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Serum Amyloid A Is a Ligand for Scavenger Receptor Class B Type I and Inhibits High Density Lipoprotein Binding and Selective Lipid Uptake

The Journal of biological chemistry, 2005-01, Vol.280 (4), p.2954-2961

2005

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Serum amyloid A3 is a high density lipoprotein-associated acute-phase protein

Journal of lipid research, 2018-02, Vol.59 (2), p.339-347

2018

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Serum amyloid A binds to fibrin(ogen), promoting fibrin amyloid formation

Scientific reports, 2019-02, Vol.9 (1), p.3102-3102, Article 3102

2019

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Serum amyloid A is not incorporated into HDL during HDL biogenesis[S]

Journal of lipid research, 2020-03, Vol.61 (3), p.328-337

2020

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HDL remodeling during the acute phase response

Arteriosclerosis, thrombosis, and vascular biology, 2009-02, Vol.29 (2), p.261-267

2009

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Nascent HDL formation in hepatocytes and role of ABCA1, ABCG1, and SR-BI

Journal of lipid research, 2012-03, Vol.53 (3), p.446-455

2012

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High-density lipoprotein inhibits serum amyloid A–mediated reactive oxygen species generation and NLRP3 inflammasome activation

The Journal of biological chemistry, 2018-08, Vol.293 (34), p.13257-13269

2018

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Impact of individual acute phase serum amyloid A isoforms on HDL metabolism in mice[S]

Journal of lipid research, 2016-06, Vol.57 (6), p.969-979

2016

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Serum Amyloid A Promotes Cholesterol Efflux Mediated by Scavenger Receptor B-I

The Journal of biological chemistry, 2005-10, Vol.280 (43), p.35890-35895

2005

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Impact of serum amyloid A on high density lipoprotein composition and levels

Journal of lipid research, 2010-11, Vol.51 (11), p.3117-3125

2010

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Serum amyloid A impairs the antiinflammatory properties of HDL

The Journal of clinical investigation, 2016-01, Vol.126 (1), p.266-281

2016

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Serum Amyloid A Is an Exchangeable Apolipoprotein

Arteriosclerosis, thrombosis, and vascular biology, 2018-08, Vol.38 (8), p.1890-1900

2018

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High-Density Lipoprotein Suppresses the Type I Interferon Response, a Family of Potent Antiviral Immunoregulators, in Macrophages Challenged With Lipopolysaccharide

Circulation (New York, N.Y.), 2010-11, Vol.122 (19), p.1919-1927

2010

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Deficiency of Endogenous Acute-Phase Serum Amyloid A Protects apoE-/- Mice From Angiotensin II-Induced Abdominal Aortic Aneurysm Formation

Arteriosclerosis, thrombosis, and vascular biology, 2015-05, Vol.35 (5), p.1156-1165

2015

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Deficiency of endogenous acute phase serum amyloid A does not affect atherosclerotic lesions in apolipoprotein E-deficient mice

Arteriosclerosis, thrombosis, and vascular biology, 2014-02, Vol.34 (2), p.255-261

2014

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Serum amyloid A impairs the antiinflammatory properties of HDL

The Journal of clinical investigation, 2016-02, Vol.126 (2), p.796-796

2016

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SAA does not induce cytokine production in physiological conditions

Cytokine (Philadelphia, Pa.), 2013-02, Vol.61 (2), p.506-512

2013

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Serum amyloid A facilitates the binding of high-density lipoprotein from mice injected with lipopolysaccharide to vascular proteoglycans

Arteriosclerosis, thrombosis, and vascular biology, 2011-06, Vol.31 (6), p.1326-1332

2011

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Low-density lipoprotein from apolipoprotein E-deficient mice induces macrophage lipid accumulation in a CD36 and scavenger receptor class A-dependent manner

Arteriosclerosis, thrombosis, and vascular biology, 2005-01, Vol.25 (1), p.168-173

2005

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The Impairment of Macrophage-to-Feces Reverse Cholesterol Transport during Inflammation Does Not Depend on Serum Amyloid A

Journal of Lipids, 2013-01, Vol.2013 (2013), p.57-67

2013

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Scavenger receptor SR-BI in macrophage lipid metabolism

Atherosclerosis, 2011-07, Vol.217 (1), p.106-112

2011

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Serum Amyloid A Facilitates Early Lesion Development in Ldlr-/- Mice

Journal of the American Heart Association, 2015-07, Vol.4 (7)

2015

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ATP binding cassette G1-dependent cholesterol efflux during inflammation

Journal of lipid research, 2011-02, Vol.52 (2), p.345-353

2011

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Lipoproteins are substrates for human secretory group IIA phospholipase A2: preferential hydrolysis of acute phase HDL

Journal of lipid research, 1998-11, Vol.39 (11), p.2150-2160

1998

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Lack of a direct role for macrosialin in oxidized LDL metabolism

Journal of lipid research, 2003-04, Vol.44 (4), p.674-685

2003

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Serum Amyloid A Is a Ligand for Scavenger Receptor Class B Type I and Inhibits High Density Lipoprotein Binding and Selective Lipid Uptake

Serum amyloid A3 is a high density lipoprotein-associated acute-phase protein

Serum amyloid A binds to fibrin(ogen), promoting fibrin amyloid formation

Serum amyloid A is not incorporated into HDL during HDL biogenesis[S]

HDL remodeling during the acute phase response

Nascent HDL formation in hepatocytes and role of ABCA1, ABCG1, and SR-BI

High-density lipoprotein inhibits serum amyloid A–mediated reactive oxygen species generation and NLRP3 inflammasome activation

Impact of individual acute phase serum amyloid A isoforms on HDL metabolism in mice[S]

Serum Amyloid A Promotes Cholesterol Efflux Mediated by Scavenger Receptor B-I

Impact of serum amyloid A on high density lipoprotein composition and levels

Serum amyloid A impairs the antiinflammatory properties of HDL

Serum Amyloid A Is an Exchangeable Apolipoprotein

High-Density Lipoprotein Suppresses the Type I Interferon Response, a Family of Potent Antiviral Immunoregulators, in Macrophages Challenged With Lipopolysaccharide

Deficiency of Endogenous Acute-Phase Serum Amyloid A Protects apoE-/- Mice From Angiotensin II-Induced Abdominal Aortic Aneurysm Formation

Deficiency of endogenous acute phase serum amyloid A does not affect atherosclerotic lesions in apolipoprotein E-deficient mice

Serum amyloid A impairs the antiinflammatory properties of HDL

SAA does not induce cytokine production in physiological conditions

Serum amyloid A facilitates the binding of high-density lipoprotein from mice injected with lipopolysaccharide to vascular proteoglycans

Low-density lipoprotein from apolipoprotein E-deficient mice induces macrophage lipid accumulation in a CD36 and scavenger receptor class A-dependent manner

The Impairment of Macrophage-to-Feces Reverse Cholesterol Transport during Inflammation Does Not Depend on Serum Amyloid A

Scavenger receptor SR-BI in macrophage lipid metabolism

Serum Amyloid A Facilitates Early Lesion Development in Ldlr-/- Mice

ATP binding cassette G1-dependent cholesterol efflux during inflammation

Lipoproteins are substrates for human secretory group IIA phospholipase A2: preferential hydrolysis of acute phase HDL

Lack of a direct role for macrosialin in oxidized LDL metabolism

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