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Assessing bioenergetic function in response to oxidative stress by metabolic profiling

Free radical biology & medicine, 2011-11, Vol.51 (9), p.1621-1635

2011

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Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal

Biochemical journal, 2009-10, Vol.424 (1), p.99-107

2009

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Modification of STIM1 by O-linked N-Acetylglucosamine (O-GlcNAc) Attenuates Store-operated Calcium Entry in Neonatal Cardiomyocytes

The Journal of biological chemistry, 2012-11, Vol.287 (46), p.39094-39106

2012

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O-GlcNAcylation, Novel Post-Translational Modification Linking Myocardial Metabolism and Cardiomyocyte Circadian Clock

The Journal of biological chemistry, 2011-12, Vol.286 (52), p.44606-44619

2011

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Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function

American journal of physiology. Heart and circulatory physiology, 2014-04, Vol.306 (8), p.H1231-H1239

2014

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Sustained Increases in Cardiomyocyte Protein O -Linked β-N-Acetylglucosamine Levels Lead to Cardiac Hypertrophy and Reduced Mitochondrial Function Without Systolic Contractile Impairment

Journal of the American Heart Association, 2023-10, Vol.12 (19), p.e029898

2023

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The Identification of a Novel Calcium-Dependent Link Between NAD + and Glucose Deprivation-Induced Increases in Protein O-GlcNAcylation and ER Stress

Frontiers in molecular biosciences, 2021-12, Vol.8, p.780865-780865

2021

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Aging influences cardiac mitochondrial gene expression and cardiovascular function following hemorrhage injury

Molecular medicine (Cambridge, Mass.), 2011-05, Vol.17 (5-6), p.542-549

2011

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Nicotinamide adenine dinucleotide hydrate (NAD+) regulates glucose deprivation‐induced activation of protein O‐GlcNAcylation, ER stress and autophagy (1154.2)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a

2014

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Best practices in assessing cardiac protein O -GlcNAcylation by immunoblot

American journal of physiology. Heart and circulatory physiology, 2023-10, Vol.325 (4), p.H601-H616

2023

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The effects of NAD on glucose deprivation induced activation of protein O‐GlcNAcylation, ER stress and autophagy mediated by CD38 and cADPR

The FASEB journal, 2015-04, Vol.29 (S1)

2015

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The role of STIM1 and store operated calcium entry in glucose‐induced insulin secretion

The FASEB journal, 2013-04, Vol.27 (S1)

2013

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Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O -GlcNAcylation and attenuation of NF-κB signaling

American journal of physiology. Heart and circulatory physiology, 2009-02, Vol.296 (2), p.H515-H523

2009

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Acute increases in O -GlcNAc indirectly impair mitochondrial bioenergetics through dysregulation of LonP1-mediated mitochondrial protein complex turnover

American Journal of Physiology: Cell Physiology, 2019-06, Vol.316 (6), p.C862-C875

2019

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Novel role of the ER/SR Ca2+ sensor STIM1 in the regulation of cardiac metabolism

American journal of physiology. Heart and circulatory physiology, 2019-05, Vol.316 (5), p.H1014-H1026

2019

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Glucose Deprivation-induced Increase in Protein O-GlcNAcylation in Cardiomyocytes Is Calcium-dependent

The Journal of biological chemistry, 2012-10, Vol.287 (41), p.34419-34431

2012

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In cardiomyocytes glucose deprivation induced increase in O‐GlcNAc is calcium dependent

The FASEB journal, 2011-04, Vol.25 (S1), p.1033.20-1033.20

2011

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Stress induced increase in protein O‐linked‐N‐acetylglucosamine (O‐GlcNAc) levels is CaMKII dependent

The FASEB journal, 2012-04, Vol.26 (S1)

2012

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Adult rat cardiomyocytes exhibit capacitative calcium entry

American journal of physiology. Heart and circulatory physiology, 2004-03, Vol.55 (3), p.H1124-H1132

2004

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Glucose deprivation induced Cyclooxygenase‐2 and Heme oxygenase‐1 expression via P38 pathway in neonatal rat ventricular cardiomyocytes

