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Diabetic Nephropathy: Novel Molecular Mechanisms and Therapeutic Targets

Frontiers in pharmacology, 2020-12, Vol.11, p.586892-586892

2020

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Manipulating Sirtuin 3 pathway ameliorates renal damage in experimental diabetes

Scientific reports, 2020-05, Vol.10 (1), p.8418-8418, Article 8418

2020

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Empagliflozin protects glomerular endothelial cell architecture in experimental diabetes through the VEGF‐A/caveolin‐1/PV‐1 signaling pathway

The Journal of pathology, 2022-04, Vol.256 (4), p.468-479

2022

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Shiga toxin triggers endothelial and podocyte injury: the role of complement activation

Pediatric nephrology (Berlin, West), 2019-03, Vol.34 (3), p.379-388

2019

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Post-translational modifications by SIRT3 de-2-hydroxyisobutyrylase activity regulate glycolysis and enable nephrogenesis

Scientific reports, 2021-12, Vol.11 (1), p.23580-23580, Article 23580

2021

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Sirtuin 3 Deficiency Aggravates Kidney Disease in Response to High-Fat Diet through Lipotoxicity-Induced Mitochondrial Damage

International journal of molecular sciences, 2022-08, Vol.23 (15), p.8345

2022

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Renal expression of FGF23 in progressive renal disease of diabetes and the effect of ACE inhibitor

PloS one, 2013-08, Vol.8 (8), p.e70775

2013

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SGLT2 inhibitor dapagliflozin limits podocyte damage in proteinuric nondiabetic nephropathy

JCI insight, 2018-08, Vol.3 (15)

2018

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Endothelial Glycocalyx of Peritubular Capillaries in Experimental Diabetic Nephropathy: A Target of ACE Inhibitor-Induced Kidney Microvascular Protection

International journal of molecular sciences, 2023-11, Vol.24 (22), p.16543

2023

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Complement Activation Contributes to the Pathophysiology of Shiga Toxin-Associated Hemolytic Uremic Syndrome

Microorganisms (Basel), 2019-01, Vol.7 (1), p.15

2019

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Lipoprotein X Causes Renal Disease in LCAT Deficiency

PloS one, 2016-02, Vol.11 (2), p.e0150083

2016

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ET and Diabetic Nephropathy: Preclinical and Clinical Studies

Seminars in nephrology, 2015-03, Vol.35 (2), p.188-196

2015

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Protective Effects of Human Nonrenal and Renal Stromal Cells and Their Conditioned Media in a Rat Model of Chronic Kidney Disease

Cell transplantation, 2020-01, Vol.29, p.963689720965467-963689720965467

2020

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C3a receptor blockade protects podocytes from injury in diabetic nephropathy

JCI insight, 2020-03, Vol.5 (5)

2020

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Shiga Toxin Promotes Podocyte Injury in Experimental Hemolytic Uremic Syndrome via Activation of the Alternative Pathway of Complement

Journal of the American Society of Nephrology, 2014-08, Vol.25 (8), p.1786-1798

2014

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Evaluation of the Zucker diabetic fatty (ZDF) rat as a model for human disease based on urinary peptidomic profiles

PloS one, 2012-12, Vol.7 (12), p.e51334-e51334

2012

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Add-On Cyclic Angiotensin-(1-7) with Cyclophosphamide Arrests Progressive Kidney Disease in Rats with ANCA Associated Glomerulonephritis

Cells (Basel, Switzerland), 2022-08, Vol.11 (15), p.2434

2022

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Low Nephron Number Induced by Maternal Protein Restriction Is Prevented by Nicotinamide Riboside Supplementation Depending on Sirtuin 3 Activation

Cells (Basel, Switzerland), 2022-10, Vol.11 (20), p.3316

2022

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Therapeutic potential of stromal cells of non-renal or renal origin in experimental chronic kidney disease

Stem cell research & therapy, 2018-08, Vol.9 (1), p.220-220, Article 220

2018

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Effects of MCP-1 Inhibition by Bindarit Therapy in a Rat Model of Polycystic Kidney Disease

