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Functional transcription promoters at DNA double-strand breaks mediate RNA-driven phase separation of damage-response factors

Nature cell biology, 2019-10, Vol.21 (10), p.1286

2019

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Site-specific DICER and DROSHA RNA products control the DNA―damage response

Nature (London), 2012-08, Vol.488 (7410), p.231-235

2012

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Telomeric DNA damage is irreparable and causes persistent DNA-damage-response activation

Nature cell biology, 2012-04, Vol.14 (4), p.355

2012

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BRCA2 controls DNA:RNA hybrid level at DSBs by mediating RNase H2 recruitment

Nature communications, 2018-12, Vol.9 (1), p.5376-17, Article 5376

2018

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From “Cellular” RNA to “Smart” RNA: Multiple Roles of RNA in Genome Stability and Beyond

Chemical reviews, 2018-04, Vol.118 (8), p.4365-4403

2018

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Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication

Nature, 2006-11, Vol.444 (7119), p.638-642

2006

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Inhibition of DNA damage response at telomeres improves the detrimental phenotypes of Hutchinson-Gilford Progeria Syndrome

Nature communications, 2019-11, Vol.10 (1), p.4990-11, Article 4990

2019

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DNA damage response inhibition at dysfunctional telomeres by modulation of telomeric DNA damage response RNAs

Nature communications, 2017-02, Vol.8 (1), p.13980-14, Article 13980

2017

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DNA damage response at telomeres boosts the transcription of SARS‐CoV‐2 receptor ACE2 during aging

EMBO reports, 2022-02, Vol.23 (2), p.e53658-n/a

2022

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BRCA1 deficiency specific base substitution mutagenesis is dependent on translesion synthesis and regulated by 53BP1

Nature communications, 2022-01, Vol.13 (1), p.226-13, Article 226

2022

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Interplay between oncogene-induced DNA damage response and heterochromatin in senescence and cancer

Nature cell biology, 2011-03, Vol.13 (3), p.292

2011

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A damaged genome's transcriptional landscape through multilayered expression profiling around in situ-mapped DNA double-strand breaks

Nature communications, 2017-05, Vol.8 (1), p.15656-12, Article 15656

2017

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Cellular senescence: when bad things happen to good cells

Nature reviews. Molecular cell biology, 2007-09, Vol.8 (9), p.729-740

2007

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Pharmacological boost of DNA damage response and repair by enhanced biogenesis of DNA damage response RNAs

Scientific reports, 2019-04, Vol.9 (1), p.6460, Article 6460

2019

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MRE11-RAD50-NBS1 Complex Is Sufficient to Promote Transcription by RNA Polymerase II at Double-Strand Breaks by Melting DNA Ends

Cell reports (Cambridge), 2021-01, Vol.34 (1), p.108565-108565, Article 108565

2021

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Cellular senescence in ageing: from mechanisms to therapeutic opportunities

Nature reviews. Molecular cell biology, 2021-02, Vol.22 (2), p.75

2021

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A DNA damage checkpoint response in telomere-initiated senescence

Nature, 2003-11, Vol.426 (6963), p.194-198

2003

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Polycomb proteins control proliferation and transformation independently of cell cycle checkpoints by regulating DNA replication

Nature communications, 2014-04, Vol.5 (1), p.3649, Article 3649

2014

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Alternative lengthening of telomeres (ALT) cells viability is dependent on C-rich telomeric RNAs

Nature communications, 2023-11, Vol.14 (1), p.7086-7086, Article 7086

2023

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Damage-induced lncRNAs control the DNA damage response through interaction with DDRNAs at individual double-strand breaks

Nature cell biology, 2017-12, Vol.19 (12), p.1400

2017

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Transcription and DNA Damage: Holding Hands or Crossing Swords?

Journal of molecular biology, 2017-10, Vol.429 (21), p.3215-3229

2017

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Crosstalk between chromatin state and DNA damage response in cellular senescence and cancer

Nature reviews. Cancer, 2012-10, Vol.12 (10), p.709

2012

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Colorectal cancer patient-derived organoids and cell lines harboring ATRX and/or DAXX mutations lack Alternative Lengthening of Telomeres (ALT)

Cell death & disease, 2023-02, Vol.14 (2), p.96-96, Article 96

2023

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Telomere dysfunction in ageing and age-related diseases

Nature cell biology, 2022-02, Vol.24 (2), p.135

2022

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DICER, DROSHA and DNA damage response RNAs are necessary for the secondary recruitment of DNA damage response factors

Journal of cell science, 2016-04, Vol.129 (7), p.1468-1476

2016

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Functional transcription promoters at DNA double-strand breaks mediate RNA-driven phase separation of damage-response factors

Site-specific DICER and DROSHA RNA products control the DNA―damage response

Telomeric DNA damage is irreparable and causes persistent DNA-damage-response activation

BRCA2 controls DNA:RNA hybrid level at DSBs by mediating RNase H2 recruitment

From “Cellular” RNA to “Smart” RNA: Multiple Roles of RNA in Genome Stability and Beyond

Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication

Inhibition of DNA damage response at telomeres improves the detrimental phenotypes of Hutchinson-Gilford Progeria Syndrome

DNA damage response inhibition at dysfunctional telomeres by modulation of telomeric DNA damage response RNAs

DNA damage response at telomeres boosts the transcription of SARS‐CoV‐2 receptor ACE2 during aging

BRCA1 deficiency specific base substitution mutagenesis is dependent on translesion synthesis and regulated by 53BP1

Interplay between oncogene-induced DNA damage response and heterochromatin in senescence and cancer

A damaged genome's transcriptional landscape through multilayered expression profiling around in situ-mapped DNA double-strand breaks

Cellular senescence: when bad things happen to good cells

Pharmacological boost of DNA damage response and repair by enhanced biogenesis of DNA damage response RNAs

MRE11-RAD50-NBS1 Complex Is Sufficient to Promote Transcription by RNA Polymerase II at Double-Strand Breaks by Melting DNA Ends

Cellular senescence in ageing: from mechanisms to therapeutic opportunities

A DNA damage checkpoint response in telomere-initiated senescence

Polycomb proteins control proliferation and transformation independently of cell cycle checkpoints by regulating DNA replication

Alternative lengthening of telomeres (ALT) cells viability is dependent on C-rich telomeric RNAs

Damage-induced lncRNAs control the DNA damage response through interaction with DDRNAs at individual double-strand breaks

Transcription and DNA Damage: Holding Hands or Crossing Swords?

Crosstalk between chromatin state and DNA damage response in cellular senescence and cancer

Colorectal cancer patient-derived organoids and cell lines harboring ATRX and/or DAXX mutations lack Alternative Lengthening of Telomeres (ALT)

Telomere dysfunction in ageing and age-related diseases

DICER, DROSHA and DNA damage response RNAs are necessary for the secondary recruitment of DNA damage response factors

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