The FASEB journal, 2010-04, Vol.24 (S1), p.787.5-787.5

2010

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Novel role of the ER/SR Ca 2+ sensor STIM1 in the regulation of cardiac metabolism

American journal of physiology. Heart and circulatory physiology, 2019-05, Vol.316 (5), p.H1014-H1026

2019

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High-fat, low-carbohydrate diet promotes arrhythmic death and increases myocardial ischemia-reperfusion injury in rats

American journal of physiology. Heart and circulatory physiology, 2014-08, Vol.307 (4), p.H598-H608

2014

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In cardiomyocytes LPS‐induced activation of IκBα and COX‐2 expression are mediated in part by the hexosamine biosynthesis pathway

The FASEB journal, 2012-04, Vol.26 (S1)

2012

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Nicotinamide adenine dinucleotide hydrate (NAD) contributes to the regulation of O‐linked‐N‐acetylglucosamine (O‐GlcNAc) levels on cardiomyocyte proteins

The FASEB journal, 2011-04, Vol.25 (S1), p.1097.3-1097.3

2011

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The ER/SR Ca2+ sensing protein STIM1 is essential for normal cardiomyocyte function (1150.8)

The FASEB journal, 2014-04, Vol.28, p.n/a

2014

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Assessing bioenergetic function in response to oxidative stress by metabolic profiling

Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal

Modification of STIM1 by O-linked N-Acetylglucosamine (O-GlcNAc) Attenuates Store-operated Calcium Entry in Neonatal Cardiomyocytes

O-GlcNAcylation, Novel Post-Translational Modification Linking Myocardial Metabolism and Cardiomyocyte Circadian Clock

Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function

Sustained Increases in Cardiomyocyte Protein O -Linked β-N-Acetylglucosamine Levels Lead to Cardiac Hypertrophy and Reduced Mitochondrial Function Without Systolic Contractile Impairment

The Identification of a Novel Calcium-Dependent Link Between NAD + and Glucose Deprivation-Induced Increases in Protein O-GlcNAcylation and ER Stress

Aging influences cardiac mitochondrial gene expression and cardiovascular function following hemorrhage injury

Nicotinamide adenine dinucleotide hydrate (NAD+) regulates glucose deprivation‐induced activation of protein O‐GlcNAcylation, ER stress and autophagy (1154.2)

Best practices in assessing cardiac protein O -GlcNAcylation by immunoblot

The effects of NAD on glucose deprivation induced activation of protein O‐GlcNAcylation, ER stress and autophagy mediated by CD38 and cADPR

The role of STIM1 and store operated calcium entry in glucose‐induced insulin secretion

Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O -GlcNAcylation and attenuation of NF-κB signaling

Acute increases in O -GlcNAc indirectly impair mitochondrial bioenergetics through dysregulation of LonP1-mediated mitochondrial protein complex turnover

Novel role of the ER/SR Ca2+ sensor STIM1 in the regulation of cardiac metabolism

Glucose Deprivation-induced Increase in Protein O-GlcNAcylation in Cardiomyocytes Is Calcium-dependent

In cardiomyocytes glucose deprivation induced increase in O‐GlcNAc is calcium dependent

Stress induced increase in protein O‐linked‐N‐acetylglucosamine (O‐GlcNAc) levels is CaMKII dependent

Adult rat cardiomyocytes exhibit capacitative calcium entry

Glucose deprivation induced Cyclooxygenase‐2 and Heme oxygenase‐1 expression via P38 pathway in neonatal rat ventricular cardiomyocytes

Novel role of the ER/SR Ca 2+ sensor STIM1 in the regulation of cardiac metabolism

High-fat, low-carbohydrate diet promotes arrhythmic death and increases myocardial ischemia-reperfusion injury in rats

In cardiomyocytes LPS‐induced activation of IκBα and COX‐2 expression are mediated in part by the hexosamine biosynthesis pathway

Nicotinamide adenine dinucleotide hydrate (NAD) contributes to the regulation of O‐linked‐N‐acetylglucosamine (O‐GlcNAc) levels on cardiomyocyte proteins

The ER/SR Ca2+ sensing protein STIM1 is essential for normal cardiomyocyte function (1150.8)

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