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Myocarditis and inflammatory cardiomyopathy: current evidence and future directions

Nature reviews cardiology, 2021-03, Vol.18 (3), p.169-193

2021

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Inflammation – Cause or Consequence of Heart Failure or Both?

Current heart failure reports, 2017-08, Vol.14 (4), p.251-265

2017

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Cpxm2 as a novel candidate for cardiac hypertrophy and failure in hypertension

Hypertension research, 2022-02, Vol.45 (2), p.292-307

2022

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Myeloid-Derived Suppressor Cells Restrain Natural Killer Cell Activity in Acute Coxsackievirus B3-Induced Myocarditis

Viruses, 2021-05, Vol.13 (5), p.889

2021

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Management of Myocarditis-Related Cardiomyopathy in Adults

Circulation research, 2019-05, Vol.124 (11), p.1568-1583

2019

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Application Route and Immune Status of the Host Determine Safety and Oncolytic Activity of Oncolytic Coxsackievirus B3 Variant PD-H

Viruses, 2021-09, Vol.13 (10), p.1918

2021

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Coronary microvascular dysfunction in hypertrophy and heart failure

Cardiovascular research, 2020-03, Vol.116 (4), p.806-816

2020

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Spontaneous Degenerative Aortic Valve Disease in New Zealand Obese Mice

Journal of the American Heart Association, 2021-12, Vol.10 (23), p.e023131

2021

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SARS‐CoV2‐related myocarditis‐like syndroms: Shakespeare´s question: What´s in a name?

European journal of heart failure, 2020-05

2020

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Immuno-cardio-oncology: Killing two birds with one stone?

Frontiers in immunology, 2022-11, Vol.13, p.1018772-1018772

2022

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Mechanical Unloading by Fulminant Myocarditis: LV-IMPELLA, ECMELLA, BI-PELLA, and PROPELLA Concepts

Journal of cardiovascular translational research, 2019-04, Vol.12 (2), p.116-123

2019

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Cardiac (myo)fibroblasts modulate the migration of monocyte subsets

Scientific reports, 2018-04, Vol.8 (1), p.5575-11, Article 5575

2018

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Left ventricle- and skeletal muscle-derived fibroblasts exhibit a differential inflammatory and metabolic responsiveness to interleukin-6

Frontiers in immunology, 2022-07, Vol.13, p.947267-947267

2022

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COVID-19-related cardiac complications from clinical evidences to basic mechanisms: opinion paper of the ESC Working Group on Cellular Biology of the Heart

Cardiovascular research, 2021-09, Vol.117 (10), p.2148-2160

2021

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Mode-of-action of the PROPELLA concept in fulminant myocarditis

European heart journal, 2019-07, Vol.40 (26), p.2164-2169

2019

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Elimination of innate immune responses and liver inflammation by PEGylation of adenoviral vectors and methylprednisolone

Human gene therapy, 2005-12, Vol.16 (12), p.1439-1451

2005

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Opioid-Induced Immunomodulation: Consequences for the Experimental Coxsackievirus B3-Induced Myocarditis Model

Biology (Basel, Switzerland), 2020-10, Vol.9 (10), p.335

2020

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Mesenchymal Stromal Cells Modulate Monocytes Trafficking in Coxsackievirus B3‐Induced Myocarditis

Stem cells translational medicine, 2017-04, Vol.6 (4), p.1249-1261

2017

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Mesenchymal stromal cells inhibit NLRP3 inflammasome activation in a model of Coxsackievirus B3-induced inflammatory cardiomyopathy

Scientific reports, 2018-02, Vol.8 (1), p.2820-16, Article 2820

2018

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Epicardial Fat Expansion in Diabetic and Obese Patients With Heart Failure and Preserved Ejection Fraction—A Specific HFpEF Phenotype

Frontiers in cardiovascular medicine, 2021-09, Vol.8, p.720690-720690

2021

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Nerve Growth Factor Promotes Cardiac Repair following Myocardial Infarction

Circulation research, 2010-04, Vol.106 (7), p.1275-1284

2010

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Therapeutic potential of HDL in cardioprotection and tissue repair

Handbook of experimental pharmacology, 2015-01, Vol.224, p.527

2015

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Elevated Sera sST2 Is Associated With Heart Failure in Men ≤50 Years Old With Myocarditis

Journal of the American Heart Association, 2019-01, Vol.8 (2), p.e008968

2019

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Cardiac Extracellular Vesicles (EVs) Released in the Presence or Absence of Inflammatory Cues Support Angiogenesis in Different Manners

International journal of molecular sciences, 2019-12, Vol.20 (24), p.6363

2019

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Myocarditis and inflammatory cardiomyopathy: current evidence and future directions

Inflammation – Cause or Consequence of Heart Failure or Both?

Cpxm2 as a novel candidate for cardiac hypertrophy and failure in hypertension

Myeloid-Derived Suppressor Cells Restrain Natural Killer Cell Activity in Acute Coxsackievirus B3-Induced Myocarditis

Management of Myocarditis-Related Cardiomyopathy in Adults

Application Route and Immune Status of the Host Determine Safety and Oncolytic Activity of Oncolytic Coxsackievirus B3 Variant PD-H

Coronary microvascular dysfunction in hypertrophy and heart failure

Spontaneous Degenerative Aortic Valve Disease in New Zealand Obese Mice

Empagliflozin improves endothelial and cardiomyocyte function in human heart failure with preserved ejection fraction via reduced pro-inflammatory-oxidative pathways and protein kinase Gα oxidation

SARS‐CoV2‐related myocarditis‐like syndroms: Shakespeare´s question: What´s in a name?

Immuno-cardio-oncology: Killing two birds with one stone?

Mechanical Unloading by Fulminant Myocarditis: LV-IMPELLA, ECMELLA, BI-PELLA, and PROPELLA Concepts

Cardiac (myo)fibroblasts modulate the migration of monocyte subsets

Left ventricle- and skeletal muscle-derived fibroblasts exhibit a differential inflammatory and metabolic responsiveness to interleukin-6

COVID-19-related cardiac complications from clinical evidences to basic mechanisms: opinion paper of the ESC Working Group on Cellular Biology of the Heart

Mode-of-action of the PROPELLA concept in fulminant myocarditis

Elimination of innate immune responses and liver inflammation by PEGylation of adenoviral vectors and methylprednisolone

Opioid-Induced Immunomodulation: Consequences for the Experimental Coxsackievirus B3-Induced Myocarditis Model

Mesenchymal Stromal Cells Modulate Monocytes Trafficking in Coxsackievirus B3‐Induced Myocarditis

Mesenchymal stromal cells inhibit NLRP3 inflammasome activation in a model of Coxsackievirus B3-induced inflammatory cardiomyopathy

Epicardial Fat Expansion in Diabetic and Obese Patients With Heart Failure and Preserved Ejection Fraction—A Specific HFpEF Phenotype

Nerve Growth Factor Promotes Cardiac Repair following Myocardial Infarction

Therapeutic potential of HDL in cardioprotection and tissue repair

Elevated Sera sST2 Is Associated With Heart Failure in Men ≤50 Years Old With Myocarditis

Cardiac Extracellular Vesicles (EVs) Released in the Presence or Absence of Inflammatory Cues Support Angiogenesis in Different Manners

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