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Italian neurologists’ perception on cognitive symptoms in major depressive disorder

Neurological sciences, 2016-09, Vol.37 (9), p.1437-1442

2016

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High frequency of exon 10 mutations in the NOTCH3 gene in Italian CADASIL families: phenotypic peculiarities

Journal of neurology, 2010-06, Vol.257 (6), p.1039-1042

2010

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The economic burden of stroke in Italy. The EcLIPSE Study: Economic Longitudinal Incidence-based Project for Stroke Evaluation

Neurological sciences, 2005-06, Vol.26 (2), p.72-80

2005

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Comparison of extent of tau pathology in patients with frontotemporal dementia with Parkinsonism linked to chromosome 17 (FTDP-17), frontotemporal lobar degeneration with Pick bodies and early onset Alzheimer's disease

Neuropathology and applied neurobiology, 2006-08, Vol.32 (4), p.374-387

2006

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Cognitive impairment and its relation with disease measures in mildly disabled patients with relapsing–remitting multiple sclerosis: baseline results from the Cognitive Impairment in Multiple Sclerosis (COGIMUS) study

Multiple sclerosis, 2009-07, Vol.15 (7), p.779-788

2009

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Subcutaneous interferon β-1a may protect against cognitive impairment in patients with relapsing-remitting multiple sclerosis: 5-year follow-up of the COGIMUS study

PloS one, 2013-08, Vol.8 (8), p.e74111

2013

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Slow wave and rem sleep mechanisms are differently altered in hereditary pick disease associated with the TAU G389R mutation

Archives italiennes de biologie, 2005-02, Vol.143 (1), p.65

2005

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Activation of the JNK/p38 Pathway Occurs in Diseases Characterized by Tau Protein Pathology and Is Related to Tau Phosphorylation But Not to Apoptosis

Journal of neuropathology and experimental neurology, 2001-12, Vol.60 (12), p.1190-1197

2001

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Quality of life, depression and fatigue in mildly disabled patients with relapsing–remitting multiple sclerosis receiving subcutaneous interferon beta-1a: 3-year results from the COGIMUS (COGnitive Impairment in MUltiple Sclerosis) study

Multiple sclerosis, 2011-08, Vol.17 (8), p.991-1001

2011

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Tau Gene Mutation G389R Causes a Tauopathy with Abundant Pick Body-like Inclusions and Axonal Deposits

Journal of neuropathology and experimental neurology, 1999-12, Vol.58 (12), p.1207-1226

1999

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Treatment of first tonic–clonic seizure does not affect mortality: long-term follow-up of a randomised clinical trial

Journal of neurology, neurosurgery and psychiatry, 2011-08, Vol.82 (8), p.924-927

2011

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Changes in magnetic resonance imaging disease measures over 3 years in mildly disabled patients with relapsing-remitting multiple sclerosis receiving interferon β-1a in the COGnitive Impairment in MUltiple Sclerosis (COGIMUS) study

BMC neurology, 2011-10, Vol.11 (1), p.125-125, Article 125

2011

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Progress in Hereditary Tauopathies: A Mutation in the Tau Gene (G389R) Causes a Pick Disease-like Syndrome

Annals of the New York Academy of Sciences, 2000-01, Vol.920 (1), p.52-62

2000

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Social Aspects of Epilepsy in the Adult in Seven European Countries

Epilepsia (Copenhagen), 2000-08, Vol.41 (8), p.998-1004

The RESt‐1 Group

2000

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Psychiatric events in epilepsy

Seizure (London, England), 2007-10, Vol.16 (7), p.586-592

2007

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Effects of frontal lesions on a selective attention task

Italian journal of neurological sciences, 1993-06, Vol.14 (5), p.355-359

1993

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Cost-of-illness of epilepsy in Italy. Data from a multicentre observational study (Episcreen)

PharmacoEconomics, 2000-02, Vol.17 (2), p.197-208

2000

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Prognosis of Epilepsy in Newly Referred Patients: A Multicenter Prospective Study of the Effects of Monotherapy on the Long‐Term Course of Epilepsy

Epilepsia (Copenhagen), 1992-01, Vol.33 (1), p.45-51

Collaborative Group for the Study of Epilepsy

1992

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Istiocitosi a cellule di Langherans: Coinvolgimento pachimeningeo ed otomastoideo

Rivista di neuroradiologia, 2003-05, Vol.16 (1_suppl), p.177-178

2003

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Predictive performance of pharmacokinetic methods for phenytoin dosing: a multi-center evaluation in 282 patients with epilepsy

