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Oligodendrocyte dysfunction in the pathogenesis of amyotrophic lateral sclerosis

Brain (London, England : 1878), 2013-02, Vol.136 (2), p.471-482

2013

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Efficient Recombinase-Mediated Cassette Exchange in hPSCs to Study the Hepatocyte Lineage Reveals AAVS1 Locus-Mediated Transgene Inhibition

Stem cell reports, 2015-11, Vol.5 (5), p.918-931

2015

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Defective axonal transport: A common pathological mechanism in inherited and acquired peripheral neuropathies

Neurobiology of disease, 2017-09, Vol.105, p.300-320

2017

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Progranulin functions as a neurotrophic factor to regulate neurite outgrowth and enhance neuronal survival

The Journal of cell biology, 2008-04, Vol.181 (1), p.37-41

2008

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Potential Therapeutic Role of HDAC Inhibitors in FUS-ALS

Frontiers in molecular neuroscience, 2021-08, Vol.14, p.686995-686995

2021

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Triad of TDP43 control in neurodegeneration: autoregulation, localization and aggregation

Nature reviews. Neuroscience, 2021-04, Vol.22 (4), p.197-208

2021

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RNA toxicity in non‐coding repeat expansion disorders

The EMBO journal, 2020-01, Vol.39 (1), p.e101112-n/a

2020

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Astrocyte-derived Jagged-1 mitigates deleterious Notch signaling in amyotrophic lateral sclerosis

Neurobiology of disease, 2018-11, Vol.119, p.26-40

2018

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Energy metabolism in ALS: an underappreciated opportunity?

Acta neuropathologica, 2018-04, Vol.135 (4), p.489-509

2018

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A generic approach to study the kinetics of liquid–liquid phase separation under near-native conditions

Communications biology, 2021-01, Vol.4 (1), p.77-77, Article 77

2021

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Protein Phase Separation: A New Phase in Cell Biology

Trends in cell biology, 2018-06, Vol.28 (6), p.420-435

2018

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Astrocytes regulate GluR2 expression in motor neurons and their vulnerability to excitotoxicity

Proceedings of the National Academy of Sciences - PNAS, 2007-09, Vol.104 (37), p.14825-14830

2007

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C9orf72 loss-of-function: a trivial, stand-alone or additive mechanism in C9 ALS/FTD?

Acta neuropathologica, 2020-11, Vol.140 (5), p.625-643

2020

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Antisense, but not sense, repeat expanded RNAs activate PKR/eIF2α-dependent ISR in C9ORF72 FTD/ALS

eLife, 2023-04, Vol.12

2023

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Generation of Human Induced Pluripotent Stem Cell (hiPSC)-Derived Astrocytes for Amyotrophic Lateral Sclerosis and Other Neurodegenerative Disease Studies

Bio-protocol, 2024-02, Vol.14 (4), p.e4936

2024

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Spontaneous driving forces give rise to protein−RNA condensates with coexisting phases and complex material properties

Proceedings of the National Academy of Sciences - PNAS, 2019-04, Vol.116 (16), p.7889-7898

2019

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Modelling amyotrophic lateral sclerosis: progress and possibilities

Disease models & mechanisms, 2017-05, Vol.10 (5), p.537-549

2017

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TDP-43 proteinopathies: a new wave of neurodegenerative diseases

Journal of neurology, neurosurgery and psychiatry, 2021-01, Vol.92 (1), p.86-95

2021

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Inside out: the role of nucleocytoplasmic transport in ALS and FTLD

Acta Neuropathologica, 2016-08, Vol.132 (2), p.159-173

2016

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Proteostasis plays an important role in demyelinating Charcot Marie Tooth disease

Biochemical pharmacology, 2023-10, Vol.216, p.115760-115760, Article 115760

2023

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Exploring the alternative: Fish, flies and worms as preclinical models for ALS

Neuroscience letters, 2021-08, Vol.759, p.136041-136041, Article 136041

2021

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Restoration of progranulin expression rescues cortical neuron generation in an induced pluripotent stem cell model of frontotemporal dementia

Stem cell reports, 2015-01, Vol.4 (1), p.16-24

2015

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Mutant FUS causes DNA ligation defects to inhibit oxidative damage repair in Amyotrophic Lateral Sclerosis

Nature communications, 2018-09, Vol.9 (1), p.3683-18, Article 3683

2018

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The Role of Nucleocytoplasmic Transport Defects in Amyotrophic Lateral Sclerosis

International journal of molecular sciences, 2021-11, Vol.22 (22), p.12175

2021

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Oligodendrocyte dysfunction in the pathogenesis of amyotrophic lateral sclerosis

Efficient Recombinase-Mediated Cassette Exchange in hPSCs to Study the Hepatocyte Lineage Reveals AAVS1 Locus-Mediated Transgene Inhibition

Defective axonal transport: A common pathological mechanism in inherited and acquired peripheral neuropathies

Progranulin functions as a neurotrophic factor to regulate neurite outgrowth and enhance neuronal survival

Potential Therapeutic Role of HDAC Inhibitors in FUS-ALS

Triad of TDP43 control in neurodegeneration: autoregulation, localization and aggregation

RNA toxicity in non‐coding repeat expansion disorders

Astrocyte-derived Jagged-1 mitigates deleterious Notch signaling in amyotrophic lateral sclerosis

Energy metabolism in ALS: an underappreciated opportunity?

A generic approach to study the kinetics of liquid–liquid phase separation under near-native conditions

Protein Phase Separation: A New Phase in Cell Biology

Astrocytes regulate GluR2 expression in motor neurons and their vulnerability to excitotoxicity

C9orf72 loss-of-function: a trivial, stand-alone or additive mechanism in C9 ALS/FTD?

Antisense, but not sense, repeat expanded RNAs activate PKR/eIF2α-dependent ISR in C9ORF72 FTD/ALS

Generation of Human Induced Pluripotent Stem Cell (hiPSC)-Derived Astrocytes for Amyotrophic Lateral Sclerosis and Other Neurodegenerative Disease Studies

Spontaneous driving forces give rise to protein−RNA condensates with coexisting phases and complex material properties

Modelling amyotrophic lateral sclerosis: progress and possibilities

TDP-43 proteinopathies: a new wave of neurodegenerative diseases

Inside out: the role of nucleocytoplasmic transport in ALS and FTLD

Proteostasis plays an important role in demyelinating Charcot Marie Tooth disease

Deletion of the hypoxia-response element in the vascular endothelial growth factor promoter causes motor neuron degeneration

Exploring the alternative: Fish, flies and worms as preclinical models for ALS

Restoration of progranulin expression rescues cortical neuron generation in an induced pluripotent stem cell model of frontotemporal dementia

Mutant FUS causes DNA ligation defects to inhibit oxidative damage repair in Amyotrophic Lateral Sclerosis

The Role of Nucleocytoplasmic Transport Defects in Amyotrophic Lateral Sclerosis

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