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AMP-Activated Protein Kinase Regulates GLUT4 Transcription by Phosphorylating Histone Deacetylase 5

Diabetes (New York, N.Y.), 2008-04, Vol.57 (4), p.860-867

2008

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Single phosphorylation sites in Acc1 and Acc2 regulate lipid homeostasis and the insulin-sensitizing effects of metformin

Nature medicine, 2013-12, Vol.19 (12), p.1649-1654

2013

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Hematopoietic AMPK β1 reduces mouse adipose tissue macrophage inflammation and insulin resistance in obesity

The Journal of clinical investigation, 2011-12, Vol.121 (12), p.4903-4915

2011

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The C-terminal protein interaction domain of the chromatin reader Yaf9 is critical for pathogenesis of Candida albicans

mSphere, 2024-03, Vol.9 (3), p.e0069623

2024

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Direct repression of MYB by ZEB1 suppresses proliferation and epithelial gene expression during epithelial-to-mesenchymal transition of breast cancer cells

Breast cancer research : BCR, 2013-11, Vol.15 (6), p.R113-R113, Article R113

2013

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AMP-activated protein kinase (AMPK) β1β2 muscle null mice reveal an essential role for AMPK in maintaining mitochondrial content and glucose uptake during exercise

Proceedings of the National Academy of Sciences - PNAS, 2011-09, Vol.108 (38), p.16092-16097

2011

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AMPK phosphorylation of ACC2 is required for skeletal muscle fatty acid oxidation and insulin sensitivity in mice

Diabetologia, 2014-08, Vol.57 (8), p.1693-1702

2014

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Adipose Triglyceride Lipase Regulation of Skeletal Muscle Lipid Metabolism and Insulin Responsiveness

Molecular endocrinology (Baltimore, Md.), 2008-05, Vol.22 (5), p.1200-1212

2008

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The functional GRHL3-filaggrin axis maintains a tumor differentiation potential and influences drug sensitivity

Molecular therapy, 2021-08, Vol.29 (8), p.2571-2582

2021

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Exemplary multiplex bisulfite amplicon data used to demonstrate the utility of Methpat

Gigascience, 2015-11, Vol.4 (1), p.55-55, Article 55

2015

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The to and fro of tumour spread

Nature (London), 2013-01, Vol.493 (7433), p.487-488

2013

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Cancer: The to and fro of tumour spread

Nature (London), 2013-01, Vol.493 (7433), p.487-488

2013

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Remodeling of purinergic receptor-mediated Ca2+ signaling as a consequence of EGF-induced epithelial-mesenchymal transition in breast cancer cells

PloS one, 2011-08, Vol.6 (8), p.e23464-e23464

2011

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AMPK couples plasma renin to cellular metabolism by phosphorylation of ACC1

American journal of physiology. Renal physiology, 2013-09, Vol.305 (5), p.F679-F690

2013

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AMP-Activated Protein Kinase Is Not Down-Regulated in Human Skeletal Muscle of Obese Females

The journal of clinical endocrinology and metabolism, 2004-09, Vol.89 (9), p.4575-4580

2004

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Tumor necrosis factor α-induced skeletal muscle insulin resistance involves suppression of AMP-kinase signaling

Cell metabolism, 2006-12, Vol.4 (6), p.465-474

2006

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Mutations in the Gal83 Glycogen-Binding Domain Activate the Snf1/Gal83 Kinase Pathway by a Glycogen-Independent Mechanism

Molecular and Cellular Biology, 2004-01, Vol.24 (1), p.352-361

2004

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Impaired Skeletal Muscle β-Adrenergic Activation and Lipolysis Are Associated with Whole-Body Insulin Resistance in Rats Bred for Low Intrinsic Exercise Capacity

Endocrinology (Philadelphia), 2009-11, Vol.150 (11), p.4883-4891

2009

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Skeletal muscle ACC2 S212 phosphorylation is not required for the control of fatty acid oxidation during exercise

