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Blood-based protein profiling identifies serum protein c-KIT as a novel biomarker for hypertrophic cardiomyopathy

Scientific reports, 2021-01, Vol.11 (1), p.1755-11, Article 1755

2021

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C1q-TNF-Related Protein-9 Promotes Cardiac Hypertrophy and Failure

Circulation research, 2017-01, Vol.120 (1), p.66-77

2017

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Predictors of Mortality in Patients With Biopsy-Proven Viral Myocarditis: 10-Year Outcome Data

Journal of the American Heart Association, 2020-08, Vol.9 (16), p.e015351-e015351

2020

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Thromboembolic characteristics and role of anticoagulation in long-standing Fontan circulation

International journal of cardiology congenital heart disease, 2022-03, Vol.7, p.100328, Article 100328

2022

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Cabergoline treatment promotes myocardial recovery in peripartum cardiomyopathy

ESC Heart Failure, 2023-02, Vol.10 (1), p.465-477

2023

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C1q and Tumor Necrosis Factor Related Protein 9 Protects from Diabetic Cardiomyopathy by Alleviating Cardiac Insulin Resistance and Inflammation

Cells (Basel, Switzerland), 2023-01, Vol.12 (3), p.443

2023

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Mineralocorticoid receptor antagonists for therapy of coronary artery disease and related complications

Current opinion in pharmacology, 2013-04, Vol.13 (2), p.280-286

2013

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Molecular Imaging Identifies Fibroblast Activation Beyond the Infarct Region After Acute Myocardial Infarction

Journal of the American College of Cardiology, 2021-04, Vol.77 (14), p.1835-1837

2021

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Serum circular RNAs act as blood-based biomarkers for hypertrophic obstructive cardiomyopathy

Scientific reports, 2019-12, Vol.9 (1), p.20350-8, Article 20350

2019

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Giant cell myocarditis after first dose of BNT162b2 – a case report

European journal of heart failure, 2022-07, Vol.24 (7), p.1319-1322

2022

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Antiandrogenic Therapy With Finasteride Attenuates Cardiac Hypertrophy and Left Ventricular Dysfunction

Circulation (New York, N.Y.), 2015-03, Vol.131 (12), p.1071-1081

2015

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Molecular Imaging of Myocardial Fibroblast Activation in Patients with Advanced Aortic Stenosis Before Transcatheter Aortic Valve Replacement: A Pilot Study

Journal of Nuclear Medicine, 2023-08, Vol.64 (8), p.1279-1286

2023

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Catch me if you can

International journal of cardiology, 2012-02, Vol.155 (1), p.e13-e13

2012

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Gene expression profiling of calcium-channel antagonists in the heart of hypertensive and normotensive rats reveals class specific effects

Vascular pharmacology, 2016-12, Vol.87, p.121-128

2016

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Anti-androgenic therapy with finasteride in patients with chronic heart failure - a retrospective propensity score based analysis

Scientific reports, 2019-07, Vol.9 (1), p.10139-8, Article 10139

2019

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Prediction of heart failure and death in an adult population of Fontan patients

Cardiology in the young, 2019-05, Vol.29 (5), p.602-609

2019

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Anti-androgenic therapy with finasteride improves cardiac function, attenuates remodeling and reverts pathologic gene-expression after myocardial infarction in mice

Journal of molecular and cellular cardiology, 2018-09, Vol.122, p.114-124

2018

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Dihydrotestosterone—a culprit in left ventricular hypertrophy

International journal of cardiology, 2012-03, Vol.155 (3), p.452-456

2012

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Abstract 153: Inhibition of the 5-a-reductase Reduces Cardiac Hypertrophy and Improves Cardiac Function

Circulation research, 2013-08, Vol.113 (suppl_1)

2013

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Bromocriptine treatment in patients with peripartum cardiomyopathy and right ventricular dysfunction

Clinical research in cardiology, 2019-03, Vol.108 (3), p.290-297

2019

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Corrigendum to “Anti-androgenic therapy with finasteride improves cardiac function, attenuates remodeling and reverts pathologic gene-expression after myocardial infarction in mice” Journal of Molecular and Cellular Cardiology 122 (2018) 114–124

Journal of molecular and cellular cardiology, 2018-12, Vol.125, p.205-205

2018

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The myosin ATPase inhibitor 2,3-butanedione monoxime dictates transcriptional activation of ion channels and Ca(2+)-handling proteins

Molecular pharmacology, 2004-09, Vol.66 (3), p.708

2004

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Disease-associated changes in the expression of ion channels, ion receptors, ion exchangers and Ca(2+)-handling proteins in heart hypertrophy

Toxicology and applied pharmacology, 2005-09, Vol.207 (3), p.244-256

2005

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Disease-associated changes in the expression of ion channels, ion receptors, ion exchangers and Ca2+-handling proteins in heart hypertrophy

Toxicology and applied pharmacology, 2005-09, Vol.207 (3), p.244-256

2005

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Blood-based protein profiling identifies serum protein c-KIT as a novel biomarker for hypertrophic cardiomyopathy

C1q-TNF-Related Protein-9 Promotes Cardiac Hypertrophy and Failure

Predictors of Mortality in Patients With Biopsy-Proven Viral Myocarditis: 10-Year Outcome Data

Thromboembolic characteristics and role of anticoagulation in long-standing Fontan circulation

Cabergoline treatment promotes myocardial recovery in peripartum cardiomyopathy

C1q and Tumor Necrosis Factor Related Protein 9 Protects from Diabetic Cardiomyopathy by Alleviating Cardiac Insulin Resistance and Inflammation

Mineralocorticoid receptor antagonists for therapy of coronary artery disease and related complications

Molecular Imaging Identifies Fibroblast Activation Beyond the Infarct Region After Acute Myocardial Infarction

Serum circular RNAs act as blood-based biomarkers for hypertrophic obstructive cardiomyopathy

Giant cell myocarditis after first dose of BNT162b2 – a case report

Skeletal muscle derived Musclin protects the heart during pathological overload

Antiandrogenic Therapy With Finasteride Attenuates Cardiac Hypertrophy and Left Ventricular Dysfunction

Molecular Imaging of Myocardial Fibroblast Activation in Patients with Advanced Aortic Stenosis Before Transcatheter Aortic Valve Replacement: A Pilot Study

Catch me if you can

Gene expression profiling of calcium-channel antagonists in the heart of hypertensive and normotensive rats reveals class specific effects

Anti-androgenic therapy with finasteride in patients with chronic heart failure - a retrospective propensity score based analysis

Prediction of heart failure and death in an adult population of Fontan patients

Anti-androgenic therapy with finasteride improves cardiac function, attenuates remodeling and reverts pathologic gene-expression after myocardial infarction in mice

Dihydrotestosterone—a culprit in left ventricular hypertrophy

Abstract 153: Inhibition of the 5-a-reductase Reduces Cardiac Hypertrophy and Improves Cardiac Function

Bromocriptine treatment in patients with peripartum cardiomyopathy and right ventricular dysfunction

Corrigendum to “Anti-androgenic therapy with finasteride improves cardiac function, attenuates remodeling and reverts pathologic gene-expression after myocardial infarction in mice” Journal of Molecular and Cellular Cardiology 122 (2018) 114–124

The myosin ATPase inhibitor 2,3-butanedione monoxime dictates transcriptional activation of ion channels and Ca(2+)-handling proteins

Disease-associated changes in the expression of ion channels, ion receptors, ion exchangers and Ca(2+)-handling proteins in heart hypertrophy

Disease-associated changes in the expression of ion channels, ion receptors, ion exchangers and Ca2+-handling proteins in heart hypertrophy

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