Nephron (2015), 2015-01, Vol.129 (1), p.52-61

2015

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Lack of the lectin-like domain of thrombomodulin worsens Shiga toxin-associated hemolytic uremic syndrome in mice

The Journal of immunology (1950), 2012-10, Vol.189 (7), p.3661-3668

2012

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Renal primordia activate kidney regenerative events in a rat model of progressive renal disease

PloS one, 2015-03, Vol.10 (3), p.e0120235-e0120235

2015

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Thyroid hormone treatment counteracts cellular phenotypical remodeling in diabetic organs

iScience, 2023-10, Vol.26 (10), p.107826-107826, Article 107826

2023

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Shiga Toxin 2 Triggers C3a-Dependent Glomerular and Tubular Injury through Mitochondrial Dysfunction in Hemolytic Uremic Syndrome

Cells (Basel, Switzerland), 2022-05, Vol.11 (11), p.1755

2022

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Increased renal versican expression is associated with progression of chronic kidney disease

PloS one, 2012-09, Vol.7 (9), p.e44891-e44891

2012

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Diabetic Nephropathy: Novel Molecular Mechanisms and Therapeutic Targets

Manipulating Sirtuin 3 pathway ameliorates renal damage in experimental diabetes

Empagliflozin protects glomerular endothelial cell architecture in experimental diabetes through the VEGF‐A/caveolin‐1/PV‐1 signaling pathway

Shiga toxin triggers endothelial and podocyte injury: the role of complement activation

Post-translational modifications by SIRT3 de-2-hydroxyisobutyrylase activity regulate glycolysis and enable nephrogenesis

Sirtuin 3 Deficiency Aggravates Kidney Disease in Response to High-Fat Diet through Lipotoxicity-Induced Mitochondrial Damage

Renal expression of FGF23 in progressive renal disease of diabetes and the effect of ACE inhibitor

SGLT2 inhibitor dapagliflozin limits podocyte damage in proteinuric nondiabetic nephropathy

Endothelial Glycocalyx of Peritubular Capillaries in Experimental Diabetic Nephropathy: A Target of ACE Inhibitor-Induced Kidney Microvascular Protection

Complement Activation Contributes to the Pathophysiology of Shiga Toxin-Associated Hemolytic Uremic Syndrome

Lipoprotein X Causes Renal Disease in LCAT Deficiency

ET and Diabetic Nephropathy: Preclinical and Clinical Studies

Protective Effects of Human Nonrenal and Renal Stromal Cells and Their Conditioned Media in a Rat Model of Chronic Kidney Disease

C3a receptor blockade protects podocytes from injury in diabetic nephropathy

Shiga Toxin Promotes Podocyte Injury in Experimental Hemolytic Uremic Syndrome via Activation of the Alternative Pathway of Complement

Evaluation of the Zucker diabetic fatty (ZDF) rat as a model for human disease based on urinary peptidomic profiles

Add-On Cyclic Angiotensin-(1-7) with Cyclophosphamide Arrests Progressive Kidney Disease in Rats with ANCA Associated Glomerulonephritis

Low Nephron Number Induced by Maternal Protein Restriction Is Prevented by Nicotinamide Riboside Supplementation Depending on Sirtuin 3 Activation

Therapeutic potential of stromal cells of non-renal or renal origin in experimental chronic kidney disease

Effects of MCP-1 Inhibition by Bindarit Therapy in a Rat Model of Polycystic Kidney Disease

Lack of the lectin-like domain of thrombomodulin worsens Shiga toxin-associated hemolytic uremic syndrome in mice

Renal primordia activate kidney regenerative events in a rat model of progressive renal disease

Thyroid hormone treatment counteracts cellular phenotypical remodeling in diabetic organs

Shiga Toxin 2 Triggers C3a-Dependent Glomerular and Tubular Injury through Mitochondrial Dysfunction in Hemolytic Uremic Syndrome

Increased renal versican expression is associated with progression of chronic kidney disease

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