Epilepsy research, 1989-05, Vol.3 (3), p.253-261

1989

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Effects of immunomodulatory treatment with subcutaneous interferon beta-1a on cognitive decline in mildly disabled patients with relapsing-remitting multiple sclerosis

Multiple sclerosis, 2010-01, Vol.16 (1), p.68-77

2010

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PICK DISEASE WITH GLIAL TAU INCLUSIONS IN THE WHITE MATTER

Journal of neuropathology and experimental neurology, 1999-05, Vol.58 (5), p.546

1999

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Allopurinol as Add‐on Therapy in Refractory Epilepsy: A Double‐Blind Placebo‐Controlled Randomized Study Italy, on his retirement

Epilepsia (Copenhagen), 1994-01, Vol.35 (1), p.107-112

1994

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Allopurinol as add-on therapy in refractory epilepsy : a double-blind placebo-controlled randomized study

Epilepsia (Copenhagen), 1994, Vol.35 (1), p.107-112

1994

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Concordance of Clinical Forms of Epilepsy in Families with Several Affected Members

Epilepsia (Copenhagen), 1993-09, Vol.34 (5), p.819-826

Italian League Against Epilepsy Genetic Collaborative Group

1993

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Italian neurologists’ perception on cognitive symptoms in major depressive disorder

High frequency of exon 10 mutations in the NOTCH3 gene in Italian CADASIL families: phenotypic peculiarities

The economic burden of stroke in Italy. The EcLIPSE Study: Economic Longitudinal Incidence-based Project for Stroke Evaluation

Comparison of extent of tau pathology in patients with frontotemporal dementia with Parkinsonism linked to chromosome 17 (FTDP-17), frontotemporal lobar degeneration with Pick bodies and early onset Alzheimer's disease

Cognitive impairment and its relation with disease measures in mildly disabled patients with relapsing–remitting multiple sclerosis: baseline results from the Cognitive Impairment in Multiple Sclerosis (COGIMUS) study

Subcutaneous interferon β-1a may protect against cognitive impairment in patients with relapsing-remitting multiple sclerosis: 5-year follow-up of the COGIMUS study

Slow wave and rem sleep mechanisms are differently altered in hereditary pick disease associated with the TAU G389R mutation

Activation of the JNK/p38 Pathway Occurs in Diseases Characterized by Tau Protein Pathology and Is Related to Tau Phosphorylation But Not to Apoptosis

Quality of life, depression and fatigue in mildly disabled patients with relapsing–remitting multiple sclerosis receiving subcutaneous interferon beta-1a: 3-year results from the COGIMUS (COGnitive Impairment in MUltiple Sclerosis) study

Tau Gene Mutation G389R Causes a Tauopathy with Abundant Pick Body-like Inclusions and Axonal Deposits

Treatment of first tonic–clonic seizure does not affect mortality: long-term follow-up of a randomised clinical trial

Changes in magnetic resonance imaging disease measures over 3 years in mildly disabled patients with relapsing-remitting multiple sclerosis receiving interferon β-1a in the COGnitive Impairment in MUltiple Sclerosis (COGIMUS) study

Progress in Hereditary Tauopathies: A Mutation in the Tau Gene (G389R) Causes a Pick Disease-like Syndrome

Social Aspects of Epilepsy in the Adult in Seven European Countries

Psychiatric events in epilepsy

Effects of frontal lesions on a selective attention task

Cost-of-illness of epilepsy in Italy. Data from a multicentre observational study (Episcreen)

Prognosis of Epilepsy in Newly Referred Patients: A Multicenter Prospective Study of the Effects of Monotherapy on the Long‐Term Course of Epilepsy

Istiocitosi a cellule di Langherans: Coinvolgimento pachimeningeo ed otomastoideo

Predictive performance of pharmacokinetic methods for phenytoin dosing: a multi-center evaluation in 282 patients with epilepsy

Effects of immunomodulatory treatment with subcutaneous interferon beta-1a on cognitive decline in mildly disabled patients with relapsing-remitting multiple sclerosis

PICK DISEASE WITH GLIAL TAU INCLUSIONS IN THE WHITE MATTER

Allopurinol as Add‐on Therapy in Refractory Epilepsy: A Double‐Blind Placebo‐Controlled Randomized Study Italy, on his retirement

Allopurinol as add-on therapy in refractory epilepsy : a double-blind placebo-controlled randomized study

Concordance of Clinical Forms of Epilepsy in Families with Several Affected Members

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