Physiological reports, 2015-07, Vol.3 (7), p.e12444-n/a

2015

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AMPK/SNF1 structure: a menage a trois of energy-sensing

Frontiers in bioscience, 2009-01, Vol.14 (2), p.596-610

2009

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AMP-activated Protein Kinase Subunit Interactions: β1:γ1 ASSOCIATION REQUIRES β1 Thr-263 AND Tyr-267

The Journal of biological chemistry, 2008-02, Vol.283 (8), p.4799-4807

2008

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AMPK β1 Deletion Reduces Appetite, Preventing Obesity and Hepatic Insulin Resistance

The Journal of biological chemistry, 2010-01, Vol.285 (1), p.115-122

2010

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Whole Body Deletion of AMP-activated Protein Kinase β2 Reduces Muscle AMPK Activity and Exercise Capacity

The Journal of biological chemistry, 2010-11, Vol.285 (48), p.37198-37209

2010

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Structural Basis for Glycogen Recognition by AMP-Activated Protein Kinase

Structure (London), 2005-10, Vol.13 (10), p.1453-1462

2005

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AMP-activated Protein Kinase Subunit Interactions

The Journal of biological chemistry, 2008-02, Vol.283 (8), p.4799-4807

2008

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AMP-Activated Protein Kinase Regulates GLUT4 Transcription by Phosphorylating Histone Deacetylase 5

Single phosphorylation sites in Acc1 and Acc2 regulate lipid homeostasis and the insulin-sensitizing effects of metformin

Hematopoietic AMPK β1 reduces mouse adipose tissue macrophage inflammation and insulin resistance in obesity

The C-terminal protein interaction domain of the chromatin reader Yaf9 is critical for pathogenesis of Candida albicans

Direct repression of MYB by ZEB1 suppresses proliferation and epithelial gene expression during epithelial-to-mesenchymal transition of breast cancer cells

AMP-activated protein kinase (AMPK) β1β2 muscle null mice reveal an essential role for AMPK in maintaining mitochondrial content and glucose uptake during exercise

AMPK phosphorylation of ACC2 is required for skeletal muscle fatty acid oxidation and insulin sensitivity in mice

Adipose Triglyceride Lipase Regulation of Skeletal Muscle Lipid Metabolism and Insulin Responsiveness

The functional GRHL3-filaggrin axis maintains a tumor differentiation potential and influences drug sensitivity

Exemplary multiplex bisulfite amplicon data used to demonstrate the utility of Methpat

The to and fro of tumour spread

Cancer: The to and fro of tumour spread

Remodeling of purinergic receptor-mediated Ca2+ signaling as a consequence of EGF-induced epithelial-mesenchymal transition in breast cancer cells

AMPK couples plasma renin to cellular metabolism by phosphorylation of ACC1

AMP-Activated Protein Kinase Is Not Down-Regulated in Human Skeletal Muscle of Obese Females

Tumor necrosis factor α-induced skeletal muscle insulin resistance involves suppression of AMP-kinase signaling

Mutations in the Gal83 Glycogen-Binding Domain Activate the Snf1/Gal83 Kinase Pathway by a Glycogen-Independent Mechanism

Impaired Skeletal Muscle β-Adrenergic Activation and Lipolysis Are Associated with Whole-Body Insulin Resistance in Rats Bred for Low Intrinsic Exercise Capacity

Skeletal muscle ACC2 S212 phosphorylation is not required for the control of fatty acid oxidation during exercise

AMPK/SNF1 structure: a menage a trois of energy-sensing

AMP-activated Protein Kinase Subunit Interactions: β1:γ1 ASSOCIATION REQUIRES β1 Thr-263 AND Tyr-267

AMPK β1 Deletion Reduces Appetite, Preventing Obesity and Hepatic Insulin Resistance

Whole Body Deletion of AMP-activated Protein Kinase β2 Reduces Muscle AMPK Activity and Exercise Capacity

Structural Basis for Glycogen Recognition by AMP-Activated Protein Kinase

AMP-activated Protein Kinase Subunit Interactions

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