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Activation of nicotinamide adenine dinucleotide phosphate oxidase is the primary trigger of epileptic seizures in rodent models

Annals of neurology, 2019-06, Vol.85 (6), p.907-920

2019

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The vicious circle of hypometabolism in neurodegenerative diseases: Ways and mechanisms of metabolic correction

Journal of neuroscience research, 2017-11, Vol.95 (11), p.2217-2235

2017

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Glycolysis and Oxidative Phosphorylation in Neurons and Astrocytes during Network Activity in Hippocampal Slices

Journal of cerebral blood flow and metabolism, 2014-03, Vol.34 (3), p.397-407

2014

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Aβ initiates brain hypometabolism, network dysfunction and behavioral abnormalities via NOX2-induced oxidative stress in mice

Communications biology, 2021-09, Vol.4 (1), p.1054-1054, Article 1054

2021

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Seizure-induced reduction in glucose utilization promotes brain hypometabolism during epileptogenesis

Neurobiology of disease, 2018-08, Vol.116, p.28-38

2018

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In Vivo Chimeric Alzheimer’s Disease Modeling of Apolipoprotein E4 Toxicity in Human Neurons

Cell reports (Cambridge), 2020-07, Vol.32 (4), p.107962-107962, Article 107962

2020

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Neurons require glucose uptake and glycolysis in vivo

Cell reports (Cambridge), 2023-04, Vol.42 (4), p.112335-112335, Article 112335

2023

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Unifying mechanism behind the onset of acquired epilepsy

Trends in pharmacological sciences (Regular ed.), 2022-02, Vol.43 (2), p.87-96

2022

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Secretagogin is a Ca²⁺-binding protein specifying subpopulations of telencephalic neurons

Proceedings of the National Academy of Sciences - PNAS, 2009-12, Vol.106 (52), p.22492-22497

2009

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Reality of Inhibitory GABA in Neonatal Brain: Time to Rewrite the Textbooks?

The Journal of neuroscience, 2016-10, Vol.36 (40), p.10242-10244

Zilberter, Misha

2016

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Input Specificity and Dependence of Spike Timing–Dependent Plasticity on Preceding Postsynaptic Activity at Unitary Connections between Neocortical Layer 2/3 Pyramidal Cells

Cerebral cortex (New York, N.Y. 1991), 2009-10, Vol.19 (10), p.2308-2320

2009

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NOX-induced oxidative stress is a primary trigger of major neurodegenerative disorders

Progress in neurobiology, 2023-12, Vol.231, p.102539-102539, Article 102539

2023

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En masse in vitro functional profiling of the axonal mechanosensitivity of sensory neurons

Proceedings of the National Academy of Sciences - PNAS, 2010-09, Vol.107 (37), p.16336-16341

2010

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Amyloid beta-induced neuronal hyperexcitability triggers progressive epilepsy

The Journal of neuroscience, 2009-03, Vol.29 (11), p.3453-3462

2009

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Chronic inhibition of brain glycolysis initiates epileptogenesis

Journal of neuroscience research, 2017-11, Vol.95 (11), p.2195-2206

2017

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Chemically defined, albumin-free human cardiomyocyte generation

Nature methods, 2015-07, Vol.12 (7), p.595-596

2015

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Activation of NADPH oxidase is the primary trigger of epileptic seizures in rodent models

Annals of neurology, 2019

2019

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Amyloid-β-induced action potential desynchronization and degradation of hippocampal gamma oscillations is prevented by interference with peptide conformation change and aggregation

The Journal of neuroscience, 2014-08, Vol.34 (34), p.11416-11425

2014

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Adult trkB Signaling in Parvalbumin Interneurons is Essential to Prefrontal Network Dynamics

The Journal of neuroscience, 2021-04, Vol.41 (14), p.3120-3141

2021

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Dietary energy substrates reverse early neuronal hyperactivity in a mouse model of Alzheimer's disease

Journal of neurochemistry, 2013-04, Vol.125 (1), p.157-171

2013

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Molecular reorganization of endocannabinoid signalling in Alzheimer's disease

Brain (London, England : 1878), 2011-04, Vol.134 (Pt 4), p.1041-1060

2011

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Neuronal APOE4 removal protects against tau-mediated gliosis, neurodegeneration and myelin deficits

Nature aging, 2023-03, Vol.3 (3), p.275-296

2023

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GABAergic control of backpropagating action potentials in striatal medium spiny neurons

BMC neuroscience, 2008-07, Vol.9 (S1), p.P105-P105, Article P105

2008

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Activation of nicotinamide adenine dinucleotide phosphate oxidase is the primary trigger of epileptic seizures in rodent models

The vicious circle of hypometabolism in neurodegenerative diseases: Ways and mechanisms of metabolic correction

Glycolysis and Oxidative Phosphorylation in Neurons and Astrocytes during Network Activity in Hippocampal Slices

Aβ initiates brain hypometabolism, network dysfunction and behavioral abnormalities via NOX2-induced oxidative stress in mice

Seizure-induced reduction in glucose utilization promotes brain hypometabolism during epileptogenesis

In Vivo Chimeric Alzheimer’s Disease Modeling of Apolipoprotein E4 Toxicity in Human Neurons

Neurons require glucose uptake and glycolysis in vivo

Experimental and real-world evidence supporting the computational repurposing of bumetanide for APOE4-related Alzheimer’s disease

Unifying mechanism behind the onset of acquired epilepsy

Secretagogin is a Ca²⁺-binding protein specifying subpopulations of telencephalic neurons

Reality of Inhibitory GABA in Neonatal Brain: Time to Rewrite the Textbooks?

Input Specificity and Dependence of Spike Timing–Dependent Plasticity on Preceding Postsynaptic Activity at Unitary Connections between Neocortical Layer 2/3 Pyramidal Cells

NOX-induced oxidative stress is a primary trigger of major neurodegenerative disorders

Author Correction: Experimental and real-world evidence supporting the computational repurposing of bumetanide for APOE4-related Alzheimer’s disease

En masse in vitro functional profiling of the axonal mechanosensitivity of sensory neurons

Amyloid beta-induced neuronal hyperexcitability triggers progressive epilepsy

Chronic inhibition of brain glycolysis initiates epileptogenesis

Chemically defined, albumin-free human cardiomyocyte generation

Activation of NADPH oxidase is the primary trigger of epileptic seizures in rodent models

Amyloid-β-induced action potential desynchronization and degradation of hippocampal gamma oscillations is prevented by interference with peptide conformation change and aggregation

Adult trkB Signaling in Parvalbumin Interneurons is Essential to Prefrontal Network Dynamics

Dietary energy substrates reverse early neuronal hyperactivity in a mouse model of Alzheimer's disease

Molecular reorganization of endocannabinoid signalling in Alzheimer's disease

Neuronal APOE4 removal protects against tau-mediated gliosis, neurodegeneration and myelin deficits

GABAergic control of backpropagating action potentials in striatal medium spiny neurons

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