resilience and extends longevity

Aging cell, 2024-07, p.e14262

2024

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The Rab GTPase activating protein TBC-2 regulates endosomal localization of DAF-16 FOXO and lifespan

PLoS genetics, 2022-08, Vol.18 (8), p.e1010328-e1010328

2022

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Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans

PLoS genetics, 2018-03, Vol.14 (3), p.e1007268-e1007268

2018

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RNA Sequencing of Pooled Samples Effectively Identifies Differentially Expressed Genes

Biology (Basel, Switzerland), 2023-06, Vol.12 (6), p.812

2023

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Mitochondrial pyruvate carrier regulates autophagy, inflammation, and neurodegeneration in experimental models of Parkinson's disease

Science translational medicine, 2016-12, Vol.8 (368), p.368ra174

2016

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Mitochondrial unfolded protein response transcription factor ATFS-1 promotes longevity in a long-lived mitochondrial mutant through activation of stress response pathways

BMC biology, 2018-12, Vol.16 (1), p.147-147, Article 147

2018

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Evolutionarily Conserved Role of Thioredoxin Systems in Determining Longevity

Antioxidants, 2023-04, Vol.12 (4), p.944

2023

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Measuring Oxidative Stress in Caenorhabditis elegans : Paraquat and Juglone Sensitivity Assays

Bio-protocol, 2017-01, Vol.7 (1)

2017

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Multiple genetic pathways regulating lifespan extension are neuroprotective in a G2019S LRRK2 nematode model of Parkinson's disease

Neurobiology of disease, 2021-04, Vol.151, p.105267-105267, Article 105267

2021

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Oxidative stress is increased in C. elegans models of Huntington’s disease but does not contribute to polyglutamine toxicity phenotypes

Neurobiology of disease, 2016-12, Vol.96, p.1-11

2016

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Novel animal model defines genetic contributions for neuron-to-neuron transfer of α-synuclein

Scientific reports, 2017-08, Vol.7 (1), p.7506-10, Article 7506

2017

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Aging causes decreased resistance to multiple stresses and a failure to activate specific stress response pathways

Aging (Albany, NY.), 2016-04, Vol.8 (4), p.777-795

2016

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Targeting Mitochondrial Network Disorganization is Protective in C. elegans Models of Huntington’s Disease

Aging and disease, 2021-10, Vol.12 (7), p.1753-1772

2021

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Identification of Novel Therapeutic Targets for Polyglutamine Diseases That Target Mitochondrial Fragmentation

International journal of molecular sciences, 2021-12, Vol.22 (24), p.13447

2021

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Mitochondrial thioredoxin system is required for enhanced stress resistance and extended longevity in long-lived mitochondrial mutants

Redox biology, 2022-07, Vol.53, p.102335-102335, Article 102335

2022

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Cognitive Dysfunction Precedes Neuropathology and Motor Abnormalities in the YAC128 Mouse Model of Huntington's Disease

The Journal of neuroscience, 2005-04, Vol.25 (16), p.4169-4180

2005

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Modeling Parkinson's Disease in C. elegans

Journal of Parkinson's disease, 2018-01, Vol.8 (1), p.17-32

2018

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The glyoxylate shunt protein ICL-1 protects from mitochondrial superoxide stress through activation of the mitochondrial unfolded protein response

Free radical biology & medicine, 2023-11, Vol.208, p.771-779

2023

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Mechanisms underlying longevity: A genetic switch model of aging

Experimental gerontology, 2018-07, Vol.107, p.136-139

Van Raamsdonk, Jeremy M.

2018

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Superoxide dismutase is dispensable for normal animal lifespan

Proceedings of the National Academy of Sciences - PNAS, 2012-04, Vol.109 (15), p.5785-5790

2012

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Delaying aging is neuroprotective in Parkinson's disease: a genetic analysis in C. elegans models

NPJ Parkinson's Disease, 2015-01, Vol.1 (1), p.15022-15022, Article 15022

2015

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Endosomal trafficking protein TBC-2 is required for the longevity of long-lived mitochondrial mutants

Frontiers in aging, 2023-05, Vol.4, p.1145198-1145198

2023

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Deletion of the mitochondrial superoxide dismutase sod-2 extends lifespan in Caenorhabditis elegans

Beneficial and Detrimental Effects of Reactive Oxygen Species on Lifespan: A Comprehensive Review of Comparative and Experimental Studies

Mitochondrial and cytoplasmic ROS have opposing effects on lifespan

Overexpression of mitochondrial fission or mitochondrial fusion genes enhances resilience and extends longevity

The Rab GTPase activating protein TBC-2 regulates endosomal localization of DAF-16 FOXO and lifespan

Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans

RNA Sequencing of Pooled Samples Effectively Identifies Differentially Expressed Genes

Mitochondrial pyruvate carrier regulates autophagy, inflammation, and neurodegeneration in experimental models of Parkinson's disease

Mitochondrial unfolded protein response transcription factor ATFS-1 promotes longevity in a long-lived mitochondrial mutant through activation of stress response pathways

Evolutionarily Conserved Role of Thioredoxin Systems in Determining Longevity

Measuring Oxidative Stress in Caenorhabditis elegans : Paraquat and Juglone Sensitivity Assays

Multiple genetic pathways regulating lifespan extension are neuroprotective in a G2019S LRRK2 nematode model of Parkinson's disease

Oxidative stress is increased in C. elegans models of Huntington’s disease but does not contribute to polyglutamine toxicity phenotypes

Novel animal model defines genetic contributions for neuron-to-neuron transfer of α-synuclein

Aging causes decreased resistance to multiple stresses and a failure to activate specific stress response pathways

Targeting Mitochondrial Network Disorganization is Protective in C. elegans Models of Huntington’s Disease

Identification of Novel Therapeutic Targets for Polyglutamine Diseases That Target Mitochondrial Fragmentation

Mitochondrial thioredoxin system is required for enhanced stress resistance and extended longevity in long-lived mitochondrial mutants

Cognitive Dysfunction Precedes Neuropathology and Motor Abnormalities in the YAC128 Mouse Model of Huntington's Disease

Modeling Parkinson's Disease in C. elegans

The glyoxylate shunt protein ICL-1 protects from mitochondrial superoxide stress through activation of the mitochondrial unfolded protein response

Mechanisms underlying longevity: A genetic switch model of aging

Superoxide dismutase is dispensable for normal animal lifespan

Delaying aging is neuroprotective in Parkinson's disease: a genetic analysis in C. elegans models

Endosomal trafficking protein TBC-2 is required for the longevity of long-lived mitochondrial mutants

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